Multinephron Segment Diuretic Therapy to Overcome Diuretic Resistance in Acute Heart Failure: A Single-Center Experience.


Journal

Journal of cardiac failure
ISSN: 1532-8414
Titre abrégé: J Card Fail
Pays: United States
ID NLM: 9442138

Informations de publication

Date de publication:
01 2022
Historique:
received: 19 04 2021
revised: 14 07 2021
accepted: 19 07 2021
pubmed: 18 8 2021
medline: 1 3 2022
entrez: 17 8 2021
Statut: ppublish

Résumé

The concept of multinephron segment diuretic therapy (MSDT) has been recommended in severe diuretic resistance with only expert opinion and case-level evidence. The purpose of this study was to investigate the safety and efficacy of MSDT, combining 4 diuretic classes, in acute heart failure (AHF) complicated by diuretic resistance. A retrospective analysis was conducted in patients hospitalized with AHF at a single medical center who received MSDT, including concomitant carbonic anhydrase inhibitor, loop, thiazide, and mineralocorticoid receptor antagonist diuretics. Subjects served as their own controls with efficacy evaluated as urine output and weight change before and after MSDT. Serum chemistries, renal replacement therapies, and in-hospital mortality were evaluated for safety. Patients with severe diuretic resistance before MSDT were analyzed as a subcohort. A total of 167 patients with AHF and diuretic resistance received MSDT. MSDT was associated with increased median 24-hour urine output in the first day of therapy compared with the previous day (2.16 L [0.95-4.14 L] to 3.08 L [1.74-4.86 L], P = .003) in the total cohort and in the Severe diuretic resistance cohort (0.91 L [0.43-1.43 L] to 2.08 L [1.13-3.96 L], P < .001). The median cumulative weight loss at day 7 or discharge was -7.4 kg (-15.3 to -3.4 kg) (P = .02). Neither serum sodium, chloride, potassium, bicarbonate, or creatinine changed significantly relative to baseline (P > .05 for all). In an AHF cohort with diuretic resistance, MSDT was associated with increased diuresis without changes in serum chemistries or kidney function. Prospective studies of MSDT in AHF and diuretic resistance are warranted.

Sections du résumé

BACKGROUND
The concept of multinephron segment diuretic therapy (MSDT) has been recommended in severe diuretic resistance with only expert opinion and case-level evidence. The purpose of this study was to investigate the safety and efficacy of MSDT, combining 4 diuretic classes, in acute heart failure (AHF) complicated by diuretic resistance.
METHODS AND RESULTS
A retrospective analysis was conducted in patients hospitalized with AHF at a single medical center who received MSDT, including concomitant carbonic anhydrase inhibitor, loop, thiazide, and mineralocorticoid receptor antagonist diuretics. Subjects served as their own controls with efficacy evaluated as urine output and weight change before and after MSDT. Serum chemistries, renal replacement therapies, and in-hospital mortality were evaluated for safety. Patients with severe diuretic resistance before MSDT were analyzed as a subcohort. A total of 167 patients with AHF and diuretic resistance received MSDT. MSDT was associated with increased median 24-hour urine output in the first day of therapy compared with the previous day (2.16 L [0.95-4.14 L] to 3.08 L [1.74-4.86 L], P = .003) in the total cohort and in the Severe diuretic resistance cohort (0.91 L [0.43-1.43 L] to 2.08 L [1.13-3.96 L], P < .001). The median cumulative weight loss at day 7 or discharge was -7.4 kg (-15.3 to -3.4 kg) (P = .02). Neither serum sodium, chloride, potassium, bicarbonate, or creatinine changed significantly relative to baseline (P > .05 for all).
CONCLUSIONS
In an AHF cohort with diuretic resistance, MSDT was associated with increased diuresis without changes in serum chemistries or kidney function. Prospective studies of MSDT in AHF and diuretic resistance are warranted.

Identifiants

pubmed: 34403831
pii: S1071-9164(21)00333-X
doi: 10.1016/j.cardfail.2021.07.016
pii:
doi:

Substances chimiques

Diuretics 0
Mineralocorticoid Receptor Antagonists 0
Sodium Potassium Chloride Symporter Inhibitors 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

21-31

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

Zachary L Cox (ZL)

Department of Pharmacy Practice, Lipscomb University College of Pharmacy, Nashville, Tennessee; Department of Pharmacy, Vanderbilt University Medical Center, Nashville, Tennessee. Electronic address: Zachary.l.cox@vumc.org.

Bonnie Ann Sarrell (BA)

Division of Nephrology and Hypertension, Vanderbilt University Medical Center, Nashville, Tennessee.

Mary Katherine Cella (MK)

Department of Pharmacy Practice, Lipscomb University College of Pharmacy, Nashville, Tennessee.

Brent Tucker (B)

Department of Pharmacy Practice, Lipscomb University College of Pharmacy, Nashville, Tennessee.

Juan P Arroyo (JP)

Division of Nephrology and Hypertension, Vanderbilt University Medical Center, Nashville, Tennessee.

Kausik Umanath (K)

Division of Nephrology and Hypertension, Henry Ford Hospital, Detroit, Michigan; Division of Nephrology and Hypertension, Wayne State University, Detroit, Michigan.

William Tidwell (W)

Department of Pharmacy, Vanderbilt University Medical Center, Nashville, Tennessee.

Andrew Guide (A)

Department of Biostatistics, Vanderbilt University Medical Center, Nashville, Tennessee.

Jeffrey M Testani (JM)

Division of Internal Medicine, Section of Cardiovascular Medicine, Yale University School of Medicine, New Haven, Connecticut.

Julia B Lewis (JB)

Division of Nephrology and Hypertension, Vanderbilt University Medical Center, Nashville, Tennessee.

Jamie P Dwyer (JP)

Division of Nephrology and Hypertension, Vanderbilt University Medical Center, Nashville, Tennessee.

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