Mechanisms of activation and desensitization of full-length glycine receptor in lipid nanodiscs.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
27 07 2020
Historique:
received: 07 02 2020
accepted: 25 06 2020
entrez: 29 7 2020
pubmed: 29 7 2020
medline: 9 9 2020
Statut: epublish

Résumé

Glycinergic synapses play a central role in motor control and pain processing in the central nervous system. Glycine receptors (GlyRs) are key players in mediating fast inhibitory neurotransmission at these synapses. While previous high-resolution structures have provided insights into the molecular architecture of GlyR, several mechanistic questions pertaining to channel function are still unanswered. Here, we present Cryo-EM structures of the full-length GlyR protein complex reconstituted into lipid nanodiscs that are captured in the unliganded (closed), glycine-bound (open and desensitized), and allosteric modulator-bound conformations. A comparison of these states reveals global conformational changes underlying GlyR channel gating and modulation. The functional state assignments were validated by molecular dynamics simulations, and the observed permeation events are in agreement with the anion selectivity and conductance of GlyR. These studies provide the structural basis for gating, ion selectivity, and single-channel conductance properties of GlyR in a lipid environment.

Identifiants

pubmed: 32719334
doi: 10.1038/s41467-020-17364-5
pii: 10.1038/s41467-020-17364-5
pmc: PMC7385131
doi:

Substances chimiques

Lipids 0
Neurotransmitter Agents 0
Receptors, Glycine 0
Zebrafish Proteins 0
glra1 protein, zebrafish 0
Glycine TE7660XO1C

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3752

Subventions

Organisme : NIH HHS
ID : S10 OD021600
Pays : United States
Organisme : NIGMS NIH HHS
ID : R35 GM134896
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM108921
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM131216
Pays : United States
Organisme : NIGMS NIH HHS
ID : P41 GM103832
Pays : United States

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Auteurs

Arvind Kumar (A)

Department of Physiology and Biophysics, Case Western Reserve University, Cleveland, OH, 44106-4970, USA.

Sandip Basak (S)

Department of Physiology and Biophysics, Case Western Reserve University, Cleveland, OH, 44106-4970, USA.

Shanlin Rao (S)

Department of Biochemistry, University of Oxford, Oxford, OX1 3QU, UK.

Yvonne Gicheru (Y)

Department of Physiology and Biophysics, Case Western Reserve University, Cleveland, OH, 44106-4970, USA.

Megan L Mayer (ML)

Division of CryoEM and Bioimaging, SSRL, SLAC National Accelerator Laboratory, Stanford University, Menlo Park, CA, 94025, USA.

Mark S P Sansom (MSP)

Department of Biochemistry, University of Oxford, Oxford, OX1 3QU, UK.

Sudha Chakrapani (S)

Department of Physiology and Biophysics, Case Western Reserve University, Cleveland, OH, 44106-4970, USA. Sudha.chakrapani@case.edu.
Department of Neuroscience, School of Medicine, Case Western Reserve University, Cleveland, OH, 44106-4970, USA. Sudha.chakrapani@case.edu.

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Classifications MeSH