Chlorthalidone with potassium citrate decreases calcium oxalate stones and increases bone quality in genetic hypercalciuric stone-forming rats.


Journal

Kidney international
ISSN: 1523-1755
Titre abrégé: Kidney Int
Pays: United States
ID NLM: 0323470

Informations de publication

Date de publication:
05 2021
Historique:
received: 01 06 2020
revised: 16 12 2020
accepted: 17 12 2020
pubmed: 9 1 2021
medline: 22 6 2021
entrez: 8 1 2021
Statut: ppublish

Résumé

To study human idiopathic hypercalciuria we developed an animal model, genetic hypercalciuric stone-forming rats, whose pathophysiology parallels that of human idiopathic hypercalciuria. Fed the oxalate precursor, hydroxyproline, every rat in this model develops calcium oxalate stones. Using this rat model, we tested whether chlorthalidone and potassium citrate combined would reduce calcium oxalate stone formation and improve bone quality more than either agent alone. These rats (113 generation) were fed a normal calcium and phosphorus diet with hydroxyproline and divided into four groups: diets plus potassium chloride as control, potassium citrate, chlorthalidone plus potassium chloride, or potassium citrate plus chlorthalidone. Urine was collected at six, 12, and 18 weeks and kidney stone formation and bone parameters were determined. Compared to potassium chloride, potassium citrate reduced urinary calcium, chlorthalidone reduced it further and potassium citrate plus chlorthalidone even further. Potassium citrate plus chlorthalidone decreased urine oxalate compared to all other groups. There were no significant differences in calcium oxalate supersaturation in any group. Neither potassium citrate nor chlorthalidone altered stone formation. However, potassium citrate plus chlorthalidone significantly reduced stone formation. Vertebral trabecular bone increased with chlorthalidone and potassium citrate plus chlorthalidone. Cortical bone area increased with chlorthalidone but not potassium citrate or potassium citrate plus chlorthalidone. Mechanical properties of trabecular bone improved with chlorthalidone, but not with potassium citrate plus chlorthalidone. Thus in genetic hypercalciuric stone-forming rats fed a diet resulting in calcium oxalate stone formation, potassium citrate plus chlorthalidone prevented stone formation better than either agent alone. Chlorthalidone alone improved bone quality, but adding potassium citrate provided no additional benefit.

Identifiants

pubmed: 33417997
pii: S0085-2538(20)31548-9
doi: 10.1016/j.kint.2020.12.023
pmc: PMC8076055
mid: NIHMS1660065
pii:
doi:

Substances chimiques

Calcium Oxalate 2612HC57YE
Potassium Citrate EE90ONI6FF
Chlorthalidone Q0MQD1073Q
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1118-1126

Subventions

Organisme : NIDDK NIH HHS
ID : R01 DK075462
Pays : United States

Informations de copyright

Copyright © 2021 International Society of Nephrology. Published by Elsevier Inc. All rights reserved.

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Auteurs

Nancy S Krieger (NS)

Division of Nephrology, Department of Medicine University of Rochester School of Medicine and Dentistry, Rochester, New York, USA. Electronic address: Nancy_Krieger@URMC.Rochester.edu.

John Asplin (J)

Litholink Corporation, Laboratory Corporation of America Holdings, Chicago, Illinois, USA.

Ignacio Granja (I)

Litholink Corporation, Laboratory Corporation of America Holdings, Chicago, Illinois, USA.

Luojing Chen (L)

Division of Nephrology, Department of Medicine University of Rochester School of Medicine and Dentistry, Rochester, New York, USA.

Daiana Spataru (D)

Lunenfeld-Tanenbaum Research Institute, Mount Sinai Hospital, Toronto, Ontario, Canada.

Tong Tong Wu (TT)

Department of Biostatistics and Computational Biology, University of Rochester School of Medicine, Rochester, New York, USA.

Marc Grynpas (M)

Lunenfeld-Tanenbaum Research Institute, Mount Sinai Hospital, Toronto, Ontario, Canada.

David A Bushinsky (DA)

Division of Nephrology, Department of Medicine University of Rochester School of Medicine and Dentistry, Rochester, New York, USA.

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Classifications MeSH