Vitamin D Inhibits IL-22 Production Through a Repressive Vitamin D Response Element in the
Basic Helix-Loop-Helix Transcription Factors
/ metabolism
Binding Sites
Biomarkers
Cell Line
Cytokines
/ biosynthesis
Gene Expression Regulation
/ drug effects
Humans
Inflammation Mediators
/ metabolism
Interleukins
/ biosynthesis
Nuclear Receptor Subfamily 1, Group F, Member 3
/ metabolism
Nucleotide Motifs
Promoter Regions, Genetic
Protein Binding
Receptors, Aryl Hydrocarbon
/ metabolism
Receptors, Calcitriol
/ metabolism
T-Lymphocyte Subsets
/ drug effects
Vitamin D
/ pharmacology
Vitamin D Response Element
Interleukin-22
IL-17
IL-22
Th22 cells
vitamin D
vitamin D receptor
vitamin D response element (VDRE)
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2021
2021
Historique:
received:
26
05
2021
accepted:
15
07
2021
entrez:
19
8
2021
pubmed:
20
8
2021
medline:
24
12
2021
Statut:
epublish
Résumé
Th22 cells constitute a recently described CD4
Identifiants
pubmed: 34408754
doi: 10.3389/fimmu.2021.715059
pmc: PMC8366496
doi:
Substances chimiques
AHR protein, human
0
Basic Helix-Loop-Helix Transcription Factors
0
Biomarkers
0
Cytokines
0
Inflammation Mediators
0
Interleukins
0
Nuclear Receptor Subfamily 1, Group F, Member 3
0
Receptors, Aryl Hydrocarbon
0
Receptors, Calcitriol
0
Vitamin D
1406-16-2
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
715059Informations de copyright
Copyright © 2021 Lopez, Al-Jaberi, Damas, Weinert, Pus, Torres-Rusillo, Woetmann, Ødum, Bonefeld, Kongsbak-Wismann and Geisler.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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