Cholesterol and beyond - The role of the mevalonate pathway in cancer biology.


Journal

Biochimica et biophysica acta. Reviews on cancer
ISSN: 1879-2561
Titre abrégé: Biochim Biophys Acta Rev Cancer
Pays: Netherlands
ID NLM: 9806362

Informations de publication

Date de publication:
04 2020
Historique:
received: 25 10 2019
revised: 14 01 2020
accepted: 30 01 2020
pubmed: 3 2 2020
medline: 22 7 2020
entrez: 3 2 2020
Statut: ppublish

Résumé

Cancer is a multifaceted global disease. Transformation of a normal to a malignant cell takes several steps, including somatic mutations, epigenetic alterations, metabolic reprogramming and loss of cell growth control. Recently, the mevalonate pathway has emerged as a crucial regulator of tumor biology and a potential therapeutic target. This pathway controls cholesterol production and posttranslational modifications of Rho-GTPases, both of which are linked to several key steps of tumor progression. Inhibitors of the mevalonate pathway induce pleiotropic antitumor-effects in several human malignancies, identifying the pathway as an attractive candidate for novel therapies. In this review, we will provide an overview about the role and regulation of the mevalonate pathway in certain aspects of cancer initiation and progression and its potential for therapeutic intervention in oncology.

Identifiants

pubmed: 32007596
pii: S0304-419X(19)30174-X
doi: 10.1016/j.bbcan.2020.188351
pii:
doi:

Substances chimiques

Antineoplastic Agents 0
Sterol Regulatory Element Binding Proteins 0
Cholesterol 97C5T2UQ7J
HMGCR protein, human EC 1.1.1.-
Hydroxymethylglutaryl CoA Reductases EC 1.1.1.-
Geranyltranstransferase EC 2.5.1.10
rho GTP-Binding Proteins EC 3.6.5.2
Mevalonic Acid S5UOB36OCZ

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

188351

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors have received grants or honorarium for advisory boards or lectures to the individual or the institution by Amgen (MR, LCH, TDR), Biomedica (AG), Novartis (LCH, TDR), and Merck (LCH, TDR).

Auteurs

Andy Göbel (A)

Division of Endocrinology, Diabetes, and Bone Diseases, Department of Medicine III, Technische Universität Dresden, Germany; German Cancer Consortium (DKTK), partner site Dresden and German Cancer Research Center (DKFZ), Heidelberg, Germany. Electronic address: Andy.Goebel@ukdd.de.

Martina Rauner (M)

Division of Endocrinology, Diabetes, and Bone Diseases, Department of Medicine III, Technische Universität Dresden, Germany; German Cancer Consortium (DKTK), partner site Dresden and German Cancer Research Center (DKFZ), Heidelberg, Germany.

Lorenz C Hofbauer (LC)

Division of Endocrinology, Diabetes, and Bone Diseases, Department of Medicine III, Technische Universität Dresden, Germany; German Cancer Consortium (DKTK), partner site Dresden and German Cancer Research Center (DKFZ), Heidelberg, Germany; Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany.

Tilman D Rachner (TD)

Division of Endocrinology, Diabetes, and Bone Diseases, Department of Medicine III, Technische Universität Dresden, Germany; German Cancer Consortium (DKTK), partner site Dresden and German Cancer Research Center (DKFZ), Heidelberg, Germany; Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany.

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Classifications MeSH