Postprandial Hypoglycemia in Patients after Gastric Bypass Surgery Is Mediated by Glucose-Induced IL-1β.


Journal

Cell metabolism
ISSN: 1932-7420
Titre abrégé: Cell Metab
Pays: United States
ID NLM: 101233170

Informations de publication

Date de publication:
07 04 2020
Historique:
received: 06 09 2019
revised: 16 11 2019
accepted: 24 02 2020
pubmed: 21 3 2020
medline: 4 9 2021
entrez: 21 3 2020
Statut: ppublish

Résumé

Postprandial hypoglycemia is a disabling complication of the treatment of obesity by gastric bypass surgery. So far, no therapy exists, and the underlying mechanisms remain unclear. Here, we hypothesized that glucose-induced IL-1β leads to an exaggerated insulin response in this condition. Therefore, we conducted a placebo-controlled, randomized, double-blind, crossover study with the SGLT2-inhibitor empagliflozin and the IL-1 receptor antagonist anakinra (clinicaltrials.govNCT03200782; n = 12). Both drugs reduced postprandial insulin release and prevented hypoglycemia (symptomatic events requiring rescue glucose: placebo = 7/12, empagliflozin = 2/12, and anakinra = 2/12, pval

Identifiants

pubmed: 32197070
pii: S1550-4131(20)30072-3
doi: 10.1016/j.cmet.2020.02.013
pii:
doi:

Substances chimiques

Benzhydryl Compounds 0
Glucosides 0
IL1B protein, human 0
Interleukin 1 Receptor Antagonist Protein 0
Interleukin-1beta 0
Sodium-Glucose Transporter 2 Inhibitors 0
empagliflozin HDC1R2M35U
Glucose IY9XDZ35W2

Banques de données

ClinicalTrials.gov
['NCT03200782']

Types de publication

Clinical Trial Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

699-709.e5

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Interests M.Y.D. is listed as an inventor on a patent submitted by University Hospital Basel covering the use of SGLT-2 inhibitors or IL-1R antagonists for reduction of hypoglycemia after bariatric surgery (EP19151525.3). All others declare no competing interests.

Auteurs

Matthias Hepprich (M)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland; Endocrinology and Metabolic Diseases, Cantonal Hospital Olten, Olten, Switzerland.

Sophia J Wiedemann (SJ)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland.

Benjamin L Schelker (BL)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland.

Beckey Trinh (B)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland.

Alessandra Stärkle (A)

Swiss Federal Institute of Technology Zurich, Department of Health Sciences and Technology, Zurich, Switzerland.

Marco Geigges (M)

Department of Biosystems Science and Engineering, ETH Zürich, Basel, Switzerland.

Jordan Löliger (J)

Immunobiology Laboratory, Department of Biomedicine, University and University Hospital of Basel, Basel, Switzerland.

Marianne Böni-Schnetzler (M)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland.

Gottfried Rudofsky (G)

Endocrinology and Metabolic Diseases, Cantonal Hospital Olten, Olten, Switzerland.

Marc Y Donath (MY)

Clinic of Endocrinology, Diabetes and Metabolism University Hospital Basel, and Department of Biomedicine, University of Basel, Basel, Switzerland. Electronic address: marc.donath@usb.ch.

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Classifications MeSH