Alveolar rhabdomyosarcoma with regional nodal involvement: Results of a combined analysis from two cooperative groups.


Journal

Pediatric blood & cancer
ISSN: 1545-5017
Titre abrégé: Pediatr Blood Cancer
Pays: United States
ID NLM: 101186624

Informations de publication

Date de publication:
03 2021
Historique:
received: 28 09 2020
revised: 26 10 2020
accepted: 08 11 2020
pubmed: 28 11 2020
medline: 3 8 2021
entrez: 27 11 2020
Statut: ppublish

Résumé

Treatment of children and adolescents with alveolar rhabdomyosarcoma (ARMS) and regional nodal involvement (N1) have been approached differently by North American and European cooperative groups. In order to define a better therapeutic strategy, we analyzed two studies conducted between 2005 and 2016 by the European paediatric Soft tissue sarcoma Study Group (EpSSG) and Children's Oncology Group (COG). We retrospectively identified patients with ARMS N1 enrolled in either EpSSG RMS2005 or in COG ARST0531. Chemotherapy in RMS2005 comprised ifosfamide + vincristine + dactinomycin + doxorubicin (IVADo), IVA and maintenance (vinorelbine, cyclophosphamide); in ARST0531, it consisted of either vincristine + dactinomycin + cyclophosphamide (VAC) or VAC alternating with vincristine + irinotecan (VI). Local treatment was similar in both protocols. The analysis of the clinical characteristics of 239 patients showed some differences between study groups: in RMS2005, advanced Intergroup Rhabdomyosarcoma Study Group (IRS) and large tumors predominated. There were no differences in outcomes between the two groups: 5-year event-free survival (EFS), 49% (95% confidence interval [CI]: 39-59) and 44% (95% CI: 30-58), and overall survival (OS), 51% (95% CI: 41-61) and 53.6% (95% CI: 40-68) in RMS2005 and ARST0531, respectively. In RMS2005, EFS of patients with FOXO1-positive tumors was significantly inferior to those with FOXO1-negative (49.3% vs 73%, P = .034). In contrast, in ARST0531, EFS of patients with FOXO1-positive tumors was 45% compared with 43.8% for those with FOXO1-negative. The outcome of patients with ARMS N1 was similar in both protocols. However, patients with FOXO1 fusion-negative tumors enrolled in RMS2005 showed a significantly better outcome, suggesting that different strategies of chemotherapy may have an impact in the outcome of this subgroup of patients.

Sections du résumé

BACKGROUND
Treatment of children and adolescents with alveolar rhabdomyosarcoma (ARMS) and regional nodal involvement (N1) have been approached differently by North American and European cooperative groups. In order to define a better therapeutic strategy, we analyzed two studies conducted between 2005 and 2016 by the European paediatric Soft tissue sarcoma Study Group (EpSSG) and Children's Oncology Group (COG).
METHODS
We retrospectively identified patients with ARMS N1 enrolled in either EpSSG RMS2005 or in COG ARST0531. Chemotherapy in RMS2005 comprised ifosfamide + vincristine + dactinomycin + doxorubicin (IVADo), IVA and maintenance (vinorelbine, cyclophosphamide); in ARST0531, it consisted of either vincristine + dactinomycin + cyclophosphamide (VAC) or VAC alternating with vincristine + irinotecan (VI). Local treatment was similar in both protocols.
RESULTS
The analysis of the clinical characteristics of 239 patients showed some differences between study groups: in RMS2005, advanced Intergroup Rhabdomyosarcoma Study Group (IRS) and large tumors predominated. There were no differences in outcomes between the two groups: 5-year event-free survival (EFS), 49% (95% confidence interval [CI]: 39-59) and 44% (95% CI: 30-58), and overall survival (OS), 51% (95% CI: 41-61) and 53.6% (95% CI: 40-68) in RMS2005 and ARST0531, respectively. In RMS2005, EFS of patients with FOXO1-positive tumors was significantly inferior to those with FOXO1-negative (49.3% vs 73%, P = .034). In contrast, in ARST0531, EFS of patients with FOXO1-positive tumors was 45% compared with 43.8% for those with FOXO1-negative.
CONCLUSIONS
The outcome of patients with ARMS N1 was similar in both protocols. However, patients with FOXO1 fusion-negative tumors enrolled in RMS2005 showed a significantly better outcome, suggesting that different strategies of chemotherapy may have an impact in the outcome of this subgroup of patients.

Identifiants

pubmed: 33245207
doi: 10.1002/pbc.28832
pmc: PMC8414760
mid: NIHMS1734154
doi:

Types de publication

Journal Article Multicenter Study Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e28832

Subventions

Organisme : NIH HHS
ID : U10CA098543
Pays : United States
Organisme : Cancer Research UK
ID : 5943
Pays : United Kingdom
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NIH HHS
ID : U10CA098413
Pays : United States
Organisme : NCI NIH HHS
ID : U10 CA098543
Pays : United States
Organisme : NCI NIH HHS
ID : U10 CA180899
Pays : United States
Organisme : NCI NIH HHS
ID : U10 CA180886
Pays : United States
Organisme : NCI NIH HHS
ID : U10 CA098413
Pays : United States
Organisme : NIH HHS
ID : U10CA180899
Pays : United States
Organisme : NIH HHS
ID : U10CA180886
Pays : United States

Informations de copyright

© 2020 Wiley Periodicals LLC.

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Auteurs

Soledad Gallego (S)

University Hospital Vall d'Hebron, Barcelona, Spain.

Yueh-Yun Chi (YY)

Children's Hospital Los Angeles, University of Southern California, Los Angeles, California.

Gian Luca De Salvo (GL)

Clinical Trials and Biostatistics Unit, Veneto Oncologic Institute IOV-IRCCS, Padua, Italy.

Minjie Li (M)

University of Florida, Gainesville, Florida.

Johannes H M Merks (JHM)

Princess Maxima Center for Pediatric Oncology, Utrecht, The Netherlands.

David A Rodeberg (DA)

East Carolina University, Greenville, North Carolina.

Sheila Terwisscha van Scheltinga (ST)

Princess Maxima Center for Pediatric Oncology, Utrecht, The Netherlands.

Leo Mascarenhas (L)

Children's Hospital Los Angeles, University of Southern California, Los Angeles, California.

Daniel Orbach (D)

SIREDO Oncology Center, Institut Curie, PSL University, Paris, France.

Meriel Jenney (M)

Children Hospital for Wales Cardiff and Vale University Health Board, Cardiff, UK.

Lynn Million (L)

Stanford University School of Medicine, Stanford, California.

Veronique Minard-Colin (V)

Gustave Roussy, University Paris-Saclay, Villejuif, France.

Suzanne Wolden (S)

Memorial Sloan Kettering Cancer Center, New York, New York.

Ilaria Zanetti (I)

Padova University Hospital, Padua, Italy.

David M Parham (DM)

Children's Hospital Los Angeles, University of Southern California, Los Angeles, California.

Henry Mandeville (H)

The Royal Marsden NHS Foundation Trust, Sutton, UK.

Rajkumar Venkatramani (R)

Texas Children's Hospital, Baylor College of Medicine, Houston, Texas.

Gianni Bisogno (G)

Padova University Hospital, Padua, Italy.

Douglas S Hawkins (DS)

Seattle Children's Hospital, Fred Hutchinson Cancer Research Center, University of Washington, Seattle, Washington.
University Hospital Vall d'Hebron, Barcelona, Spain.

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