Tumor Endothelial Cell-Mediated Antigen-Specific T-cell Suppression via the PD-1/PD-L1 Pathway.


Journal

Molecular cancer research : MCR
ISSN: 1557-3125
Titre abrégé: Mol Cancer Res
Pays: United States
ID NLM: 101150042

Informations de publication

Date de publication:
09 2020
Historique:
received: 30 08 2019
revised: 01 04 2020
accepted: 05 06 2020
pubmed: 13 6 2020
medline: 11 8 2021
entrez: 13 6 2020
Statut: ppublish

Résumé

Tumor endothelial cells (TEC) play multiple roles in the regional specialization of vascular structure and physiology. Because TECs in the tumor microenvironment come in contact with circulating immune cells, they might influence not only trafficking but also the antitumor cellular immune response. In a mouse tumor implantation model with B16 melanoma cells, TECs expressed MHC class II, costimulating molecules, and programmed death-ligand 1 (PD-L1), suggesting that they are antigen (Ag)-presenting cells with suppressive activity. Furthermore, TECs were able to take up and present tumor-derived ovalbumin (OVA) peptide on MHC class I molecules. In functional assays, B16-OVA tumor-derived TECs significantly suppressed the proliferation and Ag-specific cytotoxicity of OVA-specific CD8

Identifiants

pubmed: 32527950
pii: 1541-7786.MCR-19-0897
doi: 10.1158/1541-7786.MCR-19-0897
doi:

Substances chimiques

Antigens, Neoplasm 0
PDCD1 protein, human 0
Programmed Cell Death 1 Receptor 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1427-1440

Informations de copyright

©2020 American Association for Cancer Research.

Auteurs

Kazuhiro Taguchi (K)

Institute for Clinical Research, National Hospital Organization, Kure Medical Center/Chugoku Cancer Center, Kure City, Hiroshima, Japan.
Department of Gastroenterological and Transplant Surgery, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima City, Hiroshima, Japan.

Takashi Onoe (T)

Institute for Clinical Research, National Hospital Organization, Kure Medical Center/Chugoku Cancer Center, Kure City, Hiroshima, Japan. tonoemd@gmail.com.
Department of Gastroenterological and Transplant Surgery, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima City, Hiroshima, Japan.

Tomoaki Yoshida (T)

Institute for Clinical Research, National Hospital Organization, Kure Medical Center/Chugoku Cancer Center, Kure City, Hiroshima, Japan.

Yoshinori Yamashita (Y)

Institute for Clinical Research, National Hospital Organization, Kure Medical Center/Chugoku Cancer Center, Kure City, Hiroshima, Japan.

Yuka Tanaka (Y)

Department of Gastroenterological and Transplant Surgery, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima City, Hiroshima, Japan.

Hideki Ohdan (H)

Department of Gastroenterological and Transplant Surgery, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima City, Hiroshima, Japan.

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Classifications MeSH