A nonsense variant in the KRT14 gene in a domestic shorthair cat with epidermolysis bullosa simplex.

Felis catus dermatology genodermatosis keratin precision medicine skin whole genome sequencing

Journal

Animal genetics
ISSN: 1365-2052
Titre abrégé: Anim Genet
Pays: England
ID NLM: 8605704

Informations de publication

Date de publication:
Oct 2020
Historique:
accepted: 15 06 2020
pubmed: 14 7 2020
medline: 19 12 2020
entrez: 14 7 2020
Statut: ppublish

Résumé

Epidermolysis bullosa simplex (EBS) is a hereditary blistering disease affecting the skin and mucous membranes. It has been reported in humans, cattle, buffaloes and dogs, but so far not in cats. In humans, EBS is most frequently caused by variants in the KRT5 or KRT14 genes. Here, we report a case of feline epidermolysis bullosa simplex and describe the causative genetic variant. An 11-month-old male domestic shorthair cat presented with a history of sloughed paw pads and ulcerations in the oral cavity and inner aspect of the pinnae, starting a few weeks after birth. Clinical and histopathological findings suggested a congenital blistering disease with a split formation within the basal cell layer of the epidermis and oral mucous epithelium. The genetic investigation revealed a homozygous nonsense variant in the KRT14 gene (c.979C>T, p.Gln327*). Immunohistochemistry showed a complete absence of keratin 14 staining in all epithelia present in the biopsy. To the best of our knowledge, this is the first report of feline EBS, and the first report of a spontaneous pathogenic KRT14 variant in a non-human species. The homozygous genotype in the affected cat suggests an autosomal recessive mode of inheritance.

Identifiants

pubmed: 32657488
doi: 10.1111/age.12979
doi:

Substances chimiques

Codon, Nonsense 0
Keratin-14 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

829-832

Subventions

Organisme : Swiss National Science Foundation
ID : CRSII3_160738
Pays : Switzerland

Informations de copyright

© 2020 Stichting International Foundation for Animal Genetics.

Références

Alhaidari Z., Olivry T., Spadafora A., Thomas R.C., Perrin C., Meneguzzi G. & Ortonne J.P. (2005) Junctional epidermolysis bullosa in two domestic shorthair kittens. Veterinary Dermatology. 16, 69-73.
Batta K., Rugg E.L., Wilson N.J., West N., Goodyear H., Lane E.B., Gratian M., Dopping-Hepenstal P. & Moss C. (2000) A keratin 14 ‘knockout’ mutation in recessive epidermolysis bullosa simplex resulting in less severe disease. British Journal of Dermatology 143, 621-7.
Bolling M.C., Lemmink H.H., Jansen G.H.L. & Jonkman M.F. (2011) Mutations in KRT5 and KRT14 cause epidermolysis bullosa simplex in 75% of the patients. British Journal of Dermatology 164, 637-44.
Coulombe P.A., Kerns M.L. & Fuchs E. (2009) Epidermolysis bullosa simplex: a paradigm for disorders of tissue fragility. Journal of Clinical Investigation 119, 1784-93.
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Ford C.A., Stanfield A.M., Spelman R.J. et al. (2005) A mutation in bovine keratin 5 causing epidermolysis bullosa simplex, transmitted by a mosaic sire. Journal of Investigative Dermatology. 124, 1170-6.
Groves R.W., Liu L., Dopping-Hepenstal P.J. et al. (2010) A homozygous nonsense mutation within the dystonin gene coding for the coiled-coil domain of the epithelial isoform of BPAG1 underlies a new subtype of autosomal recessive epidermolysis bullosa simplex. Journal of Investigative Dermatology. 130, 1551-7.
Jagannathan V., Drögemüller C., Leeb T. et al. (2019) A comprehensive biomedical variant catalogue based on whole genome sequences of 582 dogs and eight wolves. Animal Genetics 50, 695-704.
Jonkman M.F., Pasmooij A.M.G., Pasmans S.G.M.A., van den Berg M.P., Ter Horst H.J., Timmer A. & Pas H.H. (2005) Loss of desmoplakin tail causes lethal acantholytic epidermolysis bullosa. The American Journal of Human Genetics. 77, 653-60.
Mauldin E.A., Wang P., Olivry T., Henthorn P.S. & Casal M.L. (2017) Epidermolysis bullosa simplex in sibling Eurasier dogs is caused by a PLEC non-sense variant. Veterinary Dermatology 28, 10-e3.
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Olivry T., Dunston S.M. & Marinkovich M.P. (1999) Reduced anchoring fibril formation and collagen VII immunoreactivity in feline dystrophic epidermolysis bullosa. Veterinary Pathology 36, 616-8.
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Auteurs

M Dettwiler (M)

Institute of Animal Pathology, Vetsuisse Faculty, University of Bern, Länggassstrasse 122, Bern, 3001, Switzerland.
Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.

F Leuthard (F)

Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.
Institute of Genetics, Vetsuisse Faculty, University of Bern, Bremgartenstrasse 109a, Bern, 3001, Switzerland.

A Bauer (A)

Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.
Institute of Genetics, Vetsuisse Faculty, University of Bern, Bremgartenstrasse 109a, Bern, 3001, Switzerland.

V Jagannathan (V)

Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.
Institute of Genetics, Vetsuisse Faculty, University of Bern, Bremgartenstrasse 109a, Bern, 3001, Switzerland.

A M Lourenço (AM)

Dermatology Service, Faculty of Veterinary Medicine, Centre for Interdisciplinary Research in Animal Health, University of Lisbon, Av. Universidade Técnica, Lisboa, 1300-477, Portugal.

H Pereira (H)

Dermatology Service, Faculty of Veterinary Medicine, Centre for Interdisciplinary Research in Animal Health, University of Lisbon, Av. Universidade Técnica, Lisboa, 1300-477, Portugal.

T Leeb (T)

Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.
Institute of Genetics, Vetsuisse Faculty, University of Bern, Bremgartenstrasse 109a, Bern, 3001, Switzerland.

M M Welle (MM)

Institute of Animal Pathology, Vetsuisse Faculty, University of Bern, Länggassstrasse 122, Bern, 3001, Switzerland.
Dermfocus, Vetsuisse Faculty, University of Bern, Bern, 3001, Switzerland.

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