Effect of leukoreduction and temperature on risk of bacterial growth in CPDA-1 whole blood: A study of Escherichia coli.


Journal

Transfusion
ISSN: 1537-2995
Titre abrégé: Transfusion
Pays: United States
ID NLM: 0417360

Informations de publication

Date de publication:
07 2021
Historique:
revised: 01 02 2021
received: 02 12 2020
accepted: 01 02 2021
entrez: 16 7 2021
pubmed: 17 7 2021
medline: 17 8 2021
Statut: ppublish

Résumé

Collection of non-leukoreduced citrate-phosphate-dextrose-adenine (CPDA-1) whole blood is performed in walking blood banks. Blood collected under field conditions may have increased risk of bacterial contamination. This study was conducted to examine the effects of WBC reduction and storage temperature on growth of Escherichia coli (ATCC® 25922™) in CPDA-1 whole blood. CPDA-1 whole blood of 450 ml from 10 group O donors was inoculated with E. coli. Two hours after inoculation, the test bags were leukoreduced with a platelet-sparing filter. The control bags remained unfiltered. Each whole blood bag was then split into three smaller bags for further storage at 2-6°C, 20-24°C, or 33-37°C. Bacterial growth was quantified immediately, 2 and 3 h after inoculation, on days 1, 3, 7, and 14 for all storage temperatures, and on days 21 and 35 for storage at 2-6°C. Whole blood was inoculated with a median of 19.5 (range 12.0-32.0) colony-forming units per ml (CFU/ml) E. coli. After leukoreduction, a median of 3.3 CFU/ml (range 0.0-33.3) E. coli remained. In the control arm, the WBCs phagocytized E. coli within 24 h at 20-24°C and 33-37°C in 9 of 10 bags. During storage at 2-6°C, a slow self-sterilization occurred over time with and without leukoreduction. Storage at 20-24°C and 33-37°C for up to 24 h before leukoreduction reduces the risk of E. coli-contamination in CPDA-1 whole blood. Subsequent storage at 2-6°C will further reduce the growth of E. coli.

Sections du résumé

BACKGROUND
Collection of non-leukoreduced citrate-phosphate-dextrose-adenine (CPDA-1) whole blood is performed in walking blood banks. Blood collected under field conditions may have increased risk of bacterial contamination. This study was conducted to examine the effects of WBC reduction and storage temperature on growth of Escherichia coli (ATCC® 25922™) in CPDA-1 whole blood.
METHODS
CPDA-1 whole blood of 450 ml from 10 group O donors was inoculated with E. coli. Two hours after inoculation, the test bags were leukoreduced with a platelet-sparing filter. The control bags remained unfiltered. Each whole blood bag was then split into three smaller bags for further storage at 2-6°C, 20-24°C, or 33-37°C. Bacterial growth was quantified immediately, 2 and 3 h after inoculation, on days 1, 3, 7, and 14 for all storage temperatures, and on days 21 and 35 for storage at 2-6°C.
RESULTS
Whole blood was inoculated with a median of 19.5 (range 12.0-32.0) colony-forming units per ml (CFU/ml) E. coli. After leukoreduction, a median of 3.3 CFU/ml (range 0.0-33.3) E. coli remained. In the control arm, the WBCs phagocytized E. coli within 24 h at 20-24°C and 33-37°C in 9 of 10 bags. During storage at 2-6°C, a slow self-sterilization occurred over time with and without leukoreduction.
CONCLUSIONS
Storage at 20-24°C and 33-37°C for up to 24 h before leukoreduction reduces the risk of E. coli-contamination in CPDA-1 whole blood. Subsequent storage at 2-6°C will further reduce the growth of E. coli.

Identifiants

pubmed: 34269444
doi: 10.1111/trf.16499
doi:

Substances chimiques

Citrates 0
citrate phosphate dextrose 51404-37-6
Glucose IY9XDZ35W2
Adenine JAC85A2161

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

S80-S89

Informations de copyright

© 2021 The Authors. Transfusion published by Wiley Periodicals LLC. on behalf of AABB.

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Auteurs

Hanne Braathen (H)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.
Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway.

Joar Sivertsen (J)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.
Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway.

Turid Helen Felli Lunde (THF)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.

Geir Strandenes (G)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.
Department of War Surgery and Emergency Medicine, Norwegian Armed Forces Medical Services, Oslo, Norway.

Paul Christoffer Lindemann (PC)

Department of Microbiology, Haukeland University Hospital, Bergen, Norway.

Jörg Assmus (J)

Centre for Clinical Research, Haukeland University Hospital, Bergen, Norway.

Tor Audun Hervig (TA)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.
Department of Clinical Sciences, University of Bergen, Bergen, Norway.
Department of Immunology and Transfusion Medicine, Haugesund Hospital, Haugesund, Norway.

Torunn Oveland Apelseth (TO)

Department of Immunology and Transfusion Medicine, Haukeland University Hospital, Bergen, Norway.
Department of War Surgery and Emergency Medicine, Norwegian Armed Forces Medical Services, Oslo, Norway.

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