Correlation of increased serum leucine-rich α2-glycoprotein levels with disease prognosis, progression, and activity of interstitial pneumonia in patients with dermatomyositis: A retrospective study.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2020
Historique:
received: 01 01 2020
accepted: 18 05 2020
entrez: 2 6 2020
pubmed: 2 6 2020
medline: 26 8 2020
Statut: epublish

Résumé

To investigate whether leucine-rich α2-glycoprotein (LRG) can be a biomarker for the disease activity, progression, and prognosis of interstitial pneumonia (IP) in patients with dermatomyositis (DM). Correlations between the clinical findings and serum LRG levels were investigated in 46 patients with DM-IP (33 with acute/subacute IP [A/SIP] and 13 patients with chronic IP [CIP], including 10 fatal cases of IP). The median serum LRG level of 18.4 (14.6-25.2) μg/mL in DM-IP patients was higher than that in healthy control subjects. The median levels of serum LRG at baseline and at 2 and 4 weeks after the initiation of treatment in the patients who died were significantly higher than those in the surviving patients (P = 0.026, 0.029, and 0.008, respectively). The median level of serum LRG in the DM-A/SIP patients was significantly higher than that in the DM-CIP patients (P = 0.0004), and that in the anti-MDA5-Ab-positive group was slightly higher than that in the anti-ARS-Ab-positive group. The serum LRG levels correlated significantly with the serum levels of LDH, C-reactive protein, ferritin, AaDO2, %DLco, and total ground-glass opacity score. The survival rate after 24 weeks in patients with an initial LRG level ≥ 17.6 μg/mL (survival rate: 40%) was significantly lower than that in patients with an initial LRG level < 17.6 μg/mL (100%) (P = 0.0009). The serum LRG level may be a promising marker of disease activity, progression, and prognosis in patients with DM-IP.

Identifiants

pubmed: 32479560
doi: 10.1371/journal.pone.0234090
pii: PONE-D-19-34203
pmc: PMC7263588
doi:

Substances chimiques

Autoantibodies 0
Biomarkers 0
Glycoproteins 0
LRG1 protein, human 0
C-Reactive Protein 9007-41-4
Ferritins 9007-73-2
IFIH1 protein, human EC 3.6.1.-
Interferon-Induced Helicase, IFIH1 EC 3.6.4.13

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0234090

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Takaaki Ishida (T)

Department of Internal Medicine (IV), Osaka Medical College, Takatsuki, Osaka, Japan.

Takuya Kotani (T)

Department of Internal Medicine (IV), Osaka Medical College, Takatsuki, Osaka, Japan.

Satoshi Serada (S)

Center for Intractable Immune Disease, Kochi Medical School, Kochi University, Nankoku, Kochi, Japan.

Minoru Fujimoto (M)

Center for Intractable Immune Disease, Kochi Medical School, Kochi University, Nankoku, Kochi, Japan.

Tohru Takeuchi (T)

Department of Internal Medicine (IV), Osaka Medical College, Takatsuki, Osaka, Japan.

Shigeki Makino (S)

Department of Internal Medicine (IV), Osaka Medical College, Takatsuki, Osaka, Japan.

Tetsuji Naka (T)

Center for Intractable Immune Disease, Kochi Medical School, Kochi University, Nankoku, Kochi, Japan.

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Classifications MeSH