Periostin Plays a Key Role in Radioresistance of Head and Neck Cancer Cells


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
May 2020
Historique:
received: 11 03 2020
revised: 24 03 2020
accepted: 01 04 2020
entrez: 6 5 2020
pubmed: 6 5 2020
medline: 15 5 2020
Statut: ppublish

Résumé

Head and neck squamous cell carcinoma (HNSCC) is an aggressive head and neck malignancy. The aim of this study was to elucidate the role of periostin (POSTN) in the epithelial-to-mesenchymal transition (EMT) process mediating the acquisition of radioresistance in HNSCC. The expression levels of EMT hallmark genes including POSTN and Erk/Akt signaling pathways were compared between radiosensitive and radioresistant HNSCC cells. POSTN mRNA expression was higher in radioresistant HNSCC cells, and silencing POSTN significantly impaired their invasiveness under the effect of EMT process represented by up-regulation of mesenchymal markers and down-regulation of an epithelial marker. Expression levels of Erk and Akt were higher in radioresistant cells. POSTN in association with the Erk and Akt signaling pathways was up-regulated during the EMT process, leading to the conversion of radiosensitive to radioresistant HNSCC cells. POSTN may be a key marker for predicting the radioresistance and therapeutic target of HNSCC.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Head and neck squamous cell carcinoma (HNSCC) is an aggressive head and neck malignancy. The aim of this study was to elucidate the role of periostin (POSTN) in the epithelial-to-mesenchymal transition (EMT) process mediating the acquisition of radioresistance in HNSCC.
MATERIALS AND METHODS METHODS
The expression levels of EMT hallmark genes including POSTN and Erk/Akt signaling pathways were compared between radiosensitive and radioresistant HNSCC cells.
RESULTS RESULTS
POSTN mRNA expression was higher in radioresistant HNSCC cells, and silencing POSTN significantly impaired their invasiveness under the effect of EMT process represented by up-regulation of mesenchymal markers and down-regulation of an epithelial marker. Expression levels of Erk and Akt were higher in radioresistant cells.
CONCLUSION CONCLUSIONS
POSTN in association with the Erk and Akt signaling pathways was up-regulated during the EMT process, leading to the conversion of radiosensitive to radioresistant HNSCC cells. POSTN may be a key marker for predicting the radioresistance and therapeutic target of HNSCC.

Identifiants

pubmed: 32366407
pii: 40/5/2627
doi: 10.21873/anticanres.14233
doi:

Substances chimiques

Biomarkers, Tumor 0
Cell Adhesion Molecules 0
POSTN protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2627-2635

Informations de copyright

Copyright© 2020, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Jung Je Park (JJ)

Department of Otolaryngology, Institute of Health Sciences, College of Medicine, Gyeongsang National University, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Young-Sool Hah (YS)

Biomedical Research Institute, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Somi Ryu (S)

Department of Otolaryngology, Institute of Health Sciences, College of Medicine, Gyeongsang National University, Gyeongsang National University Hospital, Jinju, Republic of Korea.

So Young Cheon (SY)

Department of Otolaryngology, Institute of Health Sciences, College of Medicine, Gyeongsang National University, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Hee Young Cho (HY)

Biomedical Research Institute, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Jin Pyeong Kim (JP)

Department of Otolaryngology, Institute of Health Sciences, College of Medicine, Gyeongsang National University, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Seong Jun Won (SJ)

Department of Otolaryngology, Institute of Health Sciences, College of Medicine, Gyeongsang National University, Gyeongsang National University Hospital, Jinju, Republic of Korea.

Jong Sil Lee (JS)

Department of Pathology, Urology and Pediatrics, Gyeongsang National University, Jinju, Republic of Korea.

Jeong Seok Hwa (JS)

Department of Pathology, Urology and Pediatrics, Gyeongsang National University, Jinju, Republic of Korea.

Ji Hyun Seo (JH)

Department of Pathology, Urology and Pediatrics, Gyeongsang National University, Jinju, Republic of Korea.

Hyo Won Chang (HW)

Department of Otolaryngology, Asan Medical Center, College of Medicine, University of Ulsan, Seoul, Republic of Korea.

Sang Yoon Kim (SY)

Department of Otolaryngology, Asan Medical Center, College of Medicine, University of Ulsan, Seoul, Republic of Korea sykim2@amc.seoul.kr.

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Classifications MeSH