LRIG1 gene copy number analysis by ddPCR and correlations to clinical factors in breast cancer.


Journal

BMC cancer
ISSN: 1471-2407
Titre abrégé: BMC Cancer
Pays: England
ID NLM: 100967800

Informations de publication

Date de publication:
24 May 2020
Historique:
received: 13 12 2019
accepted: 30 04 2020
entrez: 26 5 2020
pubmed: 26 5 2020
medline: 3 2 2021
Statut: epublish

Résumé

Leucine-rich repeats and immunoglobulin-like domains 1 (LRIG1) copy number alterations and unbalanced gene recombination events have been reported to occur in breast cancer. Importantly, LRIG1 loss was recently shown to predict early and late relapse in stage I-II breast cancer. We developed droplet digital PCR (ddPCR) assays for the determination of relative LRIG1 copy numbers and used these assays to analyze LRIG1 in twelve healthy individuals, 34 breast tumor samples previously analyzed by fluorescence in situ hybridization (FISH), and 423 breast tumor cytosols. Four of the LRIG1/reference gene assays were found to be precise and robust, showing copy number ratios close to 1 (mean, 0.984; standard deviation, +/- 0.031) among the healthy control population. The correlation between the ddPCR assays and previous FISH results was low, possibly because of the different normalization strategies used. One in 34 breast tumors (2.9%) showed an unbalanced LRIG1 recombination event. LRIG1 copy number ratios were associated with the breast cancer subtype, steroid receptor status, ERBB2 status, tumor grade, and nodal status. Both LRIG1 loss and gain were associated with unfavorable metastasis-free survival; however, they did not remain significant prognostic factors after adjustment for common risk factors in the Cox regression analysis. Furthermore, LRIG1 loss was not significantly associated with survival in stage I and II cases. Although LRIG1 gene aberrations may be important determinants of breast cancer biology, and prognostic markers, the results of this study do not verify an important role for LRIG1 copy number analyses in predicting the risk of relapse in early-stage breast cancer.

Sections du résumé

BACKGROUND BACKGROUND
Leucine-rich repeats and immunoglobulin-like domains 1 (LRIG1) copy number alterations and unbalanced gene recombination events have been reported to occur in breast cancer. Importantly, LRIG1 loss was recently shown to predict early and late relapse in stage I-II breast cancer.
METHODS METHODS
We developed droplet digital PCR (ddPCR) assays for the determination of relative LRIG1 copy numbers and used these assays to analyze LRIG1 in twelve healthy individuals, 34 breast tumor samples previously analyzed by fluorescence in situ hybridization (FISH), and 423 breast tumor cytosols.
RESULTS RESULTS
Four of the LRIG1/reference gene assays were found to be precise and robust, showing copy number ratios close to 1 (mean, 0.984; standard deviation, +/- 0.031) among the healthy control population. The correlation between the ddPCR assays and previous FISH results was low, possibly because of the different normalization strategies used. One in 34 breast tumors (2.9%) showed an unbalanced LRIG1 recombination event. LRIG1 copy number ratios were associated with the breast cancer subtype, steroid receptor status, ERBB2 status, tumor grade, and nodal status. Both LRIG1 loss and gain were associated with unfavorable metastasis-free survival; however, they did not remain significant prognostic factors after adjustment for common risk factors in the Cox regression analysis. Furthermore, LRIG1 loss was not significantly associated with survival in stage I and II cases.
CONCLUSIONS CONCLUSIONS
Although LRIG1 gene aberrations may be important determinants of breast cancer biology, and prognostic markers, the results of this study do not verify an important role for LRIG1 copy number analyses in predicting the risk of relapse in early-stage breast cancer.

Identifiants

pubmed: 32448168
doi: 10.1186/s12885-020-06919-w
pii: 10.1186/s12885-020-06919-w
pmc: PMC7245921
doi:

Substances chimiques

Biomarkers, Tumor 0
LRIG1 protein, human 0
Membrane Glycoproteins 0
Receptor, ErbB-2 EC 2.7.10.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

459

Subventions

Organisme : Cancer Research Foundation in Northern Sweden
ID : LP 16 2139

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Auteurs

Mahmood Faraz (M)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Andreas Tellström (A)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Christina Edwinsdotter Ardnor (CE)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Kjell Grankvist (K)

Department of Medical Biosciences, Umeå University, SE-90187, Umeå, Sweden.

Lukasz Huminiecki (L)

National Bioinformatics Infrastructure Sweden, SciLifeLab, Uppsala, Sweden.
Current address: Instytut Genetyki i Hodowli Zwierząt Polskiej Akademii Nauk, ul. Postępu 36A, 05-552, Jastrzębiec, Magdalenka, Poland.

Björn Tavelin (B)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Roger Henriksson (R)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Håkan Hedman (H)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden.

Ingrid Ljuslinder (I)

Department of Radiation Sciences, Oncology, Umeå University, SE-90187, Umeå, Sweden. ingrid.ljuslinder@umu.se.

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Classifications MeSH