Mycobacterial Trehalose 6,6'-Dimycolate-Induced M1-Type Inflammation.
Journal
The American journal of pathology
ISSN: 1525-2191
Titre abrégé: Am J Pathol
Pays: United States
ID NLM: 0370502
Informations de publication
Date de publication:
02 2020
02 2020
Historique:
received:
12
07
2019
revised:
23
09
2019
accepted:
11
10
2019
pubmed:
18
11
2019
medline:
6
5
2020
entrez:
18
11
2019
Statut:
ppublish
Résumé
Murine models of Mycobacterium tuberculosis (Mtb) infection demonstrate progression of M1-like (proinflammatory) and M2-like (anti-inflammatory) macrophage morphology following primary granuloma formation. The Mtb cell wall cording factor, trehalose 6,6'-dimycolate (TDM), is a physiologically relevant and useful molecule for modeling early macrophage-mediated events during establishment of the tuberculosis-induced granuloma pathogenesis. Here, it is shown that TDM is a major driver of the early M1-like macrophage response as seen during initiation of the granulomas of primary pathology. Proinflammatory cytokines tumor necrosis factor-α, IL-1β, IL-6, and IL-12p40 are produced in lung tissue after administration of TDM to mice. Furthermore, CD11b
Identifiants
pubmed: 31734231
pii: S0002-9440(19)30803-X
doi: 10.1016/j.ajpath.2019.10.006
pmc: PMC7013273
pii:
doi:
Substances chimiques
Adjuvants, Immunologic
0
Cord Factors
0
Inflammation Mediators
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
286-294Subventions
Organisme : NIAID NIH HHS
ID : R41 AI117990
Pays : United States
Informations de copyright
Copyright © 2020 American Society for Investigative Pathology. Published by Elsevier Inc. All rights reserved.
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