Dehydrin ERD14 activates glutathione transferase Phi9 in Arabidopsis thaliana under osmotic stress.


Journal

Biochimica et biophysica acta. General subjects
ISSN: 1872-8006
Titre abrégé: Biochim Biophys Acta Gen Subj
Pays: Netherlands
ID NLM: 101731726

Informations de publication

Date de publication:
03 2020
Historique:
received: 18 08 2019
revised: 12 12 2019
accepted: 18 12 2019
pubmed: 25 12 2019
medline: 29 8 2020
entrez: 25 12 2019
Statut: ppublish

Résumé

Fully intrinsically disordered plant dehydrin ERD14 can protect enzymes via its chaperone-like activity, but it was not formally linked with enzymes of the plant redox system yet. This is of particular interest, as the level of H The proteomic mass-spectrometry analysis of stressed plants was performed to find the candidates affected by ERD14. With cross-linking, microscale thermophoresis, and active-site titration kinetics, the interaction and influence of ERD14 on the function of two target proteins: glutathione transferase Phi9 and catalase was examined. Under osmotic stress, redox enzymes, specifically the glutathione transferase Phi enzymes, are upregulated. Using microscale thermophoresis, we showed that ERD14 directly interacts with GSTF9 with a K We propose that fully intrinsically disordered dehydrin ERD14 might protect and even activate redox enzymes, helping plants to survive oxidative stress under dehydration conditions. ERD14 has a direct effect on the activity of redox enzymes.

Sections du résumé

BACKGROUND
Fully intrinsically disordered plant dehydrin ERD14 can protect enzymes via its chaperone-like activity, but it was not formally linked with enzymes of the plant redox system yet. This is of particular interest, as the level of H
METHODS
The proteomic mass-spectrometry analysis of stressed plants was performed to find the candidates affected by ERD14. With cross-linking, microscale thermophoresis, and active-site titration kinetics, the interaction and influence of ERD14 on the function of two target proteins: glutathione transferase Phi9 and catalase was examined.
RESULTS
Under osmotic stress, redox enzymes, specifically the glutathione transferase Phi enzymes, are upregulated. Using microscale thermophoresis, we showed that ERD14 directly interacts with GSTF9 with a K
CONCLUSIONS
We propose that fully intrinsically disordered dehydrin ERD14 might protect and even activate redox enzymes, helping plants to survive oxidative stress under dehydration conditions.
GENERAL SIGNIFICANCE
ERD14 has a direct effect on the activity of redox enzymes.

Identifiants

pubmed: 31870857
pii: S0304-4165(19)30295-8
doi: 10.1016/j.bbagen.2019.129506
pii:
doi:

Substances chimiques

Arabidopsis Proteins 0
ERD14 protein, Arabidopsis 0
Plant Proteins 0
dehydrin proteins, plant 134711-03-8
Hydrogen Peroxide BBX060AN9V
GSTF9 protein, Arabidopsis EC 2.5.1.18
Glutathione Transferase EC 2.5.1.18

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

129506

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Phuong N Nguyen (PN)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium; Department of Biology, College of Natural Sciences, Cantho University, Viet Nam; Laboratory of Plant Genetics (PLAN), Vrije Universiteit Brussel (VUB), Brussels, Belgium.

Maria-Armineh Tossounian (MA)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium; Brussels Center for Redox Biology, 1050 Brussels, Belgium.

Denes S Kovacs (DS)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium.

Tran T Thu (TT)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium.

Benoit Stijlemans (B)

Lab of Cellular and Molecular Immunology, Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium; Myeloid Cell Immunology Lab, VIB Center for Inflammation Research, Brussels, Belgium.

Didier Vertommen (D)

de Duve Institute, Université Catholique de Louvain, 1200 Brussels, Belgium.

Jarne Pauwels (J)

VIB-UGent Center for Medical Biotechnology, University of Ghent, B9000 Ghent, Belgium; Department of Biomolecular Medicine, University of Ghent, B9000 Ghent, Belgium.

Kris Gevaert (K)

VIB-UGent Center for Medical Biotechnology, University of Ghent, B9000 Ghent, Belgium; Department of Biomolecular Medicine, University of Ghent, B9000 Ghent, Belgium.

Geert Angenon (G)

Laboratory of Plant Genetics (PLAN), Vrije Universiteit Brussel (VUB), Brussels, Belgium.

Joris Messens (J)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium; Brussels Center for Redox Biology, 1050 Brussels, Belgium. Electronic address: joris.messens@vub.be.

Peter Tompa (P)

VIB-VUB Center for Structural Biology (CSB), Vlaams Instituut voor Biotechnologie (VIB), Brussels, Belgium; Structural Biology Brussels (SBB), Vrije Universiteit Brussel (VUB), 1050 Brussels, Belgium; Institute of Enzymology, Research Centre for Natural Sciences of the Hungarian Academy of Sciences, Budapest, Hungary. Electronic address: peter.tompa@vub.be.

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Classifications MeSH