A double-blind, randomized, placebo-controlled pilot trial to evaluate safety and efficacy of vorapaxar on arteriovenous fistula maturation.


Journal

The journal of vascular access
ISSN: 1724-6032
Titre abrégé: J Vasc Access
Pays: United States
ID NLM: 100940729

Informations de publication

Date de publication:
Jul 2020
Historique:
pubmed: 28 11 2019
medline: 29 9 2020
entrez: 28 11 2019
Statut: ppublish

Résumé

Protease-activated receptor-1 antagonism by vorapaxar could facilitate arteriovenous fistula maturation but may increase bleeding risk. The primary objective of the Vorapaxar Study for Maturation of arteriovenous fistula for Hemodialysis Access (VorapAccess) was to determine if vorapaxar improves arteriovenous fistula functional maturation in patients with end-stage renal disease. VorapAccess was a randomized, placebo-controlled, double-blind pilot trial comparing 2.5 mg vorapaxar per day with placebo for twelve weeks starting on day two after arteriovenous fistula creation. The primary outcome was time to functional maturation defined as successful cannulation for six hemodialysis sessions within three weeks. The planned sample size was 50 participants. The study was terminated early after withdrawal of planned financial support. Given the small number of randomized patients, we performed descriptive analyses without inference testing. A total of 13 participants were randomly allocated study drug (six vorapaxar and seven placebo). The median age was 56 years and seven participants (54%) were female. The median (minimum-maximum) days to functional maturation were 169 (77-287) days in the vorapaxar group and 145 (48-198) days in the placebo group. Six of the 13 (46%) participants had arteriovenous fistula functional maturation within 180 days; two of six (33%) in the vorapaxar group and four of seven (57%) in the placebo group. There was one bleeding event in the placebo group. Fewer than half of participants had functional maturation within 180 days after surgery, suggesting a major need for agents or strategies that enhance arteriovenous fistula maturation.

Sections du résumé

BACKGROUND BACKGROUND
Protease-activated receptor-1 antagonism by vorapaxar could facilitate arteriovenous fistula maturation but may increase bleeding risk.
OBJECTIVE OBJECTIVE
The primary objective of the Vorapaxar Study for Maturation of arteriovenous fistula for Hemodialysis Access (VorapAccess) was to determine if vorapaxar improves arteriovenous fistula functional maturation in patients with end-stage renal disease.
METHODS METHODS
VorapAccess was a randomized, placebo-controlled, double-blind pilot trial comparing 2.5 mg vorapaxar per day with placebo for twelve weeks starting on day two after arteriovenous fistula creation. The primary outcome was time to functional maturation defined as successful cannulation for six hemodialysis sessions within three weeks. The planned sample size was 50 participants. The study was terminated early after withdrawal of planned financial support. Given the small number of randomized patients, we performed descriptive analyses without inference testing.
RESULTS RESULTS
A total of 13 participants were randomly allocated study drug (six vorapaxar and seven placebo). The median age was 56 years and seven participants (54%) were female. The median (minimum-maximum) days to functional maturation were 169 (77-287) days in the vorapaxar group and 145 (48-198) days in the placebo group. Six of the 13 (46%) participants had arteriovenous fistula functional maturation within 180 days; two of six (33%) in the vorapaxar group and four of seven (57%) in the placebo group. There was one bleeding event in the placebo group.
CONCLUSION CONCLUSIONS
Fewer than half of participants had functional maturation within 180 days after surgery, suggesting a major need for agents or strategies that enhance arteriovenous fistula maturation.

Identifiants

pubmed: 31774037
doi: 10.1177/1129729819887269
pmc: PMC8063544
mid: NIHMS1682370
doi:

Substances chimiques

Lactones 0
Platelet Aggregation Inhibitors 0
Pyridines 0
Receptor, PAR-1 0
vorapaxar ZCE93644N2

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

467-474

Subventions

Organisme : NIDDK NIH HHS
ID : P30 DK116074
Pays : United States

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Auteurs

Christoph B Olivier (CB)

Stanford Center for Clinical Research (SCCR), Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.
Department of Cardiology and Angiology I, Faculty of Medicine, Heart Center, University of Freiburg, Freiburg, Germany.

Vandana Sundaram (V)

Quantitative Sciences Unit, Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.

Glenn M Chertow (GM)

Division of Nephrology, School of Medicine, Stanford University, Stanford, CA, USA.

Sumana Shashidhar (S)

Stanford Center for Clinical Research (SCCR), Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.

Lori K McDonnell (LK)

Division of Vascular Surgery, School of Medicine, Stanford University, Stanford, CA, USA.

Victoria Y Ding (VY)

Quantitative Sciences Unit, Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.

Manisha Desai (M)

Quantitative Sciences Unit, Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.

Kenneth W Mahaffey (KW)

Stanford Center for Clinical Research (SCCR), Department of Medicine, School of Medicine, Stanford University, Stanford, CA, USA.

Matthew Mell (M)

Division of Vascular Surgery, School of Medicine, Stanford University, Stanford, CA, USA.
Division of Vascular Surgery, University of California at Davis, Sacramento, CA, USA.

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Classifications MeSH