TREM2 is a receptor for non-glycosylated mycolic acids of mycobacteria that limits anti-mycobacterial macrophage activation.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
16 04 2021
Historique:
received: 22 06 2020
accepted: 17 03 2021
entrez: 17 4 2021
pubmed: 18 4 2021
medline: 4 5 2021
Statut: epublish

Résumé

Mycobacterial cell-wall glycolipids elicit an anti-mycobacterial immune response via FcRγ-associated C-type lectin receptors, including Mincle, and caspase-recruitment domain family member 9 (CARD9). Additionally, mycobacteria harbor immuno-evasive cell-wall lipids associated with virulence and latency; however, a mechanism of action is unclear. Here, we show that the DAP12-associated triggering receptor expressed on myeloid cells 2 (TREM2) recognizes mycobacterial cell-wall mycolic acid (MA)-containing lipids and suggest a mechanism by which mycobacteria control host immunity via TREM2. Macrophages respond to glycosylated MA-containing lipids in a Mincle/FcRγ/CARD9-dependent manner to produce inflammatory cytokines and recruit inducible nitric oxide synthase (iNOS)-positive mycobactericidal macrophages. Conversely, macrophages respond to non-glycosylated MAs in a TREM2/DAP12-dependent but CARD9-independent manner to recruit iNOS-negative mycobacterium-permissive macrophages. Furthermore, TREM2 deletion enhances Mincle-induced macrophage activation in vitro and inflammation in vivo and accelerates the elimination of mycobacterial infection, suggesting that TREM2-DAP12 signaling counteracts Mincle-FcRγ-CARD9-mediated anti-mycobacterial immunity. Mycobacteria, therefore, harness TREM2 for immune evasion.

Identifiants

pubmed: 33863908
doi: 10.1038/s41467-021-22620-3
pii: 10.1038/s41467-021-22620-3
pmc: PMC8052348
doi:

Substances chimiques

Adaptor Proteins, Signal Transducing 0
CARD Signaling Adaptor Proteins 0
Card9 protein, mouse 0
Clecsf8 protein, mouse 0
Glycolipids 0
Lectins, C-Type 0
Membrane Glycoproteins 0
Membrane Proteins 0
Mycolic Acids 0
Receptors, IgG 0
Receptors, Immunologic 0
Trem2 protein, mouse 0
Tyrobp protein, mouse 0
Virulence Factors 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2299

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Auteurs

Ei'ichi Iizasa (E)

Department of Immunology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan.

Yasushi Chuma (Y)

Research and Development Department, Japan BCG Laboratory, Tokyo, Japan.

Takayuki Uematsu (T)

Biomedical Laboratory, Division of Biomedical Research, Kitasato University Medical Center, Kitamoto, Saitama, Japan.

Mio Kubota (M)

Division of Molecular and Cellular Immunoscience, Department of Biomolecular Sciences, Faculty of Medicine, Saga University, Saga, Japan.

Hiroaki Kawaguchi (H)

Department of Hygiene and Health Promotion Medicine, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan.

Masayuki Umemura (M)

Tropical Biosphere Research Center, University of the Ryukyus, Nishihara, Okinawa, Japan.

Kenji Toyonaga (K)

Department of Immunology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan.
Department of Molecular Immunology, Division of Host Defense, Research Institute for Microbial Disease, Osaka University, Osaka, Japan.

Hideyasu Kiyohara (H)

Research and Development Department, Japan BCG Laboratory, Tokyo, Japan.

Ikuya Yano (I)

Research and Development Department, Japan BCG Laboratory, Tokyo, Japan.
Faculty of Medicine, Osaka City University Graduate School of Medicine, Osaka, Japan.

Marco Colonna (M)

Department of Pathology and Immunology, BJC Institute of Health at Washington University, St. Louis, MO, USA.

Masahiko Sugita (M)

Laboratory of Cell Regulation, Institute for Virus Research, Graduate School of Biostudies, Kyoto University, Kyoto, Japan.

Goro Matsuzaki (G)

Tropical Biosphere Research Center, University of the Ryukyus, Nishihara, Okinawa, Japan.

Sho Yamasaki (S)

Department of Molecular Immunology, Division of Host Defense, Research Institute for Microbial Disease, Osaka University, Osaka, Japan.

Hiroki Yoshida (H)

Division of Molecular and Cellular Immunoscience, Department of Biomolecular Sciences, Faculty of Medicine, Saga University, Saga, Japan.

Hiromitsu Hara (H)

Department of Immunology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan. harah@m2.kufm.kagoshima-u.ac.jp.

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