Altered Cl
Animals
Cell Movement
/ physiology
Chlorides
/ metabolism
Cytoskeletal Proteins
/ genetics
GABAergic Neurons
/ metabolism
GTPase-Activating Proteins
/ genetics
Homeostasis
Intellectual Disability
/ metabolism
Interneurons
/ metabolism
Male
Mice
Models, Animal
Neural Stem Cells
/ metabolism
Neurogenesis
Nuclear Proteins
/ metabolism
Prosencephalon
/ metabolism
Signal Transduction
rhoA GTP-Binding Protein
/ metabolism
2P imaging
Cl homeostasis
OPHN1
intellectual disability
interneuron migration
Journal
Proceedings of the National Academy of Sciences of the United States of America
ISSN: 1091-6490
Titre abrégé: Proc Natl Acad Sci U S A
Pays: United States
ID NLM: 7505876
Informations de publication
Date de publication:
12 01 2021
12 01 2021
Historique:
entrez:
30
12
2020
pubmed:
31
12
2020
medline:
19
5
2021
Statut:
ppublish
Résumé
Impairments of inhibitory circuits are at the basis of most, if not all, cognitive deficits. The impact of OPHN1, a gene associate with intellectual disability (ID), on inhibitory neurons remains elusive. We addressed this issue by analyzing the postnatal migration of inhibitory interneurons derived from the subventricular zone in a validated mouse model of ID (OPHN1
Identifiants
pubmed: 33376209
pii: 2016034118
doi: 10.1073/pnas.2016034118
pmc: PMC7812749
pii:
doi:
Substances chimiques
Chlorides
0
Cytoskeletal Proteins
0
GTPase-Activating Proteins
0
Nuclear Proteins
0
Ophn1 protein, mouse
0
RhoA protein, mouse
EC 3.6.5.2
rhoA GTP-Binding Protein
EC 3.6.5.2
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Déclaration de conflit d'intérêts
The authors declare no competing interest.
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