Human ALS/FTD brain organoid slice cultures display distinct early astrocyte and targetable neuronal pathology.


Journal

Nature neuroscience
ISSN: 1546-1726
Titre abrégé: Nat Neurosci
Pays: United States
ID NLM: 9809671

Informations de publication

Date de publication:
11 2021
Historique:
received: 02 04 2021
accepted: 16 08 2021
pubmed: 23 10 2021
medline: 11 11 2021
entrez: 22 10 2021
Statut: ppublish

Résumé

Amyotrophic lateral sclerosis overlapping with frontotemporal dementia (ALS/FTD) is a fatal and currently untreatable disease characterized by rapid cognitive decline and paralysis. Elucidating initial cellular pathologies is central to therapeutic target development, but obtaining samples from presymptomatic patients is not feasible. Here, we report the development of a cerebral organoid slice model derived from human induced pluripotent stem cells (iPSCs) that recapitulates mature cortical architecture and displays early molecular pathology of C9ORF72 ALS/FTD. Using a combination of single-cell RNA sequencing and biological assays, we reveal distinct transcriptional, proteostasis and DNA repair disturbances in astroglia and neurons. We show that astroglia display increased levels of the autophagy signaling protein P62 and that deep layer neurons accumulate dipeptide repeat protein poly(GA), DNA damage and undergo nuclear pyknosis that could be pharmacologically rescued by GSK2606414. Thus, patient-specific iPSC-derived cortical organoid slice cultures are a reproducible translational platform to investigate preclinical ALS/FTD mechanisms as well as novel therapeutic approaches.

Identifiants

pubmed: 34675437
doi: 10.1038/s41593-021-00923-4
pii: 10.1038/s41593-021-00923-4
pmc: PMC8553627
doi:

Substances chimiques

C9orf72 Protein 0
C9orf72 protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1542-1554

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/P008658/1
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 204845/Z/16/Z
Pays : United Kingdom

Informations de copyright

© 2021. The Author(s).

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Auteurs

Kornélia Szebényi (K)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

Léa M D Wenger (LMD)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

Yu Sun (Y)

UK Dementia Research Institute, Cambridge Biomedical Campus, Cambridge, UK.
Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

Alexander W E Dunn (AWE)

Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK.

Colleen A Limegrover (CA)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

George M Gibbons (GM)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

Elena Conci (E)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK.

Ole Paulsen (O)

Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK.

Susanna B Mierau (SB)

Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK.

Gabriel Balmus (G)

UK Dementia Research Institute, Cambridge Biomedical Campus, Cambridge, UK. gb318@cam.ac.uk.
Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK. gb318@cam.ac.uk.

András Lakatos (A)

John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge Biomedical Campus, Cambridge, UK. AL291@cam.ac.uk.
Wellcome Trust-MRC Cambridge Stem Cell Institute, Cambridge Biomedical Campus, Cambridge, UK. AL291@cam.ac.uk.

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