Physiologic effects and functional outcome after treatment of dysfunctional right ventricular outflow tract in congenital heart disease using a two-stage intervention.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 Dec 2020
Historique:
received: 11 08 2019
revised: 15 04 2020
accepted: 14 06 2020
pubmed: 1 7 2020
medline: 15 5 2021
entrez: 29 6 2020
Statut: ppublish

Résumé

Pathophysiological differences in relief of pulmonary stenosis (PS) as opposed to pulmonary regurgitation (PR) by percutaneous pulmonary valve implantation (PPVI) remain elusive, but might impact current assessment of procedural success and ultimately indications. Invasive pressure measurements, cardiac magnetic resonance imaging and cardiopulmonary exercise testing were performed before pre-stenting (BMS), after BMS and after PPVI in patients with either PS or PR. In PS (n = 14), BMS reduced the right ventricular (RV) to systemic pressure ratio (0.8 ± 0.2 vs. 0.4 ± 0.1%; p < .01), improved RF EF (53 ± 14 vs. 59 ± 12%; p = .01) but introduced free PR (PR fraction post 39 ± 12%; p < .01) with no changes in effective RV stroke volume (SV). PPVI eliminated PR (PR fraction 5 ± 3%; p < .01) and improved effective RV SV (p < .01) with no changes in RV EF (p = .47). Peak VO2 improved significantly after BMS, with no changes following PPVI (26 ± 9 vs. 30 ± 11 vs. 31 ± 10 ml/kg*min). In PR (n = 14), BMS exaggerated PR (PR fraction post 47 ± 10) with reduction in effective RV SV (pre 43 ± 9 vs. post 38 ± 8 ml/m Exercise capacity in patients with right ventricular outflow tract dysfunction is primarily afterload-dependent.

Sections du résumé

BACKGROUND BACKGROUND
Pathophysiological differences in relief of pulmonary stenosis (PS) as opposed to pulmonary regurgitation (PR) by percutaneous pulmonary valve implantation (PPVI) remain elusive, but might impact current assessment of procedural success and ultimately indications.
METHODS METHODS
Invasive pressure measurements, cardiac magnetic resonance imaging and cardiopulmonary exercise testing were performed before pre-stenting (BMS), after BMS and after PPVI in patients with either PS or PR.
RESULTS RESULTS
In PS (n = 14), BMS reduced the right ventricular (RV) to systemic pressure ratio (0.8 ± 0.2 vs. 0.4 ± 0.1%; p < .01), improved RF EF (53 ± 14 vs. 59 ± 12%; p = .01) but introduced free PR (PR fraction post 39 ± 12%; p < .01) with no changes in effective RV stroke volume (SV). PPVI eliminated PR (PR fraction 5 ± 3%; p < .01) and improved effective RV SV (p < .01) with no changes in RV EF (p = .47). Peak VO2 improved significantly after BMS, with no changes following PPVI (26 ± 9 vs. 30 ± 11 vs. 31 ± 10 ml/kg*min). In PR (n = 14), BMS exaggerated PR (PR fraction post 47 ± 10) with reduction in effective RV SV (pre 43 ± 9 vs. post 38 ± 8 ml/m
CONCLUSIONS CONCLUSIONS
Exercise capacity in patients with right ventricular outflow tract dysfunction is primarily afterload-dependent.

Identifiants

pubmed: 32593726
pii: S0167-5273(20)33404-5
doi: 10.1016/j.ijcard.2020.06.026
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

69-74

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Tobias Kister (T)

Heart Center Leipzig at University Leipzig, Department of Internal Medicine/Cardiology, Germany.

Robert Wagner (R)

Heart Center Leipzig at University Leipzig, Department of Paediatric Cardiology, Germany.

Karl Philipp Rommel (KP)

Heart Center Leipzig at University Leipzig, Department of Internal Medicine/Cardiology, Germany.

Stephan Blazek (S)

Heart Center Leipzig at University Leipzig, Department of Internal Medicine/Cardiology, Germany.

Peter Kinzel (P)

Heart Center Leipzig at University Leipzig, Department of Paediatric Cardiology, Germany.

Matthias Grothoff (M)

Heart Center Leipzig at University Leipzig, Department of Radiology, Germany.

Matthias Gutberlet (M)

Heart Center Leipzig at University Leipzig, Department of Radiology, Germany.

Holger Thiele (H)

Heart Center Leipzig at University Leipzig, Department of Internal Medicine/Cardiology, Germany.

Ingo Dähnert (I)

Heart Center Leipzig at University Leipzig, Department of Paediatric Cardiology, Germany.

Frank-Thomas Riede (FT)

Heart Center Leipzig at University Leipzig, Department of Paediatric Cardiology, Germany.

Philipp Lurz (P)

Heart Center Leipzig at University Leipzig, Department of Internal Medicine/Cardiology, Germany. Electronic address: philipp.lurz@medizin.uni-leipzig.de.

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