A PCR-amplified transgene fragment flanked by a single copy of a truncated inverted terminal repeat for recombinant adeno-associated virus production prevents unnecessary plasmid DNA packaging.


Journal

Gene therapy
ISSN: 1476-5462
Titre abrégé: Gene Ther
Pays: England
ID NLM: 9421525

Informations de publication

Date de publication:
08 2022
Historique:
received: 16 10 2020
accepted: 27 09 2021
revised: 16 09 2021
pubmed: 12 10 2021
medline: 20 8 2022
entrez: 11 10 2021
Statut: ppublish

Résumé

The application of recombinant adeno-associated viruses (rAAVs) for gene therapy faces certain challenges, including genome packaging of non-vector sequences. Inverted terminal repeats (ITRs) flanking the rAAV genome, comprising three inverted repeat regions (A, B, and C) and a non-inverted repeat region (D), contribute to non-vector genome packaging. We aimed to circumvent this issue by comparing the properties of rAAV containing DNA plasmids and PCR-amplified transgenes, including a single copy of the AD sequence (rAAV-pAD/L-AD, respectively), which is a truncated form of ITR, with those of wild-type ITR genome (single-stranded and self-complementary AAV; ssAAV and scAAV). The packaging efficiency of rAAV-pAD/L-AD was found to be comparable to that of scAAV, whereas the transduction efficiency of rAAV-pAD/L-AD was lower than that of ss/scAAV. Remarkably, rAAV-L-AD reduced the plasmid backbone packaging contamination compared to ss/scAAV. Furthermore, to confirm the functionality of this system, we generated a rAAV-L-AD harboring a short hairpin RNA targeting ATP5B (rAAV-L-AD-shATP5B) and found that it caused a significant decrease in ATP5B mRNA levels when transduced into HEK293EB cells, suggesting that it was functional. Thus, our system successfully packaged L-AD into capsids with minimal contamination of plasmid DNA, offering a novel functional packaging platform without causing plasmid backbone encapsidation.

Identifiants

pubmed: 34629464
doi: 10.1038/s41434-021-00299-x
pii: 10.1038/s41434-021-00299-x
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

449-457

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Kumi Adachi (K)

Department of Biochemistry and Molecular Biology, Nippon Medical School, Tokyo, Japan.

Taro Tomono (T)

Department of Biochemistry and Molecular Biology, Nippon Medical School, Tokyo, Japan.
Department of Neurology, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.

Hironori Okada (H)

Department of Biochemistry and Molecular Biology, Nippon Medical School, Tokyo, Japan.

Yusuke Shiozawa (Y)

Laboratory of Molecular Analysis, Nippon Medical School, Tokyo, Japan.

Motoko Yamamoto (M)

Department of Biochemistry and Molecular Biology, Nippon Medical School, Tokyo, Japan.

Yoshitaka Miyagawa (Y)

Department of Biochemistry and Molecular Biology, Nippon Medical School, Tokyo, Japan. yoshitaka-miyagawa@nms.ac.jp.

Takashi Okada (T)

Division of Molecular and Medical Genetics, Center for Gene and Cell Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan. t-okada@ims.u-tokyo.ac.jp.

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