The Effect of Atrasentan on Kidney and Heart Failure Outcomes by Baseline Albuminuria and Kidney Function: A


Journal

Clinical journal of the American Society of Nephrology : CJASN
ISSN: 1555-905X
Titre abrégé: Clin J Am Soc Nephrol
Pays: United States
ID NLM: 101271570

Informations de publication

Date de publication:
12 2021
Historique:
received: 24 05 2021
accepted: 22 09 2021
pubmed: 3 12 2021
medline: 14 1 2023
entrez: 2 12 2021
Statut: ppublish

Résumé

Atrasentan reduces the risk of kidney failure but increases the risk of edema and, possibly, heart failure. Patients with severe CKD may obtain greater absolute kidney benefits from atrasentan but may also be at higher risk of heart failure. We assessed relative and absolute effects of atrasentan on kidney and heart failure events according to baseline eGFR and urinary albumin-creatinine ratio (UACR) in a The effect of atrasentan versus placebo in 3668 patients with type 2 diabetes and CKD with elevated albuminuria was examined in the SONAR trial. We used Cox proportional hazards regression analysis to study effects on the primary kidney outcome (composite of doubling of serum creatinine, kidney failure, or kidney death) and heart failure hospitalization across subgroups of eGFR (<30, ≥30-45, and ≥45 ml/min per 1.73 m Atrasentan reduced the relative risk of the primary kidney outcome (hazard ratio, 0.71; 95% confidence interval, 0.58 to 0.88) consistently across all subgroups of baseline eGFR and UACR (all Atrasentan reduced the relative risk of the primary kidney outcome consistently across baseline UACR and eGFR subgroups. The absolute risk reduction was greater among patients in the lowest eGFR and highest albuminuria category who were at highest baseline risk. Conversely, the relative and absolute risks of heart failure hospitalization were similar across baseline UACR and eGFR subgroups.

Sections du résumé

BACKGROUND AND OBJECTIVES
Atrasentan reduces the risk of kidney failure but increases the risk of edema and, possibly, heart failure. Patients with severe CKD may obtain greater absolute kidney benefits from atrasentan but may also be at higher risk of heart failure. We assessed relative and absolute effects of atrasentan on kidney and heart failure events according to baseline eGFR and urinary albumin-creatinine ratio (UACR) in a
DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS
The effect of atrasentan versus placebo in 3668 patients with type 2 diabetes and CKD with elevated albuminuria was examined in the SONAR trial. We used Cox proportional hazards regression analysis to study effects on the primary kidney outcome (composite of doubling of serum creatinine, kidney failure, or kidney death) and heart failure hospitalization across subgroups of eGFR (<30, ≥30-45, and ≥45 ml/min per 1.73 m
RESULTS
Atrasentan reduced the relative risk of the primary kidney outcome (hazard ratio, 0.71; 95% confidence interval, 0.58 to 0.88) consistently across all subgroups of baseline eGFR and UACR (all
CONCLUSIONS
Atrasentan reduced the relative risk of the primary kidney outcome consistently across baseline UACR and eGFR subgroups. The absolute risk reduction was greater among patients in the lowest eGFR and highest albuminuria category who were at highest baseline risk. Conversely, the relative and absolute risks of heart failure hospitalization were similar across baseline UACR and eGFR subgroups.

Identifiants

pubmed: 34853062
pii: 01277230-202112000-00011
doi: 10.2215/CJN.07340521
pmc: PMC8729501
doi:

Substances chimiques

Atrasentan V6D7VK2215

Banques de données

ClinicalTrials.gov
['NCT01858532']

Types de publication

Randomized Controlled Trial Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1824-1832

Informations de copyright

Copyright © 2021 by the American Society of Nephrology.

Références

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Auteurs

Simke W Waijer (SW)

Department Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands.

Ron T Gansevoort (RT)

Department of Internal Medicine, Division of Nephrology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands.

George L Bakris (GL)

American Society of Hypertension Comprehensive Hypertension Center, University of Chicago Medicine and Biological Sciences, Chicago, Illinois.

Ricardo Correa-Rotter (R)

National Medical Science and Nutrition Institute Salvador Zubirán, Mexico City, Mexico.

Fan-Fan Hou (FF)

Division of Nephrology, Nanfang Hospital, Southern Medical University, National Clinical Research Center for Kidney Disease, Guangzhou, China.

Donald E Kohan (DE)

Division of Nephrology, University of Utah School of Medicine, Salt Lake City, Utah.

Dalane W Kitzman (DW)

Section on Cardiovascular Medicine, Wake Forest School of Medicine, Winston-Salem, North Carolina.

Hirofumi Makino (H)

Okayama University, Okayama, Japan.

John J V McMurray (JJV)

British Heart Foundation Cardiovascular Research Centre, University of Glasgow, Glasgow, United Kingdom.

Vlado Perkovic (V)

George Institute for Global Health and University of New South Wales, Sydney, New South Wales, Australia.

Sheldon Tobe (S)

Division of Nephrology, Sunnybrook Health Sciences Centre, University of Toronto and the Northern Ontario School of Medicine, Toronto, Ontario, Canada.

Hans-Henrik Parving (HH)

Department of Medical Endocrinology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Faculty of Health Science, Aarhus University, Aarhus, Denmark.

Dick de Zeeuw (D)

Department Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands.

Hiddo J L Heerspink (HJL)

Department Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands.
George Institute for Global Health and University of New South Wales, Sydney, New South Wales, Australia.

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