Modulators of CFTR. Updates on clinical development and future directions.


Journal

European journal of medicinal chemistry
ISSN: 1768-3254
Titre abrégé: Eur J Med Chem
Pays: France
ID NLM: 0420510

Informations de publication

Date de publication:
05 Mar 2021
Historique:
received: 03 08 2020
revised: 08 01 2021
accepted: 11 01 2021
pubmed: 2 2 2021
medline: 1 5 2021
entrez: 1 2 2021
Statut: ppublish

Résumé

Cystic fibrosis (CF) is the most frequent life-limiting autosomal recessive disorder in the Caucasian population. It is due to mutations in the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) gene. Current symptomatic CF therapies, which treat the downstream consequences of CFTR mutations, have increased survival. Better knowledge of the CFTR protein has enabled pharmacologic therapy aiming to restore mutated CFTR expression and function. These CFTR "modulators" have revolutionised the CF therapeutic landscape, with the potential to transform prognosis for a considerable number of patients. This review provides a brief summary of their mechanism of action and presents a thorough review of the results obtained from clinical trials of CFTR modulators.

Identifiants

pubmed: 33524685
pii: S0223-5234(21)00044-1
doi: 10.1016/j.ejmech.2021.113195
pii:
doi:

Substances chimiques

Aminophenols 0
Aminopyridines 0
Benzodioxoles 0
CFTR protein, human 0
Indoles 0
Quinolones 0
tezacaftor 0
Cystic Fibrosis Transmembrane Conductance Regulator 126880-72-6
ivacaftor 1Y740ILL1Z
lumacaftor EGP8L81APK

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

113195

Informations de copyright

Copyright © 2021 Elsevier Masson SAS. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Emmanuelle Bardin (E)

Institut Necker Enfants Malades. INSERM U1151, Paris, France.

Alexandra Pastor (A)

LCBPT, UMR CNRS 8601, Paris, France; Université de Paris, Paris, France.

Michaela Semeraro (M)

Centre d'Investigation Clinique, Unité de Recherche Clinique-CIC P1419, Hôpital Necker Enfants Malades, Université de Paris, Paris, France.

Anita Golec (A)

Institut Necker Enfants Malades. INSERM U1151, Paris, France.

Kate Hayes (K)

Clinical Trial Network, European Cystic Fibrosis Society, Belfast, Ireland.

Benoit Chevalier (B)

Institut Necker Enfants Malades. INSERM U1151, Paris, France.

Farouk Berhal (F)

LCBPT, UMR CNRS 8601, Paris, France; Université de Paris, Paris, France.

Guillaume Prestat (G)

LCBPT, UMR CNRS 8601, Paris, France; Université de Paris, Paris, France.

Alexandre Hinzpeter (A)

Institut Necker Enfants Malades. INSERM U1151, Paris, France.

Christine Gravier-Pelletier (C)

LCBPT, UMR CNRS 8601, Paris, France; Université de Paris, Paris, France.

Iwona Pranke (I)

Institut Necker Enfants Malades. INSERM U1151, Paris, France.

Isabelle Sermet-Gaudelus (I)

Institut Necker Enfants Malades. INSERM U1151, Paris, France; Université de Paris, Paris, France; Clinical Trial Network, European Cystic Fibrosis Society, Belfast, Ireland; Centre de Référence Maladies Rares, Mucoviscidose et Maladies de CFTR, Hôpital Necker Enfants Malades, Paris, France; European Respiratory Network Lung, Paris, France. Electronic address: Isabelle.sermet@aphp.fr.

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Classifications MeSH