The influence of comorbid depression and overweight status on peripheral inflammation and cortisol levels.


Journal

Psychological medicine
ISSN: 1469-8978
Titre abrégé: Psychol Med
Pays: England
ID NLM: 1254142

Informations de publication

Date de publication:
10 2022
Historique:
pubmed: 19 3 2021
medline: 5 1 2023
entrez: 18 3 2021
Statut: ppublish

Résumé

Depression and overweight are each associated with abnormal immune system activation. We sought to disentangle the extent to which depressive symptoms and overweight status contributed to increased inflammation and abnormal cortisol levels. Participants were recruited through the Wellcome Trust NIMA Consortium. The sample of 216 participants consisted of 69 overweight patients with depression; 35 overweight controls; 55 normal-weight patients with depression and 57 normal-weight controls. Peripheral inflammation was measured as high-sensitivity C-Reactive Protein (hsCRP) in serum. Salivary cortisol was collected at multiple points throughout the day to measure cortisol awakening response and diurnal cortisol levels. Overweight patients with depression had significantly higher hsCRP compared with overweight controls ( Comorbid depression and overweight status are associated with increased hsCRP, and the coexistence of these conditions amplified the risk of clinically elevated hsCRP levels. Overweight status contributed most to the risk of clinically elevated hsCRP levels, but depression also contributed to a significant risk. We observed no differences in cortisol levels between groups.

Sections du résumé

BACKGROUND
Depression and overweight are each associated with abnormal immune system activation. We sought to disentangle the extent to which depressive symptoms and overweight status contributed to increased inflammation and abnormal cortisol levels.
METHODS
Participants were recruited through the Wellcome Trust NIMA Consortium. The sample of 216 participants consisted of 69 overweight patients with depression; 35 overweight controls; 55 normal-weight patients with depression and 57 normal-weight controls. Peripheral inflammation was measured as high-sensitivity C-Reactive Protein (hsCRP) in serum. Salivary cortisol was collected at multiple points throughout the day to measure cortisol awakening response and diurnal cortisol levels.
RESULTS
Overweight patients with depression had significantly higher hsCRP compared with overweight controls (
CONCLUSION
Comorbid depression and overweight status are associated with increased hsCRP, and the coexistence of these conditions amplified the risk of clinically elevated hsCRP levels. Overweight status contributed most to the risk of clinically elevated hsCRP levels, but depression also contributed to a significant risk. We observed no differences in cortisol levels between groups.

Identifiants

pubmed: 33731235
doi: 10.1017/S0033291721000088
pii: S0033291721000088
pmc: PMC9693673
doi:

Substances chimiques

Hydrocortisone WI4X0X7BPJ
C-Reactive Protein 9007-41-4

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3289-3296

Subventions

Organisme : Medical Research Council
ID : MR/L014815/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_G0802534
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/J002739/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : G108/603
Pays : United Kingdom
Organisme : MRF
ID : MRF_MRF-160-0005-ELP-MONDE
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/N029488/1
Pays : United Kingdom

Auteurs

Anna P McLaughlin (AP)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.
National Institute for Health Research (NIHR) Maudsley Biomedical Research Centre, South London and Maudsley NHS Foundation Trust, King's College London, London, UK.

Naghmeh Nikkheslat (N)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Caitlin Hastings (C)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Maria A Nettis (MA)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.
National Institute for Health Research (NIHR) Maudsley Biomedical Research Centre, South London and Maudsley NHS Foundation Trust, King's College London, London, UK.

Melisa Kose (M)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Courtney Worrell (C)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Zuzanna Zajkowska (Z)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Nicole Mariani (N)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Daniela Enache (D)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Giulia Lombardo (G)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.

Linda Pointon (L)

Department of Psychiatry, University of Cambridge, Cambridge, UK.

Philip Cowen (P)

University Department of Psychiatry, Warneford Hospital, Oxford, UK.

Jonathan Cavanagh (J)

Mental Health and Wellbeing, Sackler Institute, Neurology block, Queen Elizabeth University hospital, Glasgow, UK.

Neil Harrison (N)

Division of Psychological Medicine and Clinical Sciences, Cardiff University Brain Research Imaging Centre (CUBRIC), Cardiff, UK.

Edward Bullmore (E)

Department of Psychiatry, University of Cambridge, Cambridge, UK.

Carmine M Pariante (CM)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.
National Institute for Health Research (NIHR) Maudsley Biomedical Research Centre, South London and Maudsley NHS Foundation Trust, King's College London, London, UK.

Valeria Mondelli (V)

Department of Psychological Medicine, King's College London, Institute of Psychiatry, Psychology and Neuroscience, London, UK.
National Institute for Health Research (NIHR) Maudsley Biomedical Research Centre, South London and Maudsley NHS Foundation Trust, King's College London, London, UK.

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Classifications MeSH