Epidermal growth factor receptor signaling suppresses αvβ6 integrin and promotes periodontal inflammation and bone loss.


Journal

Journal of cell science
ISSN: 1477-9137
Titre abrégé: J Cell Sci
Pays: England
ID NLM: 0052457

Informations de publication

Date de publication:
05 12 2019
Historique:
received: 15 07 2019
accepted: 05 11 2019
pubmed: 15 11 2019
medline: 22 9 2020
entrez: 15 11 2019
Statut: epublish

Résumé

In periodontal disease (PD), bacterial biofilms cause gingival inflammation, leading to bone loss. In healthy individuals, αvβ6 integrin in junctional epithelium maintains anti-inflammatory transforming growth factor-β1 (TGF-β1) signaling, whereas its expression is lost in individuals with PD. Bacterial biofilms suppress β6 integrin expression in cultured gingival epithelial cells (GECs) by attenuating TGF-β1 signaling, leading to an enhanced pro-inflammatory response. In the present study, we show that GEC exposure to biofilms induced activation of mitogen-activated protein kinases and epidermal growth factor receptor (EGFR). Inhibition of EGFR and ERK stunted both the biofilm-induced

Identifiants

pubmed: 31722981
pii: jcs.236588
doi: 10.1242/jcs.236588
pii:
doi:

Substances chimiques

Antigens, Neoplasm 0
Inflammation Mediators 0
Integrins 0
Transforming Growth Factor beta1 0
integrin alphavbeta6 0
ErbB Receptors EC 2.7.10.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : CIHR
ID : PJT-153379
Pays : Canada
Organisme : CIHR
ID : PJT-156387
Pays : Canada

Informations de copyright

© 2019. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interestsThe authors declare no competing or financial interests.

Auteurs

Jiarui Bi (J)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Leeni Koivisto (L)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Jiayin Dai (J)

Department of Stomatology, The Fourth Affiliated Hospital, Harbin Medical University, Harbin, 150001, China.

Deshu Zhuang (D)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.
Department of Stomatology, The Fourth Affiliated Hospital, Harbin Medical University, Harbin, 150001, China.

Guoqiao Jiang (G)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Milla Larjava (M)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Ya Shen (Y)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Liangjia Bi (L)

Department of Stomatology, The Fourth Affiliated Hospital, Harbin Medical University, Harbin, 150001, China.

Fang Liu (F)

Center for Advanced Biotechnology and Medicine, Susan Lehman Cullman Laboratory for Cancer Research, Ernest Mario School of Pharmacy, Rutgers Cancer Institute of New Jersey, Rutgers, The State University of New Jersey, Piscataway, NJ 08854, USA.

Markus Haapasalo (M)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Lari Häkkinen (L)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada.

Hannu Larjava (H)

Faculty of Dentistry, Department of Oral Biological and Medical Sciences, University of British Columbia, Vancouver, BC V6T 1Z3, Canada larjava@dentistry.ubc.ca.

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Classifications MeSH