Weighted Correlation Network Analysis Reveals CDK2 as a Regulator of a Ubiquitous Environmental Toxin-Induced Cell-Cycle Arrest.


Journal

Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052

Informations de publication

Date de publication:
07 01 2020
Historique:
received: 09 12 2019
revised: 20 12 2019
accepted: 23 12 2019
entrez: 16 1 2020
pubmed: 16 1 2020
medline: 16 1 2021
Statut: epublish

Résumé

Environmental food contaminants constitute a threat to human health. For instance, the globally spread mycotoxin Ochratoxin A (OTA) contributes to chronic kidney damage by affecting proximal tubule cells via unknown mechanisms. We applied a top-down approach to identify relevant toxicological mechanisms of OTA using RNA-sequencing followed by in-depth bioinformatics analysis and experimental validation. Differential expression analyses revealed that OTA led to the regulation of gene expression in kidney human cell lines, including for genes enriched in cell cycle-related pathways, and OTA-induced gap 1 and 2 (G1 and G2) cell-cycle arrests were observed. Weighted correlation network analysis highlighted cyclin dependent kinase 2 (CDK2) as a putative key regulator of this effect. CDK2 was downregulated by OTA exposure, and its overexpression partially blocked the OTA-induced G1 but not G2 cell-cycle arrest. We, therefore, propose CDK2 as one of the key regulators of the G1 cell-cycle arrest induced by low nanomolar concentrations of OTA.

Identifiants

pubmed: 31936152
pii: cells9010143
doi: 10.3390/cells9010143
pmc: PMC7017252
pii:
doi:

Substances chimiques

CDKN1A protein, human 0
Cyclin-Dependent Kinase Inhibitor p21 0
E2F1 Transcription Factor 0
E2F1 protein, human 0
E2F4 Transcription Factor 0
E2F4 protein, human 0
Environmental Pollutants 0
Ochratoxins 0
ochratoxin A 1779SX6LUY
Cyclin-Dependent Kinase 2 EC 2.7.11.22

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Déclaration de conflit d'intérêts

The authors declare no conflicts of interest.

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Auteurs

Virginie Dubourg (V)

Julius-Bernstein-Institute for Physiology, Martin-Luther-University Halle-Wittenberg, 06112 Halle (Saale), Germany.

Alexander Nolze (A)

Julius-Bernstein-Institute for Physiology, Martin-Luther-University Halle-Wittenberg, 06112 Halle (Saale), Germany.

Michael Kopf (M)

Julius-Bernstein-Institute for Physiology, Martin-Luther-University Halle-Wittenberg, 06112 Halle (Saale), Germany.

Michael Gekle (M)

Julius-Bernstein-Institute for Physiology, Martin-Luther-University Halle-Wittenberg, 06112 Halle (Saale), Germany.

Gerald Schwerdt (G)

Julius-Bernstein-Institute for Physiology, Martin-Luther-University Halle-Wittenberg, 06112 Halle (Saale), Germany.

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Classifications MeSH