Cardiac and Kidney Benefits of Empagliflozin in Heart Failure Across the Spectrum of Kidney Function: Insights From EMPEROR-Reduced.


Journal

Circulation
ISSN: 1524-4539
Titre abrégé: Circulation
Pays: United States
ID NLM: 0147763

Informations de publication

Date de publication:
26 01 2021
Historique:
pubmed: 24 10 2020
medline: 29 12 2021
entrez: 23 10 2020
Statut: ppublish

Résumé

In EMPEROR-Reduced (Empagliflozin Outcome Trial in Patients With Chronic Heart Failure With Reduced Ejection Fraction), empagliflozin reduced cardiovascular death or heart failure (HF) hospitalization and total HF hospitalizations, and slowed the progressive decline in kidney function in patients with HF and a reduced ejection fraction, with and without diabetes. We aim to study the effect of empagliflozin on cardiovascular and kidney outcomes across the spectrum of kidney function. In this prespecified analysis, patients were categorized by the presence or absence of chronic kidney disease (CKD) at baseline (estimated glomerular filtration rate [eGFR] <60 ml/min/1.73 m Of 3730 patients who were randomized to empagliflozin or placebo, 1978 (53%) had CKD. Empagliflozin reduced the primary outcome and total HF hospitalizations in patients with and without CKD: hazard ratio (HR)=0.78 (95% CI, 0.65-0.93) and HR=0.72 (95% CI, 0.58-0.90), respectively (interaction In EMPEROR-Reduced, empagliflozin had a beneficial effect on the key efficacy outcomes and slowed the rate of kidney function decline in patients with and without CKD, and regardless of the severity of kidney impairment at baseline. Registration: URL: https://www.clinicaltrials.gov; Unique identifier: NCT03057977.

Sections du résumé

BACKGROUND
In EMPEROR-Reduced (Empagliflozin Outcome Trial in Patients With Chronic Heart Failure With Reduced Ejection Fraction), empagliflozin reduced cardiovascular death or heart failure (HF) hospitalization and total HF hospitalizations, and slowed the progressive decline in kidney function in patients with HF and a reduced ejection fraction, with and without diabetes. We aim to study the effect of empagliflozin on cardiovascular and kidney outcomes across the spectrum of kidney function.
METHODS
In this prespecified analysis, patients were categorized by the presence or absence of chronic kidney disease (CKD) at baseline (estimated glomerular filtration rate [eGFR] <60 ml/min/1.73 m
RESULTS
Of 3730 patients who were randomized to empagliflozin or placebo, 1978 (53%) had CKD. Empagliflozin reduced the primary outcome and total HF hospitalizations in patients with and without CKD: hazard ratio (HR)=0.78 (95% CI, 0.65-0.93) and HR=0.72 (95% CI, 0.58-0.90), respectively (interaction
CONCLUSIONS
In EMPEROR-Reduced, empagliflozin had a beneficial effect on the key efficacy outcomes and slowed the rate of kidney function decline in patients with and without CKD, and regardless of the severity of kidney impairment at baseline. Registration: URL: https://www.clinicaltrials.gov; Unique identifier: NCT03057977.

Identifiants

pubmed: 33095032
doi: 10.1161/CIRCULATIONAHA.120.051685
pmc: PMC7834910
doi:

Substances chimiques

Benzhydryl Compounds 0
Glucosides 0
Sodium-Glucose Transporter 2 Inhibitors 0
empagliflozin HDC1R2M35U

Banques de données

ClinicalTrials.gov
['NCT03057977']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

310-321

Commentaires et corrections

Type : CommentIn
Type : CommentIn
Type : ErratumIn
Type : CommentIn

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Auteurs

Faiez Zannad (F)

Université de Lorraine, INSERM, Centre d'Investigations Cliniques Plurithématique 1433, INSERM 1116, CHRU de Nancy, FCRIN INI-CRCT, Nancy, France (F.Z., J.P.F.).

João Pedro Ferreira (JP)

Université de Lorraine, INSERM, Centre d'Investigations Cliniques Plurithématique 1433, INSERM 1116, CHRU de Nancy, FCRIN INI-CRCT, Nancy, France (F.Z., J.P.F.).

Stuart J Pocock (SJ)

Department of Medical Statistics, London School of Hygiene and Tropical Medicine, London, UK (S.J.P.).

Cordula Zeller (C)

Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany (C.Z.).

Stefan D Anker (SD)

Department of Cardiology (CVK), and Berlin Institute of Health Center for Regenerative Therapies, German Centre for Cardiovascular Research Partner Site Berlin, Charité Universitätsmedizin, Berlin, Germany (S.D.A.).

Javed Butler (J)

Department of Medicine, University of Mississippi School of Medicine, Jackson, MS. (J.B.).

Gerasimos Filippatos (G)

National and Kapodistrian University of Athens School of Medicine, Athens University Hospital Attikon, Athens, Greece (G.F.).

Sibylle Jenny Hauske (SJ)

Boehringer Ingelheim International GmbH, Ingelheim, Germany (S.J.H., M.B.).
Vth Department of Medicine, University Medical Center Mannheim (S.J.H.), University of Heidelberg, Mannheim, Germany.

Martina Brueckmann (M)

Boehringer Ingelheim International GmbH, Ingelheim, Germany (S.J.H., M.B.).
Faculty of Medicine Mannheim (M.B.), University of Heidelberg, Mannheim, Germany.

Egon Pfarr (E)

Ingelheim, Germany (E.P.).

Janet Schnee (J)

Boehringer Ingelheim Pharmaceuticals, Ridgefield, CT (J.S.).
Division of Nephrology, University Hospital, Würzburg, Germany (J.S.).

Milton Packer (M)

Baylor Heart and Vascular Institute, Baylor University Medical Center, Dallas TX (M.P.).
Imperial College, London, UK (M.P.).

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Classifications MeSH