In vivo assessment of potential for UGT-inhibition-based drug-drug interaction between sorafenib and tapentadol.


Journal

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
ISSN: 1950-6007
Titre abrégé: Biomed Pharmacother
Pays: France
ID NLM: 8213295

Informations de publication

Date de publication:
Oct 2020
Historique:
received: 11 05 2020
revised: 07 07 2020
accepted: 11 07 2020
pubmed: 28 7 2020
medline: 23 3 2021
entrez: 27 7 2020
Statut: ppublish

Résumé

Sorafenib (SR) is one of the most potent UGT (1A1, 1A9) inhibitors (in in vitro tests). The inhibition of UGT1A1 may cause hyperbilirubinaemia, whereas the inhibition of UGT1A9 and 1A1 may result in drug-drug interactions (DDIs). Tapentadol (TAP) is a synthetic μ-opioid agonist and is used to treat moderate to severe acute pain. Tapentadol is highly glucuronidated by the UGT1A9 and UGT2B7 isoenzymes. The aim of the study was to assess the DDI between SR and TAP. Wistar rats were divided into three groups, with eight animals in each. The rats were orally treated with SR (100 mg/kg) or TAP (4.64 mg/kg) or in combination with 100 mg/kg SOR and 4.64 TAP mg/kg. The concentrations of SR and sorafenib N-oxide, TAP and tapentadol glucuronide were respectively measured by means of high-performance liquid chromatography (HPLC) with ultraviolet detection and by means of ultra-performance liquid chromatography-tandem mass spectrometry. The co-administration of TAP with SR caused TAP maximum plasma concentration (C

Identifiants

pubmed: 32712531
pii: S0753-3322(20)30723-X
doi: 10.1016/j.biopha.2020.110530
pii:
doi:

Substances chimiques

Adrenergic Uptake Inhibitors 0
Antineoplastic Agents 0
Glucuronides 0
Ugt1a1 protein, rat 0
Sorafenib 9ZOQ3TZI87
Glucuronosyltransferase EC 2.4.1.17
Tapentadol H8A007M585

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

110530

Informations de copyright

Copyright © 2020. Published by Elsevier Masson SAS.

Auteurs

Agnieszka Karbownik (A)

Department of Clinical Pharmacy and Biopharmacy, Poznań University of Medical Sciences, 14 Św. Marii Magdaleny Str., 61-861, Poznań, Poland. Electronic address: agnieszkakarbownik@o2.pl.

Miłosz Miedziaszczyk (M)

Department of Clinical Pharmacy and Biopharmacy, Poznań University of Medical Sciences, 14 Św. Marii Magdaleny Str., 61-861, Poznań, Poland.

Tomasz Grabowski (T)

Polpharma Biologics SA, Trzy Lipy 3 Str., 80-172, Gdańsk, Poland.

Joanna Stanisławiak-Rudowicz (J)

Univeristy Hospital of Lord's Transfiguration, 84/86 Szamarzewskiego Str., 60-101, Poznań, Poland.

Radosław Jaźwiec (R)

Institute of Biochemistry and Biophysics PAS, Laboratory of Mas Spectromery, Polish Academy of Sciences, 5A Pawińskiego Str, 02-106, Warsaw, Poland.

Anna Wolc (A)

Department of Animal Science, Iowa State University, 239E Kildee Hall, Ames, IA, 50011, USA; Hy-Line International, 2583 240th Street, Dallas Center, IA, 50063, USA.

Edmund Grześkowiak (E)

Department of Clinical Pharmacy and Biopharmacy, Poznań University of Medical Sciences, 14 Św. Marii Magdaleny Str., 61-861, Poznań, Poland.

Edyta Szałek (E)

Department of Clinical Pharmacy and Biopharmacy, Poznań University of Medical Sciences, 14 Św. Marii Magdaleny Str., 61-861, Poznań, Poland.

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Classifications MeSH