Use of Sodium-Glucose Co-Transporter-2-Inhibitors (SGLT2-Is) and Risk of Lower Limb Amputation.


Journal

Current drug safety
ISSN: 2212-3911
Titre abrégé: Curr Drug Saf
Pays: United Arab Emirates
ID NLM: 101270895

Informations de publication

Date de publication:
2021
Historique:
received: 02 04 2020
revised: 03 06 2020
accepted: 16 06 2020
pubmed: 10 8 2020
medline: 26 10 2021
entrez: 10 8 2020
Statut: ppublish

Résumé

Treatment with sodium-glucose co-transporter-2-inhibitors (SGLT2-Is), such as canagliflozin, has been associated with an increased risk of lower limb amputations (LLAs) in type 2 diabetes mellitus (T2DM). However, conflicting results have been reported for different SGLT2-Is and the underlying mechanism is unclear. To investigate the risk of LLA and diabetic foot ulcer with SGLT2-I use compared to other anti-diabetic drugs and to explore hypovolemia as a potential underlying mechanism. A cohort study was conducted using data from the Clinical Practice Research Datalink GOLD (2013-2019). The study population (N=51,847) consisted of T2DM patients over 18 years of age with at least one prescription of a non-insulin anti-diabetic drug. Concomitant diuretic use and the presence of signs of hypovolemia were determined to assess the potential underlying mechanism. Cox proportional hazard models were used to estimate the hazard ratio (HR) for LLA in current SGLT2-I use versus current sulphonylurea (SU) use. Analyses were adjusted for lifestyle variables, comorbidities, and concomitant drug use. Current SGLT2-I use was not associated with an increased risk of LLA compared to current SU use (fully adjusted HR 0.70; 95% confidence interval 0.38-1.29). Concomitant use of diuretics and the presence of signs of hypovolemia were not associated with an increased risk of LLA. Use of SGLT2-Is, with or without signs of hypovolemia, was not associated with an increased risk of LLA or DFU versus current SU use. Future studies powered to detect potential differences between individual SGLT2-Is are required to rule out a canagliflozin-specific effect.

Sections du résumé

BACKGROUND BACKGROUND
Treatment with sodium-glucose co-transporter-2-inhibitors (SGLT2-Is), such as canagliflozin, has been associated with an increased risk of lower limb amputations (LLAs) in type 2 diabetes mellitus (T2DM). However, conflicting results have been reported for different SGLT2-Is and the underlying mechanism is unclear.
OBJECTIVE OBJECTIVE
To investigate the risk of LLA and diabetic foot ulcer with SGLT2-I use compared to other anti-diabetic drugs and to explore hypovolemia as a potential underlying mechanism.
METHODS METHODS
A cohort study was conducted using data from the Clinical Practice Research Datalink GOLD (2013-2019). The study population (N=51,847) consisted of T2DM patients over 18 years of age with at least one prescription of a non-insulin anti-diabetic drug. Concomitant diuretic use and the presence of signs of hypovolemia were determined to assess the potential underlying mechanism. Cox proportional hazard models were used to estimate the hazard ratio (HR) for LLA in current SGLT2-I use versus current sulphonylurea (SU) use. Analyses were adjusted for lifestyle variables, comorbidities, and concomitant drug use.
RESULTS RESULTS
Current SGLT2-I use was not associated with an increased risk of LLA compared to current SU use (fully adjusted HR 0.70; 95% confidence interval 0.38-1.29). Concomitant use of diuretics and the presence of signs of hypovolemia were not associated with an increased risk of LLA.
CONCLUSION CONCLUSIONS
Use of SGLT2-Is, with or without signs of hypovolemia, was not associated with an increased risk of LLA or DFU versus current SU use. Future studies powered to detect potential differences between individual SGLT2-Is are required to rule out a canagliflozin-specific effect.

Identifiants

pubmed: 32767909
pii: CDS-EPUB-108862
doi: 10.2174/1574886315666200805103053
doi:

Substances chimiques

Hypoglycemic Agents 0
Sodium-Glucose Transporter 2 0
Sodium-Glucose Transporter 2 Inhibitors 0
Sodium 9NEZ333N27
Glucose IY9XDZ35W2

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

62-72

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Nikki C C Werkman (NCC)

Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, Netherlands.

Johannes T H Nielen (JTH)

Department of Clinical Pharmacy and Toxicology, Maastricht University Medical Center+, Maastricht, Netherlands.

Joop P W van den Bergh (JPW)

School for Nutrition and Translational Research in Metabolism (NUTRIM), Maastricht University, Maastricht, Netherlands.

Niels Ejskjaer (N)

Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Johan Røikjer (J)

Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Nicolaas C Schaper (NC)

Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, Netherlands.

Bernardette Rossi (B)

Directorate Pharmaceutical Affairs, Department for Policy in Health, Ministry for Health, Valletta, Malta.

Olaf Klungel (O)

Division of Pharmacoepidemiology and Clinical Pharmacology, Utrecht Institute of Pharmaceutical Sciences, Utrecht, Netherlands.

Peter Vestergaard (P)

Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Frank de Vries (F)

Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, Netherlands.

Johanna H M Driessen (JHM)

Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, Netherlands.

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Classifications MeSH