IgG4 plasma cell myeloma: Clinicopathological characteristics and diagnosis.


Journal

Pathology international
ISSN: 1440-1827
Titre abrégé: Pathol Int
Pays: Australia
ID NLM: 9431380

Informations de publication

Date de publication:
Aug 2020
Historique:
received: 05 04 2020
revised: 14 05 2020
accepted: 15 05 2020
pubmed: 11 6 2020
medline: 16 6 2021
entrez: 11 6 2020
Statut: ppublish

Résumé

Plasma cell myeloma (PCM) is usually associated with the presence of M-protein in the serum and urine of patients, and about half of the PCM cases exhibit the IgG M-protein and increased gamma-globulin fraction on membrane electrophoresis. The IgG4 subclass is located in the beta-2 fraction on membrane electrophoresis. The aim of this study was to develop a method to evaluate IgG4-producing PCM (IgG4-PCM) and its clinicopathological characteristics. We found three cases of IgG4-PCM among 80 cases of IgG-producing PCM by membrane electrophoresis, which were confirmed by IgG4 immunostaining. None of the cases had a clinical history of IgG4-related disease, although they exhibited high levels of serum IgG4. A bone marrow aspiration specimen had an increased number of plasma cells with a relatively mature morphology. No cases exhibited lymphoplasmacytic inflammation, obliterative phlebitis or fibrosis. Immunohistochemistry revealed that tumor cells expressed CD138 and IgG4 and showed monoclonal expression of kappa. We revealed that IgG4-PCM might not be associated with IgG4-related disease and that the detection of M-protein with beta-globulin fraction by electrophoresis may be useful for screening IgG4-PCM.

Identifiants

pubmed: 32519464
doi: 10.1111/pin.12968
doi:

Substances chimiques

Immunoglobulin G 0
Syndecan-1 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

551-556

Informations de copyright

© 2020 Japanese Society of Pathology and John Wiley & Sons Australia, Ltd.

Références

Swerdlow SH, Campo E, Harris NL et al. WHO Classification of Tumours of Haematopoietic and Lymphoid Tissues, Revised 4th edn.; 241-50.
Dimopoulos MA, Sonneveld P, Leung N et al. International myeloma working group recommendations for the diagnosis and management of myeloma-related renal impairment. J Clin Oncol 2016; 34: 1544-57.
Hutchison CA, Batuman V, Behrens J et al. The pathogenesis and diagnosis of acute kidney injury in multiple myeloma. Nat Rev Nephrol 2012; 8: 43-51.
Stone JH, Zen Y, Deshpande V. Mechanisms of disease IgG4-related disease. N Engl J Med 2012; 366: 539-51.
Geyer JT, Niesvizky R, Jayabalan DS et al. IgG4 plasma cell myeloma: New insights into the pathogenesis of IgG4-related disease. Mod Pathol 2014; 27: 375-81.
Jacobs JFM, van der Molen RG, Karen DF. Relatively restricted migration of polyclonal IgG4 may mimic a monoclonal gammopathy in IgG4-related disease. Am J Clin Pathol 2014; 142: 76-81.
Foucar K, McKenna RW, Peterson LC, Kroft SH. Plasma cell neoplasms, Tumors of the bone marrow. AFIP Atlas of Tumor Pathology Series, Washington, DC: AmericanRegistry of Pathology; 2016; 24: 653-717.

Auteurs

Ai Ito (A)

Department of Pathology, Japanese Red Cross Nagoya First Hospital, Aichi, Japan.

Tatsuya Yamauchi (T)

Division of Hematology and Laboratory Medicine, Toki Municipal Hospital, Gifu, Japan.

Akinobu Nakano (A)

Division of Hematology and Laboratory Medicine, Toki Municipal Hospital, Gifu, Japan.

Masahiko Fujino (M)

Department of Pathology, Japanese Red Cross Nagoya First Hospital, Aichi, Japan.

Masafumi Ito (M)

Department of Pathology, Japanese Red Cross Nagoya First Hospital, Aichi, Japan.

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