Mucosal-Associated Invariant T (MAIT) Cells Are Highly Activated and Functionally Impaired in COVID-19 Patients.
Adaptive Immunity
COVID-19
/ immunology
Cytokines
/ metabolism
Female
Granzymes
/ metabolism
HLA-DR Antigens
Humans
Interleukin-17
/ metabolism
Killer Cells, Natural
/ immunology
Lymphocyte Activation
Male
Mucosal-Associated Invariant T Cells
/ immunology
Severity of Illness Index
T-Lymphocyte Subsets
/ immunology
Tumor Necrosis Factor-alpha
/ metabolism
COVID-19
SARS-CoV-2
mucosal-associated invariant T (MAIT) cells
Journal
Viruses
ISSN: 1999-4915
Titre abrégé: Viruses
Pays: Switzerland
ID NLM: 101509722
Informations de publication
Date de publication:
03 02 2021
03 02 2021
Historique:
received:
18
12
2020
revised:
22
01
2021
accepted:
28
01
2021
entrez:
6
2
2021
pubmed:
7
2
2021
medline:
18
2
2021
Statut:
epublish
Résumé
Coronavirus disease 2019 (COVID-19), caused by infection with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), comprises mild courses of disease as well as progression to severe disease, characterised by lung and other organ failure. The immune system is considered to play a crucial role for the pathogenesis of COVID-19, although especially the contribution of innate-like T cells remains poorly understood. Here, we analysed the phenotype and function of mucosal-associated invariant T (MAIT) cells, innate-like T cells with potent antimicrobial effector function, in patients with mild and severe COVID-19 by multicolour flow cytometry. Our data indicate that MAIT cells are highly activated in patients with COVID-19, irrespective of the course of disease, and express high levels of proinflammatory cytokines such as IL-17A and TNFα ex vivo. Of note, expression of the activation marker HLA-DR positively correlated with SAPS II score, a measure of disease severity. Upon MAIT cell-specific in vitro stimulation, MAIT cells however failed to upregulate expression of the cytokines IL-17A and TNFα, as well as cytolytic proteins, that is, granzyme B and perforin. Thus, our data point towards an altered cytokine expression profile alongside an impaired antibacterial and antiviral function of MAIT cells in COVID-19 and thereby contribute to the understanding of COVID-19 immunopathogenesis.
Identifiants
pubmed: 33546489
pii: v13020241
doi: 10.3390/v13020241
pmc: PMC7913667
pii:
doi:
Substances chimiques
Cytokines
0
HLA-DR Antigens
0
IL17A protein, human
0
Interleukin-17
0
TNF protein, human
0
Tumor Necrosis Factor-alpha
0
GZMB protein, human
EC 3.4.21.-
Granzymes
EC 3.4.21.-
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Deutsche Forschungsgemeinschaft
ID : 424774790
Organisme : Else Kröner-Fresenius-Stiftung
ID : Else Kröner Memorial Stipend
Organisme : Bavarian State Ministry for Science and the Arts
ID : n.a.
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