Concomitant changes in ventricular depolarization and repolarization and long-term outcomes of biventricular pacing.


Journal

Pacing and clinical electrophysiology : PACE
ISSN: 1540-8159
Titre abrégé: Pacing Clin Electrophysiol
Pays: United States
ID NLM: 7803944

Informations de publication

Date de publication:
11 2020
Historique:
received: 20 05 2020
revised: 20 08 2020
accepted: 06 09 2020
pubmed: 10 9 2020
medline: 14 10 2021
entrez: 9 9 2020
Statut: ppublish

Résumé

Biventricular (BiV) pacing increases transmural repolarization heterogeneity due to epicardial to endocardial conduction from the left ventricular (LV) lead. However, limited evidence is available on concomitant changes in ventricular depolarization and repolarization and long-term outcomes of BiV pacing. Therefore, we investigated associations of BiV pacing-induced concomitant changes in ventricular depolarization and repolarization with mortality (i.e., LV assist device, heart transplantation, or all-cause mortality) and sustained ventricular arrhythmia endpoints. Consecutive BiV-defibrillator recipients with digital preimplantation and postimplantation electrocardiograms recorded between 2006 and 2015 at Duke University Medical Center were included. We calculated changes in QRS duration and corrected JT (JTc) interval and split them by median values. For simplicity, these variables were named QRS We included 528 patients (median age, 68 years; male, 69%). No correlation between changes in QRS duration and JTc interval was observed (P = .295). Compared to QRS Among BiV-defibrillator recipients, QRS

Sections du résumé

BACKGROUND
Biventricular (BiV) pacing increases transmural repolarization heterogeneity due to epicardial to endocardial conduction from the left ventricular (LV) lead. However, limited evidence is available on concomitant changes in ventricular depolarization and repolarization and long-term outcomes of BiV pacing. Therefore, we investigated associations of BiV pacing-induced concomitant changes in ventricular depolarization and repolarization with mortality (i.e., LV assist device, heart transplantation, or all-cause mortality) and sustained ventricular arrhythmia endpoints.
METHODS
Consecutive BiV-defibrillator recipients with digital preimplantation and postimplantation electrocardiograms recorded between 2006 and 2015 at Duke University Medical Center were included. We calculated changes in QRS duration and corrected JT (JTc) interval and split them by median values. For simplicity, these variables were named QRS
RESULTS
We included 528 patients (median age, 68 years; male, 69%). No correlation between changes in QRS duration and JTc interval was observed (P = .295). Compared to QRS
CONCLUSION
Among BiV-defibrillator recipients, QRS

Identifiants

pubmed: 32901967
doi: 10.1111/pace.14065
pmc: PMC7669630
mid: NIHMS1635070
doi:

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1333-1343

Subventions

Organisme : NHLBI NIH HHS
ID : T32 HL069749
Pays : United States

Informations de copyright

© 2020 Wiley Periodicals LLC.

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Auteurs

Christoffer Polcwiartek (C)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.
Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.
Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Daniel J Friedman (DJ)

Section of Cardiac Electrophysiology, Yale School of Medicine, New Haven, Connecticut.

Kasper Emerek (K)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.

Claus Graff (C)

Department of Health Science and Technology, Aalborg University, Aalborg, Denmark.

Peter L Sørensen (PL)

Department of Health Science and Technology, Aalborg University, Aalborg, Denmark.

Joseph Kisslo (J)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.

Zak Loring (Z)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.
Duke Clinical Research Institute, Durham, North Carolina.

Steen M Hansen (SM)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.

Kristian Kragholm (K)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.

Bhupendar Tayal (B)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.

Svend E Jensen (SE)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.
Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Peter Søgaard (P)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.
Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Christian Torp-Pedersen (C)

Department of Cardiology, Aalborg University Hospital, Aalborg, Denmark.
Department of Cardiology and Clinical Research, Nordsjaellands Hospital, Hillerød, Denmark.

Brett D Atwater (BD)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.

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