Pharmacological interventions for the prevention of renal injury in surgical patients: a systematic literature review and meta-analysis.


Journal

British journal of anaesthesia
ISSN: 1471-6771
Titre abrégé: Br J Anaesth
Pays: England
ID NLM: 0372541

Informations de publication

Date de publication:
01 2021
Historique:
received: 23 03 2020
revised: 20 05 2020
accepted: 19 06 2020
pubmed: 24 8 2020
medline: 29 1 2021
entrez: 24 8 2020
Statut: ppublish

Résumé

The aim of this systematic review was to summarise the results of randomised controlled trials (RCTs) that have evaluated pharmacological interventions for renoprotection in people undergoing surgery. Searches were conducted to update a previous review using the Cochrane Central Register of Controlled Trials, MEDLINE, and EMBASE to August 23, 2019. RCTs evaluating the use of pharmacological interventions for renal protection in the perioperative period were included. The co-primary outcome measures were 30-day mortality and acute kidney injury (AKI). Pooled effect estimates were expressed as risk ratios (RRs) (95% confidence intervals). We included 228 trials enrolling 56 047 patients. Twenty-three trials were considered to be at low risk of bias across all domains. Atrial natriuretic peptides (14 trials; n=2207) reduced 30-day mortality (RR: 0.63 [0.41, 0.97]) and AKI events (RR: 0.43 [0.33, 0.56]) without heterogeneity. These effects were consistent across cardiac surgery and vascular surgery subgroups, and in sensitivity analyses restricted to studies at low risk of bias. Inodilators (13 trials; n=2941) reduced mortality (RR: 0.71 [0.53, 0.94]) and AKI events (RR: 0.65 [0.50, 0.85]) in the primary analysis and in cardiac surgery cohorts. Vasopressors (4 trials; n=1047) reduced AKI (RR: 0.56 [0.36, 0.86]). Nitric oxide donors, alpha-2-agonists, and calcium channel blockers reduced AKI in primary analyses, but not after exclusion of studies at risk of bias. Overall, assessment of the certainty of the effect estimates was low. There are multiple effective pharmacological renoprotective interventions for people undergoing surgery.

Sections du résumé

BACKGROUND
The aim of this systematic review was to summarise the results of randomised controlled trials (RCTs) that have evaluated pharmacological interventions for renoprotection in people undergoing surgery.
METHODS
Searches were conducted to update a previous review using the Cochrane Central Register of Controlled Trials, MEDLINE, and EMBASE to August 23, 2019. RCTs evaluating the use of pharmacological interventions for renal protection in the perioperative period were included. The co-primary outcome measures were 30-day mortality and acute kidney injury (AKI). Pooled effect estimates were expressed as risk ratios (RRs) (95% confidence intervals).
RESULTS
We included 228 trials enrolling 56 047 patients. Twenty-three trials were considered to be at low risk of bias across all domains. Atrial natriuretic peptides (14 trials; n=2207) reduced 30-day mortality (RR: 0.63 [0.41, 0.97]) and AKI events (RR: 0.43 [0.33, 0.56]) without heterogeneity. These effects were consistent across cardiac surgery and vascular surgery subgroups, and in sensitivity analyses restricted to studies at low risk of bias. Inodilators (13 trials; n=2941) reduced mortality (RR: 0.71 [0.53, 0.94]) and AKI events (RR: 0.65 [0.50, 0.85]) in the primary analysis and in cardiac surgery cohorts. Vasopressors (4 trials; n=1047) reduced AKI (RR: 0.56 [0.36, 0.86]). Nitric oxide donors, alpha-2-agonists, and calcium channel blockers reduced AKI in primary analyses, but not after exclusion of studies at risk of bias. Overall, assessment of the certainty of the effect estimates was low.
CONCLUSIONS
There are multiple effective pharmacological renoprotective interventions for people undergoing surgery.

Identifiants

pubmed: 32828488
pii: S0007-0912(20)30567-5
doi: 10.1016/j.bja.2020.06.064
pmc: PMC7844346
pii:
doi:

Substances chimiques

Adrenergic alpha-2 Receptor Agonists 0
Calcium Channel Blockers 0
Nitric Oxide Donors 0
Vasoconstrictor Agents 0
Atrial Natriuretic Factor 85637-73-6

Types de publication

Journal Article Meta-Analysis Research Support, Non-U.S. Gov't Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

131-138

Subventions

Organisme : British Heart Foundation
ID : RG/13/6/29947
Pays : United Kingdom

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Ltd.. All rights reserved.

Références

Am J Respir Crit Care Med. 2018 Nov 15;198(10):1279-1287
pubmed: 29932345
Br J Clin Pharmacol. 2017 Apr;83(4):709-720
pubmed: 27779776
BMJ. 2008 Apr 26;336(7650):924-6
pubmed: 18436948
Cochrane Database Syst Rev. 2013 Sep 11;(9):CD003590
pubmed: 24027097
Heart Fail Rev. 2011 Nov;16(6):553-67
pubmed: 21400231
BMJ. 2008 May 10;336(7652):1049-51
pubmed: 18467413
Eur Heart J. 2009 Aug;30(15):1910-7
pubmed: 19282300
BMJ. 2009 Jul 21;339:b2535
pubmed: 19622551

Auteurs

Suraj Pathak (S)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK. Electronic address: sp633@le.ac.uk.

Guido Olivieri (G)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Walid Mohamed (W)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Riccardo Abbasciano (R)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Marius Roman (M)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Sara Tomassini (S)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Florence Lai (F)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Marcin Wozniak (M)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

Gavin J Murphy (GJ)

Department of Cardiovascular Sciences, National Institute for Health Research Leicester Biomedical Research Unit in Cardiovascular Medicine, University of Leicester, Leicester, UK.

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Classifications MeSH