High disease activity status suggests more severe disease and damage accrual in systemic lupus erythematosus.


Journal

Lupus science & medicine
ISSN: 2053-8790
Titre abrégé: Lupus Sci Med
Pays: England
ID NLM: 101633705

Informations de publication

Date de publication:
05 2020
Historique:
received: 20 11 2019
revised: 19 04 2020
accepted: 24 04 2020
entrez: 30 5 2020
pubmed: 30 5 2020
medline: 11 5 2021
Statut: ppublish

Résumé

Disease severity in SLE is an important concept related to disease activity, treatment burden and prognosis. We set out to evaluate if high disease activity status (HDAS), based on ever attainment of a Systemic Lupus Erythematosus Disease Activity Index (SLEDAI) disease activity score of ≥10, is an indicator for disease severity in SLE. Using prospectively collected data, we assessed the association of HDAS with sociodemographic and disease characteristics and adverse clinical outcomes using logistic regression or generalised estimating equations. Of 286 patients with SLE, who were observed for a median (range) of 5.1 years (1-10.8 years), 43.7% experienced HDAS at least once during the observational period. Autoantibody positivity, particularly anti-dsDNA and anti-Sm positivity, were associated with increased likelihood of HDAS. Age ≥45 years at diagnosis was associated with reduced likelihood of HDAS (p=0.002). Patients with HDAS had higher Physician Global Assessment score (>1: OR 8.1, p<0.001) and were more likely to meet criteria for flare (mild/moderate flare: OR 4.4, p<0.001; severe flare: OR 17.2, p<0.001) at the time of experiencing HDAS. They were also more likely to have overall higher disease activity, as defined by time-adjusted mean SLEDAI-2K score in the highest quartile (OR 11.7, 95% CI 5.1 to 26.6; p>0.001), higher corticosteroid exposure (corticosteroid dose in highest quartile: OR 7.7, 95% CI 3.9 to 15.3; p<0.001) and damage accrual (OR 2.3, 95% CI 1.3 to 3.9; p=0.003) when compared with non-HDAS patients. HDAS is associated with more severe disease, as measured by higher disease activity across time, corticosteroid exposure and damage accrual. The occurrence of HDAS may be a useful prognostic marker in the management of SLE.

Identifiants

pubmed: 32467293
pii: 7/1/e000372
doi: 10.1136/lupus-2019-000372
pmc: PMC7259842
pii:
doi:

Substances chimiques

Adrenal Cortex Hormones 0
Autoantibodies 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

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Auteurs

Rachel Koelmeyer (R)

Monash Centre for Inflammatory Diseases, School of Clinical Sciences, Monash University, Clayton, Victoria, Australia.

Hieu Tri Nim (HT)

Faculty of Information Technology, Monash University, Clayton, Victoria, Australia.

Mandana Nikpour (M)

Department of Medicine, University of Melbourne, Fitzroy, Victoria, Australia.
Rheumatology, St Vincent Hospital Melbourne, Fitzroy, Victoria, Australia.

Ying B Sun (YB)

Global Evidence & Value Development, Merck Healthcare KGaA, Darmstadt, Germany.

Amy Kao (A)

Global Clinical Development, EMD Serono Research and Development Institute, Darmstadt, Germany.

Oliver Guenther (O)

Global Evidence & Value Development, Merck Healthcare KGaA, Darmstadt, Germany.

Eric Morand (E)

Monash Centre for Inflammatory Diseases, School of Clinical Sciences, Monash University, Clayton, Victoria, Australia.
Department of Rheumatology, Monash Health, Clayton, Victoria, Australia.

Alberta Hoi (A)

Monash Centre for Inflammatory Diseases, School of Clinical Sciences, Monash University, Clayton, Victoria, Australia alberta.hoi@monash.edu.
Department of Rheumatology, Monash Health, Clayton, Victoria, Australia.

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