Longitudinal analysis of interictal electroencephalograms in patients with temporal lobe epilepsy with hippocampal sclerosis.


Journal

Seizure
ISSN: 1532-2688
Titre abrégé: Seizure
Pays: England
ID NLM: 9306979

Informations de publication

Date de publication:
Aug 2021
Historique:
received: 26 11 2020
revised: 05 02 2021
accepted: 06 02 2021
pubmed: 21 2 2021
medline: 12 8 2021
entrez: 20 2 2021
Statut: ppublish

Résumé

While studies have shown the progression of atrophy in temporal lobe epilepsy (TLE) with hippocampal sclerosis (HS), little is known about the long-term dynamics of interictal epileptiform discharges (IEDs). To investigate long-term IEDs distribution in routine EEGs. We evaluated 314 patients with TLE and MRI signs of HS (TLE-HS). Six had bilateral, 163 had left, and 145 had right HS. We analyzed 3655 routine EEGs (average 11.6 EEGs/patient). The EEGs were classified into four groups: (i) ipsilateral-IEDs (n = 1485), EEGs with only IEDs ipsilateral to the HS; (ii) bilateral-IEDs (n = 390); (iii) contralateral-IEDs (n = 186); and (iv) normal-EEGs (n = 1594). The duration of epilepsy at the time of the EEG (average 27.9 years) was divided into four groups: (a) <8 years (n = 140), (b) 9-17 years (n = 505), (c) 18-29 years (n = 1165), and (d) >30 years (n = 1845). We performed ANOVA with Tukey's pairwise comparisons and linear regression analysis between the duration of epilepsy and the EEG groups. The ANOVA showed a difference in the distribution of IEDs over time (p < 0.0001). While there were no significant changes in the relative numbers of bilateral and contralateral-IEDs combined, there was a significant increase in ipsilateral-IEDs (p < 0.0001) and a decrease in normal-EEGs (p < 0.0001) over time. The linear regression analysis confirmed that the proportion of ipsilateral-IEDs (p < 0.0001), and to a lesser extent, bilateral-IEDs (p = 0.0002), increased over time, while contralateral-IEDs were unchanged (p = 0.923). Contrary to our expectations, contralateral-IEDs remained stable over time, whereas normal-EEGs decreased and ipsilateral-IEDs increased. Contralateral-IEDs may reflect early abnormalities and not epilepsy progression.

Sections du résumé

BACKGROUND BACKGROUND
While studies have shown the progression of atrophy in temporal lobe epilepsy (TLE) with hippocampal sclerosis (HS), little is known about the long-term dynamics of interictal epileptiform discharges (IEDs).
OBJECTIVES OBJECTIVE
To investigate long-term IEDs distribution in routine EEGs.
METHODS METHODS
We evaluated 314 patients with TLE and MRI signs of HS (TLE-HS). Six had bilateral, 163 had left, and 145 had right HS. We analyzed 3655 routine EEGs (average 11.6 EEGs/patient). The EEGs were classified into four groups: (i) ipsilateral-IEDs (n = 1485), EEGs with only IEDs ipsilateral to the HS; (ii) bilateral-IEDs (n = 390); (iii) contralateral-IEDs (n = 186); and (iv) normal-EEGs (n = 1594). The duration of epilepsy at the time of the EEG (average 27.9 years) was divided into four groups: (a) <8 years (n = 140), (b) 9-17 years (n = 505), (c) 18-29 years (n = 1165), and (d) >30 years (n = 1845). We performed ANOVA with Tukey's pairwise comparisons and linear regression analysis between the duration of epilepsy and the EEG groups.
RESULTS RESULTS
The ANOVA showed a difference in the distribution of IEDs over time (p < 0.0001). While there were no significant changes in the relative numbers of bilateral and contralateral-IEDs combined, there was a significant increase in ipsilateral-IEDs (p < 0.0001) and a decrease in normal-EEGs (p < 0.0001) over time. The linear regression analysis confirmed that the proportion of ipsilateral-IEDs (p < 0.0001), and to a lesser extent, bilateral-IEDs (p = 0.0002), increased over time, while contralateral-IEDs were unchanged (p = 0.923).
CONCLUSIONS CONCLUSIONS
Contrary to our expectations, contralateral-IEDs remained stable over time, whereas normal-EEGs decreased and ipsilateral-IEDs increased. Contralateral-IEDs may reflect early abnormalities and not epilepsy progression.

Identifiants

pubmed: 33608133
pii: S1059-1311(21)00042-X
doi: 10.1016/j.seizure.2021.02.008
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

141-144

Informations de copyright

Copyright © 2021 British Epilepsy Association. Published by Elsevier Ltd. All rights reserved.

Auteurs

Mariana R Brito (MR)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: mariana_rabelo_@hotmail.com.

Thiago S Prado (TS)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: pradoufsj@yahoo.com.br.

Marina K M Alvim (MKM)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: marinakma@gmail.com.

Lucas S R Santos (LSR)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: lucas110693@gmail.com.

Marcia Morita-Sherman (M)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: MORITAM2@ccf.org.

Clarissa L Yasuda (CL)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: cyasuda@unicamp.br.

Fernando Cendes (F)

Department of Neurology, University of Campinas (UNICAMP), Campinas, SP, Brazil. Electronic address: fcendes@unicamp.br.

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