Inherent mosaicism and extensive mutation of human placentas.


Journal

Nature
ISSN: 1476-4687
Titre abrégé: Nature
Pays: England
ID NLM: 0410462

Informations de publication

Date de publication:
04 2021
Historique:
received: 02 09 2020
accepted: 08 02 2021
pubmed: 12 3 2021
medline: 10 4 2021
entrez: 11 3 2021
Statut: ppublish

Résumé

Placentas can exhibit chromosomal aberrations that are absent from the fetus

Identifiants

pubmed: 33692543
doi: 10.1038/s41586-021-03345-1
pii: 10.1038/s41586-021-03345-1
pmc: PMC7611644
mid: EMS132530
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

80-85

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 110104
Pays : United Kingdom

Commentaires et corrections

Type : ErratumIn

Références

Kalousek, D. K. & Dill, F. J. Chromosomal mosaicism confined to the placenta in human conceptions. Science 221, 665–667 (1983).
pubmed: 6867735 doi: 10.1126/science.6867735
Brosens, I., Pijnenborg, R., Vercruysse, L. & Romero, R. The “Great Obstetrical Syndromes” are associated with disorders of deep placentation. Am. J. Obstet. Gynecol. 204, 193–201 (2011).
pubmed: 21094932 doi: 10.1016/j.ajog.2010.08.009
Kalousek, D. K. et al. Confirmation of CVS mosaicism in term placentae and high frequency of intrauterine growth retardation association with confined placental mosaicism. Prenat. Diagn. 11, 743–750 (1991).
pubmed: 1800987 doi: 10.1002/pd.1970111002
Xenopoulos, P., Kang, M. & Hadjantonakis, A. K. Cell lineage allocation within the inner cell mass of the mouse blastocyst. Results Probl. Cell Differ. 55, 185–202 (2012).
pubmed: 22918807 pmcid: 3469159 doi: 10.1007/978-3-642-30406-4_10
Bolton, H. et al. Mouse model of chromosome mosaicism reveals lineage-specific depletion of aneuploid cells and normal developmental potential. Nat. Commun. 7, 11165 (2016).
pubmed: 27021558 pmcid: 4820631 doi: 10.1038/ncomms11165
Behjati, S. et al. Genome sequencing of normal cells reveals developmental lineages and mutational processes. Nature 513, 422–425 (2014).
pubmed: 25043003 pmcid: 4227286 doi: 10.1038/nature13448
Ju, Y. S. et al. Somatic mutations reveal asymmetric cellular dynamics in the early human embryo. Nature 543, 714–718 (2017).
pubmed: 28329761 pmcid: 6169740 doi: 10.1038/nature21703
Coorens, T. H. H. et al. Embryonal precursors of Wilms tumor. Science 366, 1247–1251 (2019).
pubmed: 31806814 pmcid: 6914378 doi: 10.1126/science.aax1323
Alexandrov, L. B. et al. The repertoire of mutational signatures in human cancer. Nature 578, 94–101 (2020).
pubmed: 32025018 pmcid: 7054213 doi: 10.1038/s41586-020-1943-3
Gaccioli, F., Lager, S., Sovio, U., Charnock-Jones, D. S. & Smith, G. C. S. The pregnancy outcome prediction (POP) study: investigating the relationship between serial prenatal ultrasonography, biomarkers, placental phenotype and adverse pregnancy outcomes. Placenta 59, S17–S25 (2017).
doi: 10.1016/j.placenta.2016.10.011 pmcid: 5701771
The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium. Pan-cancer analysis of whole genomes. Nature 578, 82–93 (2020).
doi: 10.1038/s41586-020-1969-6
Lee-Six, H. et al. The landscape of somatic mutation in normal colorectal epithelial cells. Nature 574, 532–537 (2019).
pubmed: 31645730 doi: 10.1038/s41586-019-1672-7
Moore, L. et al. The mutational landscape of normal human endometrial epithelium. Nature 580, 640–646 (2020).
pubmed: 32350471 doi: 10.1038/s41586-020-2214-z
Brunner, S. F. et al. Somatic mutations and clonal dynamics in healthy and cirrhotic human liver. Nature 574, 538–542 (2019).
pubmed: 31645727 pmcid: 6837891 doi: 10.1038/s41586-019-1670-9
Poetsch, A. R. The genomics of oxidative DNA damage, repair, and resulting mutagenesis. Comput. Struct. Biotechnol. J. 18, 207–219 (2020).
pubmed: 31993111 pmcid: 6974700 doi: 10.1016/j.csbj.2019.12.013
Castellucci, M., Scheper, M., Scheffen, I., Celona, A. & Kaufmann, P. The development of the human placental villous tree. Anat. Embryol. (Berl.) 181, 117–128 (1990).
doi: 10.1007/BF00198951
Knöfler, M. et al. Human placenta and trophoblast development: key molecular mechanisms and model systems. Cell. Mol. Life Sci. 76, 3479–3496 (2019).
pubmed: 31049600 pmcid: 6697717 doi: 10.1007/s00018-019-03104-6
Gröbner, S. N. et al. The landscape of genomic alterations across childhood cancers. Nature 555, 321–327 (2018).
doi: 10.1038/nature25480 pubmed: 29489754
Choufani, S., Shuman, C. & Weksberg, R. Molecular findings in Beckwith–Wiedemann syndrome. Am. J. Med. Genet. C Semin. Med. Genet. 163, 131–140 (2013).
doi: 10.1002/ajmg.c.31363
Arima, T. et al. Association of IGF2 and H19 imprinting with choriocarcinoma development. Cancer Genet Cytogenet 93, 39–47 (1997).
Martincorena, I. et al. Somatic mutant clones colonize the human esophagus with age. Science 362, 911–917 (2018).
pubmed: 30337457 pmcid: 6298579 doi: 10.1126/science.aau3879
Martincorena, I. et al. High burden and pervasive positive selection of somatic mutations in normal human skin. Science 348, 880–886 (2015).
pubmed: 25999502 pmcid: 4471149 doi: 10.1126/science.aaa6806
Kuijk, E. et al. Early divergence of mutational processes in human fetal tissues. Sci. Adv. 5, eaaw1271 (2019).
pubmed: 31149636 pmcid: 6541467 doi: 10.1126/sciadv.aaw1271
Jauniaux, E. et al. Onset of maternal arterial blood flow and placental oxidative stress. A possible factor in human early pregnancy failure. Am. J. Pathol. 157, 2111–2122 (2000).
pubmed: 11106583 pmcid: 1885754 doi: 10.1016/S0002-9440(10)64849-3
Amor, D. J. et al. Health and developmental outcome of children following prenatal diagnosis of confined placental mosaicism. Prenat. Diagn. 26, 443–448 (2006).
pubmed: 16548008 doi: 10.1002/pd.1433
Baffero, G. M. et al. Confined placental mosaicism at chorionic villous sampling: risk factors and pregnancy outcome. Prenat. Diagn. 32, 1102–1108 (2012).
pubmed: 22961322 doi: 10.1002/pd.3965
Toutain, J., Goutte-Gattat, D., Horovitz, J. & Saura, R. Confined placental mosaicism revisited: impact on pregnancy characteristics and outcome. PLoS ONE 13, e0195905 (2018).
pubmed: 29649318 pmcid: 5897023 doi: 10.1371/journal.pone.0195905
Grati, F. R. et al. Outcomes in pregnancies with a confined placental mosaicism and implications for prenatal screening using cell-free DNA. Genet. Med. 22, 309–316 (2020).
pubmed: 31391534 doi: 10.1038/s41436-019-0630-y
Henderson, K. G. et al. Distribution of mosaicism in human placentae. Hum. Genet. 97, 650–654 (1996).
pubmed: 8655147 doi: 10.1007/BF02281877
Peñaherrera, M. S. et al. Patterns of placental development evaluated by X chromosome inactivation profiling provide a basis to evaluate the origin of epigenetic variation. Hum. Reprod. 27, 1745–1753 (2012).
pubmed: 22431562 pmcid: 3357192 doi: 10.1093/humrep/des072
Moreira de Mello, J. C. et al. Random X inactivation and extensive mosaicism in human placenta revealed by analysis of allele-specific gene expression along the X chromosome. PLoS ONE 5, e10947 (2010).
pubmed: 20532033 pmcid: 2881032 doi: 10.1371/journal.pone.0010947
Sovio, U., White, I. R., Dacey, A., Pasupathy, D. & Smith, G. C. S. Screening for fetal growth restriction with universal third trimester ultrasonography in nulliparous women in the Pregnancy Outcome Prediction (POP) study: a prospective cohort study. Lancet 386, 2089–2097 (2015).
pubmed: 26360240 pmcid: 4655320 doi: 10.1016/S0140-6736(15)00131-2
Li, H. & Durbin, R. Fast and accurate short read alignment with Burrows–Wheeler transform. Bioinformatics 25, 1754–1760 (2009).
pubmed: 19451168 pmcid: 2705234
Jones, D. et al. cgpCaVEManWrapper: simple execution of CaVEMan in order to detect somatic single nucleotide variants in NGS data. Curr. Protoc. Bioinformatics 56, 15.10.1–15.10.18 (2016).
Ye, K., Schulz, M. H., Long, Q., Apweiler, R. & Ning, Z. Pindel: a pattern growth approach to detect break points of large deletions and medium sized insertions from paired-end short reads. Bioinformatics 25, 2865–2871 (2009).
pubmed: 19561018 pmcid: 2781750 doi: 10.1093/bioinformatics/btp394
Van Loo, P. et al. Allele-specific copy number analysis of tumors. Proc. Natl Acad. Sci. USA 107, 16910–16915 (2010).
pubmed: 20837533 pmcid: 2947907 doi: 10.1073/pnas.1009843107
Buels, R. et al. JBrowse: a dynamic web platform for genome visualization and analysis. Genome Biol. 17, 66 (2016).
pubmed: 27072794 pmcid: 4830012 doi: 10.1186/s13059-016-0924-1
Coorens, T. H. H. et al. Lineage-independent tumors in bilateral neuroblastoma. N. Engl. J. Med. 383, 1860–1865 (2020).
pubmed: 33211929 pmcid: 7611571 doi: 10.1056/NEJMoa2000962
Olafsson, S. et al. Somatic evolution in non-neoplastic IBD-affected colon. Cell 182, 672–684.e11 (2020).
pubmed: 32697969 pmcid: 7427325 doi: 10.1016/j.cell.2020.06.036
Robinson, P. S. et al. Elevated somatic mutation burdens in normal human cells due to defective DNA polymerases. Preprint at https://doi.org/10.1101/2020.06.23.167668 (2020).
Hoang, D. T. et al. MPBoot: fast phylogenetic maximum parsimony tree inference and bootstrap approximation. BMC Evol. Biol. 18, 11 (2018).
pubmed: 29390973 pmcid: 5796505 doi: 10.1186/s12862-018-1131-3
Rosenthal, R., McGranahan, N., Herrero, J., Taylor, B. S. & Swanton, C. DeconstructSigs: delineating mutational processes in single tumors distinguishes DNA repair deficiencies and patterns of carcinoma evolution. Genome Biol. 17, 31 (2016).
pubmed: 26899170 pmcid: 4762164 doi: 10.1186/s13059-016-0893-4

Auteurs

Tim H H Coorens (THH)

Wellcome Sanger Institute, Hinxton, UK.

Thomas R W Oliver (TRW)

Wellcome Sanger Institute, Hinxton, UK.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Rashesh Sanghvi (R)

Wellcome Sanger Institute, Hinxton, UK.

Ulla Sovio (U)

Department of Obstetrics and Gynaecology, University of Cambridge, NIHR Cambridge Biomedical Research Centre, Cambridge, UK.

Emma Cook (E)

Department of Obstetrics and Gynaecology, University of Cambridge, NIHR Cambridge Biomedical Research Centre, Cambridge, UK.

Roser Vento-Tormo (R)

Wellcome Sanger Institute, Hinxton, UK.

Muzlifah Haniffa (M)

Wellcome Sanger Institute, Hinxton, UK.
Institute of Cellular Medicine, Newcastle University, Newcastle upon Tyne, UK.
Department of Dermatology, Royal Victoria Infirmary, Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK.

Matthew D Young (MD)

Wellcome Sanger Institute, Hinxton, UK.

Raheleh Rahbari (R)

Wellcome Sanger Institute, Hinxton, UK.

Neil Sebire (N)

Great Ormond Street Hospital for Children NHS Foundation Trust, NIHR Great Ormond Street Hospital Biomedical Research Centre, London, UK.
UCL Great Ormond Street Institute of Child Health, London, UK.

Peter J Campbell (PJ)

Wellcome Sanger Institute, Hinxton, UK.

D Stephen Charnock-Jones (DS)

Department of Obstetrics and Gynaecology, University of Cambridge, NIHR Cambridge Biomedical Research Centre, Cambridge, UK. dscj1@cam.ac.uk.
Centre for Trophoblast Research, Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK. dscj1@cam.ac.uk.

Gordon C S Smith (GCS)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK. gcss2@cam.ac.uk.
Department of Obstetrics and Gynaecology, University of Cambridge, NIHR Cambridge Biomedical Research Centre, Cambridge, UK. gcss2@cam.ac.uk.
Centre for Trophoblast Research, Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK. gcss2@cam.ac.uk.

Sam Behjati (S)

Wellcome Sanger Institute, Hinxton, UK. sb31@sanger.ac.uk.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK. sb31@sanger.ac.uk.
Department of Paediatrics, University of Cambridge, Cambridge, UK. sb31@sanger.ac.uk.

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