SAA (Serum Amyloid A): A Novel Predictor of Stroke-Associated Infections.


Journal

Stroke
ISSN: 1524-4628
Titre abrégé: Stroke
Pays: United States
ID NLM: 0235266

Informations de publication

Date de publication:
12 2020
Historique:
pubmed: 10 11 2020
medline: 18 2 2021
entrez: 9 11 2020
Statut: ppublish

Résumé

The aim of this study was to evaluate and independently validate SAA (serum amyloid A)-a recently discovered blood biomarker-to predict poststroke infections. The derivation cohort (A) was composed of 283 acute ischemic stroke patients and the independent validation cohort (B), of 367 patients. The primary outcome measure was any stroke-associated infection, defined by the criteria of the US Centers for Disease Control and Prevention, occurring during hospitalization. To determine the association of SAA levels on admission with the development of infections, logistic regression models were calculated. The discriminatory ability of SAA was assessed, by calculating the area under the receiver operating characteristic curve. After adjusting for all predictors that were significantly associated with any infection in the univariate analysis, SAA remained an independent predictor in study A (adjusted odds ratio, 1.44 [95% CI, 1.16-1.79]; Among patients with ischemic stroke, blood SAA measured on admission is a novel independent predictor of infection after stroke. SAA improved the discrimination between patients who developed an infection compared with those who did not in both derivation and validation cohorts. Registration: URL: https://www.clinicaltrials.gov. Unique identifier: NCT00390962.

Sections du résumé

BACKGROUND AND PURPOSE
The aim of this study was to evaluate and independently validate SAA (serum amyloid A)-a recently discovered blood biomarker-to predict poststroke infections.
METHODS
The derivation cohort (A) was composed of 283 acute ischemic stroke patients and the independent validation cohort (B), of 367 patients. The primary outcome measure was any stroke-associated infection, defined by the criteria of the US Centers for Disease Control and Prevention, occurring during hospitalization. To determine the association of SAA levels on admission with the development of infections, logistic regression models were calculated. The discriminatory ability of SAA was assessed, by calculating the area under the receiver operating characteristic curve.
RESULTS
After adjusting for all predictors that were significantly associated with any infection in the univariate analysis, SAA remained an independent predictor in study A (adjusted odds ratio, 1.44 [95% CI, 1.16-1.79];
CONCLUSIONS
Among patients with ischemic stroke, blood SAA measured on admission is a novel independent predictor of infection after stroke. SAA improved the discrimination between patients who developed an infection compared with those who did not in both derivation and validation cohorts. Registration: URL: https://www.clinicaltrials.gov. Unique identifier: NCT00390962.

Identifiants

pubmed: 33161846
doi: 10.1161/STROKEAHA.120.030064
doi:

Substances chimiques

Biomarkers 0
Procalcitonin 0
Serum Amyloid A Protein 0
C-Reactive Protein 9007-41-4

Banques de données

ClinicalTrials.gov
['NCT00390962']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3523-3530

Auteurs

Juliane Schweizer (J)

Department of Neurology, University Hospital Zurich, Switzerland (J.S., N.S., V.S., A.L., M.K.).

Alejandro Bustamante (A)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research, Universitat Autònoma de Barcelona, Spain (A.B., J.F., J.M.).
Department of Neurology, Hospital Universitari Vall d'Hebron, Barcelona, Spain (A.B., S.B.).

Vanessa Lapierre-Fétaud (V)

Translational Biomarker Group, Department of Human Protein Sciences, University of Geneva, Switzerland (V.L.-F., L.A., J.-C.S.).

Júlia Faura (J)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research, Universitat Autònoma de Barcelona, Spain (A.B., J.F., J.M.).

Leire Azurmendi Gil (L)

Translational Biomarker Group, Department of Human Protein Sciences, University of Geneva, Switzerland (V.L.-F., L.A., J.-C.S.).

Felix Fluri (F)

Department of Neurology, University Hospital Wuerzburg, Germany (F.F.).

Valerie Schütz (V)

Department of Neurology, University Hospital Zurich, Switzerland (J.S., N.S., V.S., A.L., M.K.).

Andreas Luft (A)

Department of Neurology, University Hospital Zurich, Switzerland (J.S., N.S., V.S., A.L., M.K.).

Sandra Boned (S)

Department of Neurology, Hospital Universitari Vall d'Hebron, Barcelona, Spain (A.B., S.B.).

Jean-Charles Sanchez (JC)

Translational Biomarker Group, Department of Human Protein Sciences, University of Geneva, Switzerland (V.L.-F., L.A., J.-C.S.).

Joan Montaner (J)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research, Universitat Autònoma de Barcelona, Spain (A.B., J.F., J.M.).

Mira Katan (M)

Department of Neurology, University Hospital Zurich, Switzerland (J.S., N.S., V.S., A.L., M.K.).

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Classifications MeSH