Impact of plasma potassium normalization on short-term mortality in patients with hypertension and hypokalemia or low normal potassium.


Journal

BMC cardiovascular disorders
ISSN: 1471-2261
Titre abrégé: BMC Cardiovasc Disord
Pays: England
ID NLM: 100968539

Informations de publication

Date de publication:
24 08 2020
Historique:
received: 16 04 2020
accepted: 04 08 2020
entrez: 26 8 2020
pubmed: 26 8 2020
medline: 12 1 2021
Statut: epublish

Résumé

Hypokalemia is common in patients treated with antihypertensive drugs, but the impact of correcting hypokalemia is insufficiently studied. We examined the consequences of hypokalemia and borderline hypokalemia correction in patients with hypertension. We identified 8976 patients with hypertension and plasma potassium concentrations ≤3.7 mmol/L within 100 days from combination antihypertensive therapy initiation. The first measurement between 6 and 100 days after the episode with potassium ≤3.7 mmol/L was retained. We investigated all-cause and cardiovascular mortality within 60-days from the second potassium measurement using Cox regression. Mortality was examined for seven predefined potassium intervals derived from the second measurement: 1.5-2.9 mmol/L (n = 271), 3.0-3.4 mmol/L (n = 1341), 3.5-3.7 (n = 1982) mmol/L, 3.8-4.0 mmol/L (n = 2398, reference), 4.1-4.6 mmol/L (n = 2498), 4.7-5.0 mmol/L (n = 352) and 5.1-7.1 mmol/L (n = 134). Multivariable analysis showed that potassium concentrations 1.5-2.9 mmol/L, 3.0-3.4 mmol/L, 4.7-5.0 mmol/L and 5.1-7.1 mmol/L were associated with increased all-cause mortality (HR 2.39, 95% CI 1.66-3.43; HR 1.36, 95% CI 1.04-1.78; HR 2.36, 95% CI 1.68-3.30 and HR 2.62, 95% CI 1.73-3.98, respectively). Potassium levels <3.0 and > 4.6 mmol/L were associated with increased cardiovascular mortality. The adjusted standardized 60-day mortality risks in the seven strata were: 11.7% (95% CI 8.3-15.0%), 7.1% (95% CI 5.8-8.5%), 6.4% (95% CI 5.3-7.5%), 5.4% (4.5-6.3%), 6.3% (5.4-7.2%), 11.6% (95% CI 8.7-14.6%) and 12.6% (95% CI 8.2-16.9%), respectively. Persistent hypokalemia was frequent and associated with increased all-cause and cardiovascular mortality. Increase in potassium to levels > 4.6 mmol/L in patients with initial hypokalemia or low normal potassium was associated with increased all-cause and cardiovascular mortality.

Sections du résumé

BACKGROUND
Hypokalemia is common in patients treated with antihypertensive drugs, but the impact of correcting hypokalemia is insufficiently studied. We examined the consequences of hypokalemia and borderline hypokalemia correction in patients with hypertension.
METHODS
We identified 8976 patients with hypertension and plasma potassium concentrations ≤3.7 mmol/L within 100 days from combination antihypertensive therapy initiation. The first measurement between 6 and 100 days after the episode with potassium ≤3.7 mmol/L was retained. We investigated all-cause and cardiovascular mortality within 60-days from the second potassium measurement using Cox regression. Mortality was examined for seven predefined potassium intervals derived from the second measurement: 1.5-2.9 mmol/L (n = 271), 3.0-3.4 mmol/L (n = 1341), 3.5-3.7 (n = 1982) mmol/L, 3.8-4.0 mmol/L (n = 2398, reference), 4.1-4.6 mmol/L (n = 2498), 4.7-5.0 mmol/L (n = 352) and 5.1-7.1 mmol/L (n = 134).
RESULTS
Multivariable analysis showed that potassium concentrations 1.5-2.9 mmol/L, 3.0-3.4 mmol/L, 4.7-5.0 mmol/L and 5.1-7.1 mmol/L were associated with increased all-cause mortality (HR 2.39, 95% CI 1.66-3.43; HR 1.36, 95% CI 1.04-1.78; HR 2.36, 95% CI 1.68-3.30 and HR 2.62, 95% CI 1.73-3.98, respectively). Potassium levels <3.0 and > 4.6 mmol/L were associated with increased cardiovascular mortality. The adjusted standardized 60-day mortality risks in the seven strata were: 11.7% (95% CI 8.3-15.0%), 7.1% (95% CI 5.8-8.5%), 6.4% (95% CI 5.3-7.5%), 5.4% (4.5-6.3%), 6.3% (5.4-7.2%), 11.6% (95% CI 8.7-14.6%) and 12.6% (95% CI 8.2-16.9%), respectively.
CONCLUSIONS
Persistent hypokalemia was frequent and associated with increased all-cause and cardiovascular mortality. Increase in potassium to levels > 4.6 mmol/L in patients with initial hypokalemia or low normal potassium was associated with increased all-cause and cardiovascular mortality.

Identifiants

pubmed: 32838735
doi: 10.1186/s12872-020-01654-3
pii: 10.1186/s12872-020-01654-3
pmc: PMC7446172
doi:

Substances chimiques

Antihypertensive Agents 0
Biomarkers 0
Potassium RWP5GA015D

Types de publication

Journal Article Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

386

Références

Eur Heart J. 2017 Jan 7;38(2):104-112
pubmed: 28158516
Crit Care. 2019 Dec 19;23(1):415
pubmed: 31856891
Adv Med. 2019 Sep 24;2019:4919707
pubmed: 31663004
Arch Intern Med. 2000 Sep 11;160(16):2429-36
pubmed: 10979053
Scand J Public Health. 2011 Jul;39(7 Suppl):12-6
pubmed: 21898916
Am J Med. 2018 Mar;131(3):318.e9-318.e19
pubmed: 29024624
BMJ. 2011 Jan 31;342:d124
pubmed: 21282258
J Clin Hypertens (Greenwich). 2011 Sep;13(9):639-43
pubmed: 21896142
Drug Intell Clin Pharm. 1986 May;20(5):370-1
pubmed: 3709348
Eur Heart J Cardiovasc Pharmacother. 2020 Jul 1;6(3):137-144
pubmed: 31263883
P T. 2015 Mar;40(3):185-90
pubmed: 25798039
Hypertension. 2012 May;59(5):926-33
pubmed: 22431578
Expert Opin Pharmacother. 2014 Apr;15(5):605-21
pubmed: 24456327
Ann Intern Med. 2009 May 5;150(9):604-12
pubmed: 19414839
Eur Heart J. 2017 Oct 7;38(38):2890-2896
pubmed: 29019614
J Hypertens. 2014 Oct;32(10):2092-7; discussion 2097
pubmed: 25105457
Scand J Public Health. 2011 Jul;39(7 Suppl):22-5
pubmed: 21775345
Eur Heart J Cardiovasc Pharmacother. 2015 Oct;1(4):245-51
pubmed: 27418967

Auteurs

Maria Lukács Krogager (ML)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark. lkcsmaria@yahoo.com.

Peter Søgaard (P)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.

Christian Torp-Pedersen (C)

Department of Cardiology and Clinical Research, Nordsjællands Hospital, Hillerød, Denmark.

Henrik Bøggild (H)

Public Health and Epidemiology Group, Department of Health Science and Technology, Aalborg University, Aalborg, Denmark.
Unit of Epidemiology and Biostatistics, Aalborg University Hospital, Aalborg, Denmark.

Christina Ji-Young Lee (CJ)

Department of Cardiology and Clinical Research, Nordsjællands Hospital, Hillerød, Denmark.
Department of Cardiology, Copenhagen University Hospital, Herlev and Gentofte, Hellerup, Denmark.

Anders Bonde (A)

Department of Cardiology, Copenhagen University Hospital, Herlev and Gentofte, Hellerup, Denmark.

Jesper Q Thomassen (JQ)

Department of Clinical Biochemistry, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.

Gunnar Gislason (G)

Department of Cardiology, Herlev and Gentofte University Hospital, Hellerup, Denmark.
The Danish Heart Foundation, Copenhagen, Denmark.
The National Institute of Public Health, University of Southern Denmark, Copenhagen, Denmark.

Manan Pareek (M)

Department of Cardiology and Clinical Research, Nordsjællands Hospital, Hillerød, Denmark.
Department of Internal Medicine, Yale New Haven Hospital, Yale University School of Medicine, New Haven, USA.
Brigham and Women's Hospital, Heart & Vascular Center, Harvard Medical School, Boston, USA.

Kristian Kragholm (K)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.
Unit of Epidemiology and Biostatistics, Aalborg University Hospital, Aalborg, Denmark.
Department of Cardiology, Regionshospital Nordjylland, Hjørring, Denmark.

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Classifications MeSH