Platelet Count Rose While D-Dimer Levels Dropped as Deaths and Thrombosis Declined-An Observational Study on Anticoagulation Shift in COVID-19.


Journal

Thrombosis and haemostasis
ISSN: 2567-689X
Titre abrégé: Thromb Haemost
Pays: Germany
ID NLM: 7608063

Informations de publication

Date de publication:
Dec 2021
Historique:
pubmed: 9 4 2021
medline: 15 12 2021
entrez: 8 4 2021
Statut: ppublish

Résumé

 High levels of D-dimer and low platelet counts are associated with poor outcome in coronavirus disease 2019 (COVID-19). As anticoagulation appeared to improve survival, hospital-wide recommendations regarding higher doses of anticoagulation were implemented on April 9, 2020.  To investigate if trends in D-dimer levels and platelet counts were associated with death, thrombosis, and the shift in anticoagulation.  Retrospective cohort study of 429 patients with COVID-19 at Karolinska University Hospital. Information on D-dimer levels and platelet counts was obtained from laboratory databases and clinical data from medical records.  Thirty-day mortality and thrombosis rates were 19% and 18%, respectively. Pulmonary embolism was common, 65/83 (78%). Increased D-dimer levels in the first week in hospital were significantly associated with death and thrombosis (odds ratio [OR]: 6.06; 95% confidence interval [CL]: 2.10-17.5 and 3.11; 95% CI: 1.20-8.10, respectively). If platelet count increased more than 35 × 10  In contrast to D-dimer levels, increase of platelet count over the first week in hospital was associated with improved survival and reduced thrombotic risk. The daily mean levels of D-dimer dropped while the platelet counts rose, coinciding with increased anticoagulation and a decline in thrombotic burden and mortality.

Sections du résumé

BACKGROUND BACKGROUND
 High levels of D-dimer and low platelet counts are associated with poor outcome in coronavirus disease 2019 (COVID-19). As anticoagulation appeared to improve survival, hospital-wide recommendations regarding higher doses of anticoagulation were implemented on April 9, 2020.
OBJECTIVES OBJECTIVE
 To investigate if trends in D-dimer levels and platelet counts were associated with death, thrombosis, and the shift in anticoagulation.
METHODS METHODS
 Retrospective cohort study of 429 patients with COVID-19 at Karolinska University Hospital. Information on D-dimer levels and platelet counts was obtained from laboratory databases and clinical data from medical records.
RESULTS RESULTS
 Thirty-day mortality and thrombosis rates were 19% and 18%, respectively. Pulmonary embolism was common, 65/83 (78%). Increased D-dimer levels in the first week in hospital were significantly associated with death and thrombosis (odds ratio [OR]: 6.06; 95% confidence interval [CL]: 2.10-17.5 and 3.11; 95% CI: 1.20-8.10, respectively). If platelet count increased more than 35 × 10
CONCLUSION CONCLUSIONS
 In contrast to D-dimer levels, increase of platelet count over the first week in hospital was associated with improved survival and reduced thrombotic risk. The daily mean levels of D-dimer dropped while the platelet counts rose, coinciding with increased anticoagulation and a decline in thrombotic burden and mortality.

Identifiants

pubmed: 33831964
doi: 10.1055/a-1477-3829
doi:

Substances chimiques

Anticoagulants 0
Biomarkers 0
Fibrin Fibrinogen Degradation Products 0
fibrin fragment D 0

Types de publication

Journal Article Multicenter Study Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1610-1621

Informations de copyright

Thieme. All rights reserved.

Déclaration de conflit d'intérêts

J.A. has received research grants from Shire, honoraria from Stago, Siemens, Sysmex, Roche, Baxter, and Sobi, and acts on advisory boards for Sobi and Novo Nordisk. M.B. acts on the advisory board for CSL Behringer and Sobi and has had consultant assignments for Novo Nordisk and received lecturer honoraria from Sobi.A.O. and M.B. were both supported by funds from Stockholm County Council. A.O. was supported by Swedish Carnegie Hero Funds. A.S., J.D.W., A.W., A.Å., and M.M. declare no competing financial interests.

Auteurs

Anna Sjöström (A)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Clinical Chemistry, Karolinska University Laboratory, Stockholm, Sweden.

Johanna Dehlsen Wersäll (JD)

Clinical Chemistry, Karolinska University Laboratory, Stockholm, Sweden.

Anna Warnqvist (A)

Division of Biostatistics, Institute of Environmental Medicine, Karolinska Institutet, Stockholm, Sweden.

Maria Farm (M)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Clinical Chemistry, Karolinska University Laboratory, Stockholm, Sweden.

Maria Magnusson (M)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Department of Clinical Science, Intervention and Technology, Karolinska Institutet, Stockholm, Sweden.
Coagulation Unit, Department of Hematology, Karolinska University Hospital, Stockholm, Sweden.

Anders Oldner (A)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.
Department of Perioperative Medicine and Intensive Care, Karolinska University Hospital, Stockholm, Sweden.

Anna Ågren (A)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Coagulation Unit, Department of Hematology, Karolinska University Hospital, Stockholm, Sweden.
Department of Clinical Sciences, Danderyd Hospital, Karolinska Institutet, Stockholm, Sweden.

Jovan Antovic (J)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Clinical Chemistry, Karolinska University Laboratory, Stockholm, Sweden.

Maria Bruzelius (M)

Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden.
Coagulation Unit, Department of Hematology, Karolinska University Hospital, Stockholm, Sweden.
Department of Medicine, Solna, Karolinska Institutet, Stockholm, Sweden.

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Classifications MeSH