Treacle and TOPBP1 control replication stress response in the nucleolus.


Journal

The Journal of cell biology
ISSN: 1540-8140
Titre abrégé: J Cell Biol
Pays: United States
ID NLM: 0375356

Informations de publication

Date de publication:
02 08 2021
Historique:
received: 16 08 2020
revised: 26 04 2021
accepted: 18 05 2021
entrez: 8 6 2021
pubmed: 9 6 2021
medline: 21 10 2021
Statut: ppublish

Résumé

Replication stress is one of the main sources of genome instability. Although the replication stress response in eukaryotic cells has been extensively studied, almost nothing is known about the replication stress response in nucleoli. Here, we demonstrate that initial replication stress-response factors, such as RPA, TOPBP1, and ATR, are recruited inside the nucleolus in response to drug-induced replication stress. The role of TOPBP1 goes beyond the typical replication stress response; it interacts with the low-complexity nucleolar protein Treacle (also referred to as TCOF1) and forms large Treacle-TOPBP1 foci inside the nucleolus. In response to replication stress, Treacle and TOPBP1 facilitate ATR signaling at stalled replication forks, reinforce ATR-mediated checkpoint activation inside the nucleolus, and promote the recruitment of downstream replication stress response proteins inside the nucleolus without forming nucleolar caps. Characterization of the Treacle-TOPBP1 interaction mode leads us to propose that these factors can form a molecular platform for efficient stress response in the nucleolus.

Identifiants

pubmed: 34100862
pii: 212262
doi: 10.1083/jcb.202008085
pmc: PMC8190600
pii:
doi:

Substances chimiques

Carrier Proteins 0
DNA, Ribosomal 0
DNA-Binding Proteins 0
Nuclear Proteins 0
Phosphoproteins 0
TCOF1 protein, human 0
TOPBP1 protein, human 0
Aphidicolin 38966-21-1
ATR protein, human EC 2.7.11.1
Ataxia Telangiectasia Mutated Proteins EC 2.7.11.1
Hydroxyurea X6Q56QN5QC

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2021 Velichko et al.

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Auteurs

Artem K Velichko (AK)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.
Center for Precision Genome Editing and Genetic Technologies for Biomedicine, Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.
Institute for Translational Medicine and Biotechnology, Sechenov First Moscow State Medical University, Moscow, Russia.

Natalia Ovsyannikova (N)

A.N. Belozersky Institute of Physico-Chemical Biology, Lomonosov Moscow State University, Moscow, Russia.

Nadezhda V Petrova (NV)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.

Artem V Luzhin (AV)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.
Center for Precision Genome Editing and Genetic Technologies for Biomedicine, Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.

Maria Vorobjeva (M)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.

Alexey S Gavrikov (AS)

Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry Russian Academy of Sciences, Moscow, Russia.

Alexander S Mishin (AS)

Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry Russian Academy of Sciences, Moscow, Russia.

Igor I Kireev (II)

A.N. Belozersky Institute of Physico-Chemical Biology, Lomonosov Moscow State University, Moscow, Russia.
V.I. Kulakov National Medical Research Center for Obstetrics, Gynecology, and Perinatology, Moscow, Russia.

Sergey V Razin (SV)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.

Omar L Kantidze (OL)

Institute of Gene Biology Russian Academy of Sciences, Moscow, Russia.

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Classifications MeSH