D variants in the population of D-negative blood donors in the north-eastern region of Croatia.


Journal

Transfusion medicine (Oxford, England)
ISSN: 1365-3148
Titre abrégé: Transfus Med
Pays: England
ID NLM: 9301182

Informations de publication

Date de publication:
Feb 2021
Historique:
received: 30 04 2020
revised: 16 09 2020
accepted: 22 09 2020
pubmed: 10 10 2020
medline: 3 11 2021
entrez: 9 10 2020
Statut: ppublish

Résumé

The aim of this study was to determine RHESUS D GENE (RHD) allelic variants among Croatian D-negative blood donors and compare our results with respective data from other European countries. Altered or reduced D antigen expression can result in D variants, which can be mistyped and can lead to the alloimmunisation of the blood recipient. RHD genotyping can distinguish D variants: weak D, partial D and DEL, thus preventing alloimmunisation. A total of 6523 samples obtained from D-negative Croatian donors were screened for the presence of RHD using the real-time polymerase chain reaction (PCR) method. PCR-SSP was performed for D variant genotyping by using commercial genotyping kits (Inno-Train, Kronberg, Germany). Genomic DNA sequencing for all 10 exons of the RHD was performed when the genotyping kits failed to assign a D variant. RHD molecular screening revealed 23 (0.35%) RHD-PCR positive samples, all C/E positive, in decreasing frequency: 11 hybrid RHD-CE (2-9) D-CE variants, 4 weak partial D type 11 and 2 weak D type 2. Six samples remained unresolved and were sequenced. For 12 of 23 samples (excluding large hybrids), an adsorption/elution of anti-D serum was performed, confirming that all 12 were RhD+. The calculated frequency of clinically significant D alleles in RhD-negative blood donors was 1:543 (0.18%) or 1:53 (1.89%) in C/E blood donors. Data on the significant frequency of D variants among serologically D-negative blood donors in the north-eastern region of Croatia could help in introducing RHD molecular screening of blood donors in a routine workflow.

Sections du résumé

OBJECTIVES OBJECTIVE
The aim of this study was to determine RHESUS D GENE (RHD) allelic variants among Croatian D-negative blood donors and compare our results with respective data from other European countries.
BACKGROUND BACKGROUND
Altered or reduced D antigen expression can result in D variants, which can be mistyped and can lead to the alloimmunisation of the blood recipient. RHD genotyping can distinguish D variants: weak D, partial D and DEL, thus preventing alloimmunisation.
MATERIAL/METHODS METHODS
A total of 6523 samples obtained from D-negative Croatian donors were screened for the presence of RHD using the real-time polymerase chain reaction (PCR) method. PCR-SSP was performed for D variant genotyping by using commercial genotyping kits (Inno-Train, Kronberg, Germany). Genomic DNA sequencing for all 10 exons of the RHD was performed when the genotyping kits failed to assign a D variant.
RESULTS RESULTS
RHD molecular screening revealed 23 (0.35%) RHD-PCR positive samples, all C/E positive, in decreasing frequency: 11 hybrid RHD-CE (2-9) D-CE variants, 4 weak partial D type 11 and 2 weak D type 2. Six samples remained unresolved and were sequenced. For 12 of 23 samples (excluding large hybrids), an adsorption/elution of anti-D serum was performed, confirming that all 12 were RhD+. The calculated frequency of clinically significant D alleles in RhD-negative blood donors was 1:543 (0.18%) or 1:53 (1.89%) in C/E blood donors.
CONCLUSION CONCLUSIONS
Data on the significant frequency of D variants among serologically D-negative blood donors in the north-eastern region of Croatia could help in introducing RHD molecular screening of blood donors in a routine workflow.

Identifiants

pubmed: 33034130
doi: 10.1111/tme.12726
doi:

Substances chimiques

Rh-Hr Blood-Group System 0
Rho(D) antigen 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

43-47

Informations de copyright

© 2020 British Blood Transfusion Society.

Références

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Auteurs

Hana Safic Stanic (H)

Department of Molecular Diagnostics, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Vesna Dogic (V)

Department of Molecular Diagnostics, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Ivona Herceg (I)

Department of Molecular Diagnostics, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Sandra Jagnjic (S)

Department of Immunohematology, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Jasna Bingulac-Popovic (J)

Department of Molecular Diagnostics, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Ivana Babic (I)

Department of Molecular Diagnostics, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.

Ante Corusic (A)

Directory, University Hospital Centre Zagreb, Zagreb, Croatia.
School of Medicine, University of Zagreb, Zagreb, Croatia.

Irena Jukic (I)

Medical Department, Croatian Institute of Transfusion Medicine, Zagreb, Croatia.
Faculty of Medicine, Josip Juraj Strossmayer University of Osijek, Osijek, Croatia.

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