The impact of bariatric surgery on serum tryptophan-kynurenine pathway metabolites.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
07 01 2022
Historique:
received: 28 05 2021
accepted: 03 12 2021
entrez: 8 1 2022
pubmed: 9 1 2022
medline: 25 2 2022
Statut: epublish

Résumé

This study aims to explore the immediate effects of bariatric surgery on serum tryptophan-kynurenine pathway metabolites in individuals with type 2 diabetes and BMI > 30. With the goal of providing insight into the link between tryptophan pathway metabolites, type 2 diabetes, and chronic obesity-induced inflammation. This longitudinal study included 20 participants. Half were diagnosed with type 2 diabetes. 11 and 9 underwent RYGB and SG respectively. Blood samples were obtained at pre-operative and 3 months post-operative timepoints. Tryptophan and downstream metabolites of the kynurenine pathway were quantified with an ultrahigh-performance liquid chromatography tandem mass spectrometry with electrospray ionisation method. At 3 months post-operation, RYGB led to significant reductions in tryptophan, kynurenic acid and xanthurenic acid levels when compared to baseline. Significant reductions of the same metabolites after surgery were also observed in individuals with T2D irrespective of surgical procedure. These metabolites were significantly correlated with serum HbA1c levels and BMI. Bariatric surgery, in particular RYGB reduces serum levels of tryptophan and its downstream kynurenine metabolites. These metabolites are associated with T2D and thought to be potentially mechanistic in the systemic processes of obesity induced inflammation leading to insulin resistance. Its reduction after surgery is associated with an improvement in glycaemic control (HbA1c).

Identifiants

pubmed: 34996930
doi: 10.1038/s41598-021-03833-4
pii: 10.1038/s41598-021-03833-4
pmc: PMC8741964
doi:

Substances chimiques

Biomarkers 0
Glycated Hemoglobin A 0
Xanthurenates 0
hemoglobin A1c protein, human 0
Kynurenine 343-65-7
xanthurenic acid 58LAB1BG8J
Tryptophan 8DUH1N11BX

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

294

Subventions

Organisme : Medical Research Council
ID : MC_PC_12025
Pays : United Kingdom

Informations de copyright

© 2022. The Author(s).

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Auteurs

Kai Tai Derek Yeung (KTD)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.

Nicholas Penney (N)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.

Luke Whiley (L)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.
Australian National Phenome Centre & Centre for Computational & Systems Medicine, Health Futures Institute, Murdoch University, Perth, WA, Australia.
Department of Metabolism, Digestion and Reproduction, Imperial College London, South Kensington, London, UK.

Hutan Ashrafian (H)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.

Matthew R Lewis (MR)

National Phenome Centre, Imperial College London, South Kensington, London, UK.
Department of Metabolism, Digestion and Reproduction, Imperial College London, South Kensington, London, UK.

Sanjay Purkayastha (S)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.

Ara Darzi (A)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK.

Elaine Holmes (E)

Department of Surgery & Cancer, Imperial College London, South Kensington, London, UK. elaine.holmes@imperial.ac.uk.
Australian National Phenome Centre & Centre for Computational & Systems Medicine, Health Futures Institute, Murdoch University, Perth, WA, Australia. elaine.holmes@imperial.ac.uk.

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Classifications MeSH