Survival relative to pacemaker status after transcatheter aortic valve implantation.


Journal

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
ISSN: 1522-726X
Titre abrégé: Catheter Cardiovasc Interv
Pays: United States
ID NLM: 100884139

Informations de publication

Date de publication:
09 2021
Historique:
received: 28 12 2020
accepted: 08 01 2021
pubmed: 28 1 2021
medline: 21 10 2021
entrez: 27 1 2021
Statut: ppublish

Résumé

To determine whether a permanent pacemaker (PPM) in situ can enhance survival after transcatheter aortic valve implantation (TAVI), in a predominantly inoperable or high risk cohort. New conduction disturbances are the most frequent complication of TAVI, often necessitating PPM implantation before hospital discharge. We performed an observational cohort analysis of the UK TAVI registry (2007-2015). Primary and secondary endpoints were 30-day post-discharge all-cause mortality and long-term survival, respectively. Of 8,651 procedures, 6,815 complete datasets were analyzed. A PPM at hospital discharge, irrespective of when implantation occurred (PPM 1.68% [22/1309] vs. no PPM 1.47% [81/5506], odds ratio [OR] 1.14, 95% confidence interval [CI] 0.71-1.84; p = .58), or a PPM implanted peri- or post-TAVI only (PPM 1.44% [11/763] vs. no PPM 1.47% [81/5506], OR 0.98 [0.51-1.85]; p = .95) did not significantly reduce the primary endpoint. Patients with a PPM at discharge were older, male, had right bundle branch block at baseline, were more likely to have received a first-generation self-expandable prosthesis and had experienced more peri- and post-procedural complications including bailout valve-in-valve rescue, bleeding and acute kidney injury. A Cox proportional hazards model demonstrated significantly reduced long-term survival in all those with a PPM, irrespective of implantation timing (hazard ratio [HR] 1.14 [1.02-1.26]; p = .019) and those receiving a PPM only at the time of TAVI (HR 1.15 [1.02-1.31]; p = .032). The reasons underlying this observation warrant further investigation. A PPM did not confer a survival advantage in the first 30 days after hospital discharge following TAVI.

Sections du résumé

OBJECTIVES
To determine whether a permanent pacemaker (PPM) in situ can enhance survival after transcatheter aortic valve implantation (TAVI), in a predominantly inoperable or high risk cohort.
BACKGROUND
New conduction disturbances are the most frequent complication of TAVI, often necessitating PPM implantation before hospital discharge.
METHODS
We performed an observational cohort analysis of the UK TAVI registry (2007-2015). Primary and secondary endpoints were 30-day post-discharge all-cause mortality and long-term survival, respectively.
RESULTS
Of 8,651 procedures, 6,815 complete datasets were analyzed. A PPM at hospital discharge, irrespective of when implantation occurred (PPM 1.68% [22/1309] vs. no PPM 1.47% [81/5506], odds ratio [OR] 1.14, 95% confidence interval [CI] 0.71-1.84; p = .58), or a PPM implanted peri- or post-TAVI only (PPM 1.44% [11/763] vs. no PPM 1.47% [81/5506], OR 0.98 [0.51-1.85]; p = .95) did not significantly reduce the primary endpoint. Patients with a PPM at discharge were older, male, had right bundle branch block at baseline, were more likely to have received a first-generation self-expandable prosthesis and had experienced more peri- and post-procedural complications including bailout valve-in-valve rescue, bleeding and acute kidney injury. A Cox proportional hazards model demonstrated significantly reduced long-term survival in all those with a PPM, irrespective of implantation timing (hazard ratio [HR] 1.14 [1.02-1.26]; p = .019) and those receiving a PPM only at the time of TAVI (HR 1.15 [1.02-1.31]; p = .032). The reasons underlying this observation warrant further investigation.
CONCLUSIONS
A PPM did not confer a survival advantage in the first 30 days after hospital discharge following TAVI.

Identifiants

pubmed: 33502784
doi: 10.1002/ccd.29498
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

E444-E452

Subventions

Organisme : Research Trainees Coordinating Centre
ID : CL-2016-27-001
Organisme : NHS National Institute for Health Research Oxford Biomedical Research Center

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

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Auteurs

Aung Myat (A)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.
Division of Clinical and Experimental Medicine, Brighton and Sussex Medical School, Brighton, UK.

Florence Mouy (F)

Division of Clinical and Experimental Medicine, Brighton and Sussex Medical School, Brighton, UK.

Luke Buckner (L)

Division of Clinical and Experimental Medicine, Brighton and Sussex Medical School, Brighton, UK.

James Cockburn (J)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

Andreas Baumbach (A)

William Harvey Research Institute, Queen Mary University of London, London, UK.
Barts Heart Center, Barts Health NHS Trust, London, UK.
Section of Cardiology, Yale University School of Medicine, New Haven, Connecticut, USA.

Philip MacCarthy (P)

Faculty of Life Sciences and Medicine, King's College London, London, UK.

Adrian P Banning (AP)

Oxford Heart Center, Oxford University Hospitals NHS Trust, Oxford, UK.

Nick Curzen (N)

Department of Cardiology, University Hospital Southampton NHS Foundation Trust, Southampton, UK.

Roland Hilling-Smith (R)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

Daniel J Blackman (DJ)

Yorkshire Heart Centre, The Leeds Teaching Hospitals NHS Trust, Leeds, UK.

Michael Mullen (M)

Barts Heart Center, Barts Health NHS Trust, London, UK.

Mark de Belder (M)

Barts Heart Center, Barts Health NHS Trust, London, UK.
Department of Cardiology, The James Cook University Hospital, Middlesbrough, UK.

Ian Cox (I)

Department of Cardiology, University Hospitals Plymouth NHS Trust, Plymouth, UK.

Jan Kovac (J)

Glenfield Hospital, University of Leicester, Leicester, UK.

Ganesh Manoharan (G)

Royal Victoria Hospital, Belfast Health and Social Care Trust, Belfast, UK.

Azfar Zaman (A)

Institute of Cellular Medicine, Newcastle University, Newcastle, UK.

Douglas Muir (D)

Department of Cardiology, The James Cook University Hospital, Middlesbrough, UK.

David Smith (D)

Department of Cardiology, Morriston Hospital, Swansea, UK.

Stephen Brecker (S)

Cardiology Clinical Academic Group, St. George's University of London, London, UK.

Mark Turner (M)

Bristol Heart Institute, Bristol, UK.

Saib Khogali (S)

Heart and Lung Center, New Cross Hospital, Wolverhampton, UK.

Iqbal S Malik (IS)

Hammersmith Hospital, Imperial College Healthcare NHS Trust, London, UK.

Osama Alsanjari (O)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

Francesca D'Auria (F)

Azienda Ospedaliera Universitaria Maggiore della Carita, Novara, Italy.

Simon Redwood (S)

Cardiothoracic Directorate, Guy's and St Thomas' NHS Foundation Trust, London, UK.

Bernard Prendergast (B)

Cardiothoracic Directorate, Guy's and St Thomas' NHS Foundation Trust, London, UK.

Uday Trivedi (U)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

Derek Robinson (D)

Department of Mathematics, University of Sussex, Brighton, UK.

Peter Ludman (P)

Cardiology Department, Queen Elizabeth Hospital, Birmingham, UK.

Adam de Belder (A)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

David Hildick-Smith (D)

Sussex Cardiac Center, Brighton and Sussex University Hospitals NHS Trust, Brighton, UK.

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