Comparison of the Hemodynamic Response to Intra-Aortic Balloon Counterpulsation in Patients With Cardiogenic Shock Resulting from Acute Myocardial Infarction Versus Acute Decompensated Heart Failure.
Acute Disease
Aged
Cardiac Output
/ physiology
Cohort Studies
Female
Heart Failure
/ complications
Hemodynamics
/ physiology
Hospital Mortality
Humans
Intra-Aortic Balloon Pumping
Male
Middle Aged
Myocardial Infarction
/ complications
Prognosis
Retrospective Studies
Risk Assessment
Shock, Cardiogenic
/ etiology
Survival Analysis
Treatment Outcome
Journal
The American journal of cardiology
ISSN: 1879-1913
Titre abrégé: Am J Cardiol
Pays: United States
ID NLM: 0207277
Informations de publication
Date de publication:
15 12 2019
15 12 2019
Historique:
received:
18
07
2019
revised:
03
09
2019
accepted:
06
09
2019
pubmed:
28
10
2019
medline:
2
4
2020
entrez:
26
10
2019
Statut:
ppublish
Résumé
The intra-aortic balloon pump (IABP) neither benefits nor harms patients with acute myocardial infarction (AMI) with cardiogenic shock (CS) but may stabilize those with chronic heart failure who decompensate into CS. We sought to compare its hemodynamic effects in these 2 populations. We performed a retrospective analysis of the hemodynamic effects of IABP for AMI or acute decompensated heart failure (ADHF) patients with hemodynamic evidence of CS. The primary outcome was cardiac output (CO) change following insertion. In total, 205 patients were treated for CS resulting from AMI (73; 35.6%) or ADHF (132; 64.4%). At baseline, both cohorts had significant hemodynamic compromise with mean arterial pressure 75.6 ± 12.3 mm Hg, CO 3.02 ± 0.84 L/min, and cardiac power index 0.26 ± 0.06 W/m
Identifiants
pubmed: 31648782
pii: S0002-9149(19)31054-9
doi: 10.1016/j.amjcard.2019.09.016
pmc: PMC6937209
mid: NIHMS1544825
pii:
doi:
Types de publication
Comparative Study
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1947-1953Subventions
Organisme : NCATS NIH HHS
ID : KL2 TR001874
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR000040
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001873
Pays : United States
Informations de copyright
Copyright © 2019 Elsevier Inc. All rights reserved.
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