Elucidation of cGMP-dependent induction of mitochondrial biogenesis through PKG and p38 MAPK in the kidney.


Journal

American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990

Informations de publication

Date de publication:
01 02 2020
Historique:
pubmed: 17 12 2019
medline: 12 5 2020
entrez: 17 12 2019
Statut: ppublish

Résumé

Previous studies have shown that cGMP increases mitochondrial biogenesis (MB). Our laboratory has determined that formoterol and LY344864, agonists of the β

Identifiants

pubmed: 31841384
doi: 10.1152/ajprenal.00533.2019
pmc: PMC7052650
doi:

Substances chimiques

Carbazoles 0
Dibenzocycloheptenes 0
Enzyme Activators 0
Fluorobenzenes 0
LY 344864 0
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha 0
Protein Kinase Inhibitors 0
Pyrazoles 0
Pyrimidines 0
skepinone-L 0
KT 5823 126643-37-6
Cyclic GMP-Dependent Protein Kinases EC 2.7.11.12
p38 Mitogen-Activated Protein Kinases EC 2.7.11.24
Cyclic GMP H2D2X058MU
riociguat RU3FE2Y4XI
Formoterol Fumarate W34SHF8J2K

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

F322-F328

Subventions

Organisme : NIEHS NIH HHS
ID : P30 ES006694
Pays : United States

Références

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Auteurs

Pallavi Bhargava (P)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.

Jaroslav Janda (J)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.

Rick G Schnellmann (RG)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.
Southern Arizona Veterans Affairs Health Care System, Tucson, Arizona.

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Classifications MeSH