Acetyl-L-Carnitine Ameliorates Metabolic and Endocrine Alterations in Women with PCOS: A Double-Blind Randomized Clinical Trial.


Journal

Advances in therapy
ISSN: 1865-8652
Titre abrégé: Adv Ther
Pays: United States
ID NLM: 8611864

Informations de publication

Date de publication:
07 2021
Historique:
received: 01 03 2021
accepted: 13 05 2021
pubmed: 29 5 2021
medline: 6 8 2021
entrez: 28 5 2021
Statut: ppublish

Résumé

Polycystic ovary syndrome (PCOS) is a common endocrine-metabolic disorder and the main cause of infertility in women of reproductive age. Affected women suffer from insulin resistance and present with an intense stress response. Treatment with insulin sensitizers alone and in combination is used to ameliorate the signs and symptoms associated with the disease. This study was designed to compare the endocrine and metabolic parameters as well as subjective and objective measures of stress in women with PCOS before and after treatment with acetyl-L-carnitine (ALC) and metformin plus pioglitazone. A total of 147 women with PCOS were randomly assigned into two groups: the combo group (n = 72) received a combination of metformin, pioglitazone, and ALC (500 mg, 15 mg, and 1500 mg, respectively), twice daily; the Met + Pio group (n = 75) received metformin plus pioglitazone (500 mg, 15 mg, respectively) and placebo (citric acid plus calcium carbonate), twice daily for 12 weeks. Medications were discontinued when pregnancy was confirmed. The Perceived Stress Scale (PSS14) and Profile of Mood States (POMS) were employed as subjective measures of stress. The endocrine and metabolic functions of women with PCOS were assessed by measuring insulin, leutinizing hormone (LH), follicle-stimulating hormone (FSH), testosterone, and adiponectin levels in fasting blood samples. Insulin resistance was calculated by Homeostatic Model Assessment of Insulin Resistance (HOMA-IR). Women at baseline had significantly elevated circulating concentration of insulin and low level of adiponectin. Treatment decreased insulin in both groups; however, the combo group showed a significant decrease (p = 0.001). Serum adiponectin level was raised significantly after treatment in both groups (p < 0.001). HOMA-IR also decreased in both groups (both p < 0.001). Testosterone, FSH, and LH significantly improved in both groups. LH also decreased in both groups; however, the change was significant only in the combo (metformin plus pioglitazone plus ALC) group (p = 0.013). Interestingly, there was a significant improvement in body circumference (p < 0.001) in the combo group. The PSS scores of the patients improved significantly (p < 0.001) in the combo group. Interestingly, regular menstrual cycles were found (97.2%) in the carnitine group, but in only 12.9% of the other group. We conclude that addition of ALC therapy is superior to metformin plus pioglitazone in ameliorating insulin resistance, polycystic ovaries, menstrual irregularities, and hypoadiponectinemia in women with PCOS. Trial registration: clinicalTrial.gov NCT04113889. Registered 3 October, 2019. https://clinicaltrials.gov/ct2/show/NCT04113889 .

Identifiants

pubmed: 34047916
doi: 10.1007/s12325-021-01789-5
pii: 10.1007/s12325-021-01789-5
doi:

Substances chimiques

Hypoglycemic Agents 0
Insulin 0
Acetylcarnitine 6DH1W9VH8Q
Metformin 9100L32L2N

Banques de données

ClinicalTrials.gov
['NCT04113889']

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Pagination

3842-3856

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Healthcare Ltd., part of Springer Nature.

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Auteurs

Saman Tauqir (S)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Momina Israr (M)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Bushra Rauf (B)

Department of Gynecology and Obstetrics, Hayatabad Medical Complex, Peshawar, Pakistan.

Muhammad Omar Malik (MO)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Syed Hamid Habib (SH)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Fawad Ali Shah (FA)

Department of Pharmacology, Riphah Institute of Pharmaceutical Sciences, Riphah International University, Islamabad, Pakistan.

Muhammad Usman (M)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Muhammad Asif Raza (MA)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Inayat Shah (I)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Haroon Badshah (H)

Department of Pharmacology, Riphah Institute of Pharmaceutical Sciences, Riphah International University, Islamabad, Pakistan.

Ehtesham Ehtesham (E)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan.

Mohsin Shah (M)

Department of Physiology, Institute of Basic Medical Sciences, Khyber Medical University, Peshawar, Pakistan. mohsin.ibms@kmu.edu.pk.

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