Effect of Empagliflozin on the Clinical Stability of Patients With Heart Failure and a Reduced Ejection Fraction: The EMPEROR-Reduced Trial.


Journal

Circulation
ISSN: 1524-4539
Titre abrégé: Circulation
Pays: United States
ID NLM: 0147763

Informations de publication

Date de publication:
26 01 2021
Historique:
pubmed: 22 10 2020
medline: 29 12 2021
entrez: 21 10 2020
Statut: ppublish

Résumé

Empagliflozin reduces the risk of cardiovascular death or hospitalization for heart failure in patients with heart failure and a reduced ejection fraction, with or without diabetes, but additional data are needed about the effect of the drug on inpatient and outpatient events that reflect worsening heart failure. We randomly assigned 3730 patients with class II to IV heart failure with an ejection fraction of ≤40% to double-blind treatment with placebo or empagliflozin (10 mg once daily), in addition to recommended treatments for heart failure, for a median of 16 months. We prospectively collected information on inpatient and outpatient events reflecting worsening heart failure and prespecified their analysis in individual and composite end points. Empagliflozin reduced the combined risk of death, hospitalization for heart failure or an emergent/urgent heart failure visit requiring intravenous treatment (415 versus 519 patients; empagliflozin versus placebo, respectively; hazard ratio [HR], 0.76; 95% CI, 0.67-0.87; In patients with heart failure and a reduced ejection fraction, empagliflozin reduced the risk and total number of inpatient and outpatient worsening heart failure events, with benefits seen early after initiation of treatment and sustained for the duration of double-blind therapy. Registration: URL: https://www.clinicaltrials.gov; Unique identifier: NCT03057977.

Sections du résumé

BACKGROUND
Empagliflozin reduces the risk of cardiovascular death or hospitalization for heart failure in patients with heart failure and a reduced ejection fraction, with or without diabetes, but additional data are needed about the effect of the drug on inpatient and outpatient events that reflect worsening heart failure.
METHODS
We randomly assigned 3730 patients with class II to IV heart failure with an ejection fraction of ≤40% to double-blind treatment with placebo or empagliflozin (10 mg once daily), in addition to recommended treatments for heart failure, for a median of 16 months. We prospectively collected information on inpatient and outpatient events reflecting worsening heart failure and prespecified their analysis in individual and composite end points.
RESULTS
Empagliflozin reduced the combined risk of death, hospitalization for heart failure or an emergent/urgent heart failure visit requiring intravenous treatment (415 versus 519 patients; empagliflozin versus placebo, respectively; hazard ratio [HR], 0.76; 95% CI, 0.67-0.87;
CONCLUSIONS
In patients with heart failure and a reduced ejection fraction, empagliflozin reduced the risk and total number of inpatient and outpatient worsening heart failure events, with benefits seen early after initiation of treatment and sustained for the duration of double-blind therapy. Registration: URL: https://www.clinicaltrials.gov; Unique identifier: NCT03057977.

Identifiants

pubmed: 33081531
doi: 10.1161/CIRCULATIONAHA.120.051783
pmc: PMC7834905
doi:

Substances chimiques

Benzhydryl Compounds 0
Glucosides 0
Sodium-Glucose Transporter 2 Inhibitors 0
empagliflozin HDC1R2M35U

Banques de données

ClinicalTrials.gov
['NCT03057977']

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

326-336

Commentaires et corrections

Type : ErratumIn
Type : CommentIn

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Auteurs

Milton Packer (M)

Baylor Heart and Vascular Institute, Baylor University Medical Center, Dallas, TX (M.P.).
Imperial College, London, UK (M.P.).

Stefan D Anker (SD)

Department of Cardiology and Berlin Institute of Health Center for Regenerative Therapies, German Centre for Cardiovascular Research Partner Site Berlin, Charité Universitätsmedizin, Berlin, Germany (S.D.A., W.D.).

Javed Butler (J)

Department of Medicine, University of Mississippi School of Medicine, Jackson (J.B.).

Gerasimos Filippatos (G)

National and Kapodistrian University of Athens School of Medicine, Athens University Hospital Attikon, Greece (G.F.).

João Pedro Ferreira (JP)

Université de Lorraine, Inserm INI-CRCT, CHRU, Nancy, France (J.P.F., F.Z.).

Stuart J Pocock (SJ)

Department of Medical Statistics, London School of Hygiene and Tropical Medicine, UK (S.J.P.).

Peter Carson (P)

Washington DC Veterans Affairs Medical Center (P.C.).

Inder Anand (I)

Department of Cardiology, University of Minnesota, Minneapolis (I.A.).

Wolfram Doehner (W)

Department of Cardiology and Berlin Institute of Health Center for Regenerative Therapies, German Centre for Cardiovascular Research Partner Site Berlin, Charité Universitätsmedizin, Berlin, Germany (S.D.A., W.D.).

Markus Haass (M)

Theresienkrankenhaus and St.Hedwig-Klinik, Mannheim, Germany (M.H.).

Michel Komajda (M)

Department of Cardiology, Hospital Saint Joseph, Paris, France (M.K.).

Alan Miller (A)

University of Florida, Jacksonville (A.M.).

Steen Pehrson (S)

Department of Cardiology, University Hospital, Rigshospitalet, Copenhagen, Denmark (S.P.).

John R Teerlink (JR)

Section of Cardiology, San Francisco Veterans Affairs Medical Center, CA (J.R.T.).

Martina Brueckmann (M)

School of Medicine, University of California, San Diego (J.R.T.).

Waheed Jamal (W)

Boehringer Ingelheim International GmbH, Ingelheim, Germany (M.B., W.J., C.Z.).

Cordula Zeller (C)

Boehringer Ingelheim International GmbH, Ingelheim, Germany (M.B., W.J., C.Z.).

Sven Schnaidt (S)

Biostatistics and Data Sciences, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany (D.M., S.S.).

Faiez Zannad (F)

Université de Lorraine, Inserm INI-CRCT, CHRU, Nancy, France (J.P.F., F.Z.).

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Classifications MeSH