[Association of the TCF7L2 (RS7903146) genotype with adiposity and metabolic markers in the Chilean adult population].

Asociación del polimorfismo rs7903146, del gen TCF7L2, con marcadores de adiposidad y metabólicos en población chilena - resultados del estudio GENADIO.

Journal

Revista medica de Chile
ISSN: 0717-6163
Titre abrégé: Rev Med Chil
Pays: Chile
ID NLM: 0404312

Informations de publication

Date de publication:
Aug 2019
Historique:
received: 15 07 2018
accepted: 07 08 2019
entrez: 21 12 2019
pubmed: 21 12 2019
medline: 25 2 2020
Statut: ppublish

Résumé

Type 2 diabetes etiology has a strong genetic component. More than 20 genetic variants have been associated with diabetes and other metabolic markers. However, the polymorphism rs7903146 of the TCF7L2 gene has shown the strongest association. To investigate the association of TCF7L2 (rs7903146) genotype with adiposity and metabolic markers in the Chilean adult population. The association of TCF7L2 (rs7093146) with adiposity and metabolic markers was studied in 301 participants. The outcomes of the study were adiposity markers (body weight, body mass index (BMI), fat mass and waist circumference) and metabolic markers (blood glucose, insulin, HOMA-IR, lipid profile, high sensitivity C-reactive protein (CRP), alanine aminotransferase (ALT), gamma glutamyl transpeptidase (GGT) and leptin). There was an association between the polymorphism TCF7L2 genotype and fasting blood glucose. The latter increased by 4.86 mg/dl per each copy of the risk allele [(95% confidence intervals (CI): 0.48; 9.24), p = 0.03] in the unadjusted adjusted model. However, this association was slightly attenuated in the fully adjusted model [4.38 mg/dl (95% IC: 0.16; 8.60), p = 0.04)]. There were no associations between the TCF7L2 genotype and any other metabolic or adiposity outcome. These findings confirm the association between the TCF7L2 (rs7903146) and fasting glucose in the Chilean population. However, further studies are needed to confirm the association between the TCF7L2 and diabetes risk in the Chilean population.

Sections du résumé

BACKGROUND BACKGROUND
Type 2 diabetes etiology has a strong genetic component. More than 20 genetic variants have been associated with diabetes and other metabolic markers. However, the polymorphism rs7903146 of the TCF7L2 gene has shown the strongest association.
AIM OBJECTIVE
To investigate the association of TCF7L2 (rs7903146) genotype with adiposity and metabolic markers in the Chilean adult population.
MATERIAL AND METHODS METHODS
The association of TCF7L2 (rs7093146) with adiposity and metabolic markers was studied in 301 participants. The outcomes of the study were adiposity markers (body weight, body mass index (BMI), fat mass and waist circumference) and metabolic markers (blood glucose, insulin, HOMA-IR, lipid profile, high sensitivity C-reactive protein (CRP), alanine aminotransferase (ALT), gamma glutamyl transpeptidase (GGT) and leptin).
RESULTS RESULTS
There was an association between the polymorphism TCF7L2 genotype and fasting blood glucose. The latter increased by 4.86 mg/dl per each copy of the risk allele [(95% confidence intervals (CI): 0.48; 9.24), p = 0.03] in the unadjusted adjusted model. However, this association was slightly attenuated in the fully adjusted model [4.38 mg/dl (95% IC: 0.16; 8.60), p = 0.04)]. There were no associations between the TCF7L2 genotype and any other metabolic or adiposity outcome.
CONCLUSIONS CONCLUSIONS
These findings confirm the association between the TCF7L2 (rs7903146) and fasting glucose in the Chilean population. However, further studies are needed to confirm the association between the TCF7L2 and diabetes risk in the Chilean population.

Identifiants

pubmed: 31859960
pii: S0034-98872019000800965
doi: 10.4067/S0034-98872019000800965
pii:
doi:

Substances chimiques

Blood Glucose 0
Genetic Markers 0
TCF7L2 protein, human 0
Transcription Factor 7-Like 2 Protein 0

Types de publication

Journal Article

Langues

spa

Sous-ensembles de citation

IM

Pagination

965-976

Auteurs

Fanny Petermann-Rocha (F)

Institute of Health and Wellbeing, University of Glasgow, Glasgow, United Kingdom.

Nicole Lasserre-Laso (N)

Escuela de Nutrición y Dietética, Facultad de Salud, Universidad Santo Tomás, Sede los Ángeles, Chile.

Marcelo Villagrán (M)

Departamento de Ciencias Básicas, Universidad Católica de la Santísima Concepción, Concepción, Chile.

Lorena Mardones (L)

Departamento de Ciencias Básicas, Universidad Católica de la Santísima Concepción, Concepción, Chile.

María Adela Martínez (MA)

Instituto de Farmacia, Facultad de Ciencias, Universidad Austral de Chile, Valdivia, Chile.

Ana María Leiva (AM)

Instituto de Anatomía, Histología y Patología, Facultad de Medicina, Universidad Austral de Chile, Valdivia, Chile.

Natalia Ulloa (N)

Departamento de Bioquímica Clínica e Inmunología, Facultad de Farmacia, Centro de Vida Saludable de la Universidad de Concepción, Concepción, Chile.

Carlos Celis-Morales (C)

BHF Glasgow Cardiovascular Research Centre, Institute of Cardiovascular and Medical Sciences, University of Glasgow, Glasgow, United Kingdom.

Articles similaires

Genome, Chloroplast Phylogeny Genetic Markers Base Composition High-Throughput Nucleotide Sequencing

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C

Classifications MeSH