Impact of Plasmodium falciparum gene deletions on malaria rapid diagnostic test performance.


Journal

Malaria journal
ISSN: 1475-2875
Titre abrégé: Malar J
Pays: England
ID NLM: 101139802

Informations de publication

Date de publication:
04 Nov 2020
Historique:
received: 27 05 2020
accepted: 23 10 2020
entrez: 5 11 2020
pubmed: 6 11 2020
medline: 10 6 2021
Statut: epublish

Résumé

Malaria rapid diagnostic tests (RDTs) have greatly improved access to diagnosis in endemic countries. Most RDTs detect Plasmodium falciparum histidine-rich protein 2 (HRP2), but their sensitivity is seriously threatened by the emergence of pfhrp2-deleted parasites. RDTs detecting P. falciparum or pan-lactate dehydrogenase (Pf- or pan-LDH) provide alternatives. The objective of this study was to systematically assess the performance of malaria RDTs against well-characterized pfhrp2-deleted P. falciparum parasites. Thirty-two RDTs were tested against 100 wild-type clinical isolates (200 parasites/µL), and 40 samples from 10 culture-adapted and clinical isolates of pfhrp2-deleted parasites. Wild-type and pfhrp2-deleted parasites had comparable Pf-LDH concentrations. Pf-LDH-detecting RDTs were also tested against 18 clinical isolates at higher density (2,000 parasites/µL) lacking both pfhrp2 and pfhrp3. RDT positivity against pfhrp2-deleted parasites was highest (> 94%) for the two pan-LDH-only RDTs. The positivity rate for the nine Pf-LDH-detecting RDTs varied widely, with similar median positivity between double-deleted (pfhrp2/3 negative; 63.9%) and single-deleted (pfhrp2-negative/pfhrp3-positive; 59.1%) parasites, both lower than against wild-type P. falciparum (93.8%). Median positivity for HRP2-detecting RDTs against 22 single-deleted parasites was 69.9 and 35.2% for HRP2-only and HRP2-combination RDTs, respectively, compared to 96.0 and 92.5% for wild-type parasites. Eight of nine Pf-LDH RDTs detected all clinical, double-deleted samples at 2,000 parasites/µL. The pan-LDH-only RDTs evaluated performed well. Performance of Pf-LDH-detecting RDTs against wild-type P. falciparum does not necessarily predict performance against pfhrp2-deleted parasites. Furthermore, many, but not all HRP2-based RDTs, detect pfhrp2-negative/pfhrp3-positive samples, with implications for the HRP2-based RDT screening approach for detection and surveillance of HRP2-negative parasites.

Sections du résumé

BACKGROUND BACKGROUND
Malaria rapid diagnostic tests (RDTs) have greatly improved access to diagnosis in endemic countries. Most RDTs detect Plasmodium falciparum histidine-rich protein 2 (HRP2), but their sensitivity is seriously threatened by the emergence of pfhrp2-deleted parasites. RDTs detecting P. falciparum or pan-lactate dehydrogenase (Pf- or pan-LDH) provide alternatives. The objective of this study was to systematically assess the performance of malaria RDTs against well-characterized pfhrp2-deleted P. falciparum parasites.
METHODS METHODS
Thirty-two RDTs were tested against 100 wild-type clinical isolates (200 parasites/µL), and 40 samples from 10 culture-adapted and clinical isolates of pfhrp2-deleted parasites. Wild-type and pfhrp2-deleted parasites had comparable Pf-LDH concentrations. Pf-LDH-detecting RDTs were also tested against 18 clinical isolates at higher density (2,000 parasites/µL) lacking both pfhrp2 and pfhrp3.
RESULTS RESULTS
RDT positivity against pfhrp2-deleted parasites was highest (> 94%) for the two pan-LDH-only RDTs. The positivity rate for the nine Pf-LDH-detecting RDTs varied widely, with similar median positivity between double-deleted (pfhrp2/3 negative; 63.9%) and single-deleted (pfhrp2-negative/pfhrp3-positive; 59.1%) parasites, both lower than against wild-type P. falciparum (93.8%). Median positivity for HRP2-detecting RDTs against 22 single-deleted parasites was 69.9 and 35.2% for HRP2-only and HRP2-combination RDTs, respectively, compared to 96.0 and 92.5% for wild-type parasites. Eight of nine Pf-LDH RDTs detected all clinical, double-deleted samples at 2,000 parasites/µL.
CONCLUSIONS CONCLUSIONS
The pan-LDH-only RDTs evaluated performed well. Performance of Pf-LDH-detecting RDTs against wild-type P. falciparum does not necessarily predict performance against pfhrp2-deleted parasites. Furthermore, many, but not all HRP2-based RDTs, detect pfhrp2-negative/pfhrp3-positive samples, with implications for the HRP2-based RDT screening approach for detection and surveillance of HRP2-negative parasites.

Identifiants

pubmed: 33148265
doi: 10.1186/s12936-020-03460-w
pii: 10.1186/s12936-020-03460-w
pmc: PMC7640408
doi:

Substances chimiques

Antigens, Protozoan 0
HRP-2 antigen, Plasmodium falciparum 0
HRP3 protein, Plasmodium falciparum 0
Protozoan Proteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

392

Subventions

Organisme : World Health Organization
ID : 001
Pays : International

Références

Malar J. 2017 Mar 20;16(1):123
pubmed: 28320390
PLoS One. 2015 Jul 07;10(7):e0131576
pubmed: 26151448
J Infect Dis. 2017 Jul 1;216(1):36-44
pubmed: 28177502
Malar J. 2015 Mar 18;14:115
pubmed: 25889624
Malar J. 2016 Feb 18;15:101
pubmed: 26891848
Am J Trop Med Hyg. 2012 Feb;86(2):194-8
pubmed: 22302847
Elife. 2017 Aug 24;6:
pubmed: 28837020
PLoS One. 2017 Mar 16;12(3):e0171150
pubmed: 28301474
PLoS One. 2016 Aug 12;11(8):e0157949
pubmed: 27518538
PLoS One. 2010 Jan 25;5(1):e8091
pubmed: 20111602
Sci Rep. 2013 Sep 30;3:2797
pubmed: 24077522
J Clin Microbiol. 2011 Jul;49(7):2694-6
pubmed: 21543573
Acta Trop. 2013 Jan;125(1):119-21
pubmed: 23041541
PLoS One. 2012;7(8):e43094
pubmed: 22952633
Sci Rep. 2017 Nov 7;7(1):14718
pubmed: 29116127
Parasitology. 1987 Oct;95 ( Pt 2):209-27
pubmed: 3320887
Malar J. 2018 Aug 31;17(1):320
pubmed: 30170596
PLoS One. 2015 May 15;10(5):e0126805
pubmed: 25978499
Malar J. 2013 Jan 24;12:34
pubmed: 23347727
Malar J. 2015 Jan 28;14:25
pubmed: 25888818
J Infect Dis. 2017 Apr 1;215(7):1156-1166
pubmed: 28329034
Acta Trop. 2015 Dec;152:26-31
pubmed: 26297799
Emerg Infect Dis. 2018 Mar;24(3):462-470
pubmed: 29460730
Science. 1976 Aug 20;193(4254):673-5
pubmed: 781840

Auteurs

Michelle L Gatton (ML)

Queensland University of Technology, Brisbane, QLD, Australia. m.gatton@qut.edu.au.

Alisha Chaudhry (A)

Queensland University of Technology, Brisbane, QLD, Australia.

Jeff Glenn (J)

The CDC Foundation, Atlanta, GA, USA.

Scott Wilson (S)

The CDC Foundation, Atlanta, GA, USA.

Yong Ah (Y)

The CDC Foundation, Atlanta, GA, USA.

Amy Kong (A)

Centers for Disease Control and Prevention, Atlanta, USA.

Rosalynn L Ord (RL)

Hospital for Tropical Diseases, London, UK.

Roxanne R Rees-Channer (RR)

Hospital for Tropical Diseases, London, UK.

Peter Chiodini (P)

Hospital for Tropical Diseases, London, UK.
London School of Hygiene and Tropical Medicine, London, UK.

Sandra Incardona (S)

Foundation for Innovative New Diagnostics (FIND), Geneva, Switzerland.

Qin Cheng (Q)

Australian Defence Force Malaria and Infectious Diseases Institute (FORMERLY Australian Army Malaria Institute), Brisbane, QLD, Australia.

Michael Aidoo (M)

Centers for Disease Control and Prevention, Atlanta, USA.

Jane Cunningham (J)

World Health Organization, Geneva, Switzerland.

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