β4-Nicotinic Receptors Are Critically Involved in Reward-Related Behaviors and Self-Regulation of Nicotine Reinforcement.
Animals
Behavior, Animal
/ drug effects
Conditioning, Operant
/ drug effects
Discrimination Learning
/ drug effects
Male
Mice
Mice, Knockout
Motivation
/ drug effects
Motor Activity
/ drug effects
Nerve Tissue Proteins
/ genetics
Nicotine
/ administration & dosage
Nicotinic Agonists
/ administration & dosage
Receptors, Nicotinic
/ genetics
Reward
Self Administration
Self-Control
Ventral Tegmental Area
/ drug effects
addiction
habenula
interpeduncular nucleus
nicotine
nicotinic receptors
reward
Journal
The Journal of neuroscience : the official journal of the Society for Neuroscience
ISSN: 1529-2401
Titre abrégé: J Neurosci
Pays: United States
ID NLM: 8102140
Informations de publication
Date de publication:
22 04 2020
22 04 2020
Historique:
received:
04
02
2019
revised:
30
01
2020
accepted:
18
02
2020
pubmed:
19
3
2020
medline:
24
9
2020
entrez:
19
3
2020
Statut:
ppublish
Résumé
Nicotine addiction, through smoking, is the principal cause of preventable mortality worldwide. Human genome-wide association studies have linked polymorphisms in the
Identifiants
pubmed: 32184221
pii: JNEUROSCI.0356-19.2020
doi: 10.1523/JNEUROSCI.0356-19.2020
pmc: PMC7178913
doi:
Substances chimiques
Chrnb4 protein, mouse
0
Nerve Tissue Proteins
0
Nicotinic Agonists
0
Receptors, Nicotinic
0
Nicotine
6M3C89ZY6R
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
3465-3477Subventions
Organisme : NIDA NIH HHS
ID : P30 DA035756
Pays : United States
Informations de copyright
Copyright © 2020 the authors.
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